BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

211 related articles for article (PubMed ID: 15304390)

  • 1. BCR/ABL oncogenic kinase promotes unfaithful repair of the reactive oxygen species-dependent DNA double-strand breaks.
    Nowicki MO; Falinski R; Koptyra M; Slupianek A; Stoklosa T; Gloc E; Nieborowska-Skorska M; Blasiak J; Skorski T
    Blood; 2004 Dec; 104(12):3746-53. PubMed ID: 15304390
    [TBL] [Abstract][Full Text] [Related]  

  • 2. BCR/ABL modifies the kinetics and fidelity of DNA double-strand breaks repair in hematopoietic cells.
    Slupianek A; Nowicki MO; Koptyra M; Skorski T
    DNA Repair (Amst); 2006 Feb; 5(2):243-50. PubMed ID: 16297667
    [TBL] [Abstract][Full Text] [Related]  

  • 3. BCR/ABL stimulates WRN to promote survival and genomic instability.
    Slupianek A; Poplawski T; Jozwiakowski SK; Cramer K; Pytel D; Stoczynska E; Nowicki MO; Blasiak J; Skorski T
    Cancer Res; 2011 Feb; 71(3):842-51. PubMed ID: 21123451
    [TBL] [Abstract][Full Text] [Related]  

  • 4. BCR/ABL downregulates DNA-PK(CS)-dependent and upregulates backup non-homologous end joining in leukemic cells.
    Poplawski T; Blasiak J
    Mol Biol Rep; 2010 Jun; 37(5):2309-15. PubMed ID: 19697154
    [TBL] [Abstract][Full Text] [Related]  

  • 5. ATR-Chk1 axis protects BCR/ABL leukemia cells from the lethal effect of DNA double-strand breaks.
    Nieborowska-Skorska M; Stoklosa T; Datta M; Czechowska A; Rink L; Slupianek A; Koptyra M; Seferynska I; Krszyna K; Blasiak J; Skorski T
    Cell Cycle; 2006 May; 5(9):994-1000. PubMed ID: 16687921
    [TBL] [Abstract][Full Text] [Related]  

  • 6. BCR/ABL and other kinases from chronic myeloproliferative disorders stimulate single-strand annealing, an unfaithful DNA double-strand break repair.
    Cramer K; Nieborowska-Skorska M; Koptyra M; Slupianek A; Penserga ET; Eaves CJ; Aulitzky W; Skorski T
    Cancer Res; 2008 Sep; 68(17):6884-8. PubMed ID: 18757400
    [TBL] [Abstract][Full Text] [Related]  

  • 7. BCR/ABL, DNA damage and DNA repair: implications for new treatment concepts.
    Skorski T
    Leuk Lymphoma; 2008 Apr; 49(4):610-4. PubMed ID: 18398719
    [TBL] [Abstract][Full Text] [Related]  

  • 8. Does the bcr/abl-mediated increase in the efficacy of DNA repair play a role in the drug resistance of cancer cells?
    Majsterek I; Blasiak J; Mlynarski W; Hoser G; Skórski T
    Cell Biol Int; 2002; 26(4):363-70. PubMed ID: 11991666
    [TBL] [Abstract][Full Text] [Related]  

  • 9. Non-homologous DNA end joining repair in normal and leukemic cells depends on the substrate ends.
    Pastwa E; Poplawski T; Czechowska A; Malinowski M; Blasiak J
    Z Naturforsch C J Biosci; 2005; 60(5-6):493-500. PubMed ID: 16042351
    [TBL] [Abstract][Full Text] [Related]  

  • 10. Genomic instability in myeloid malignancies: increased reactive oxygen species (ROS), DNA double strand breaks (DSBs) and error-prone repair.
    Sallmyr A; Fan J; Rassool FV
    Cancer Lett; 2008 Oct; 270(1):1-9. PubMed ID: 18467025
    [TBL] [Abstract][Full Text] [Related]  

  • 11. DNA Repair--A Double-Edged Sword in the Genomic Stability of Cancer Cells--The Case of Chronic Myeloid Leukemia.
    Pawlowska E; Blasiak J
    Int J Mol Sci; 2015 Nov; 16(11):27535-49. PubMed ID: 26593906
    [TBL] [Abstract][Full Text] [Related]  

  • 12. Up-regulation of WRN and DNA ligase IIIalpha in chronic myeloid leukemia: consequences for the repair of DNA double-strand breaks.
    Sallmyr A; Tomkinson AE; Rassool FV
    Blood; 2008 Aug; 112(4):1413-23. PubMed ID: 18524993
    [TBL] [Abstract][Full Text] [Related]  

  • 13. Imatinib (STI571) induces DNA damage in BCR/ABL-expressing leukemic cells but not in normal lymphocytes.
    Czechowska A; Poplawski T; Drzewoski J; Blasiak J
    Chem Biol Interact; 2005 Apr; 152(2-3):139-50. PubMed ID: 15840387
    [TBL] [Abstract][Full Text] [Related]  

  • 14. Activated IGF-1R inhibits hyperglycemia-induced DNA damage and promotes DNA repair by homologous recombination.
    Yang S; Chintapalli J; Sodagum L; Baskin S; Malhotra A; Reiss K; Meggs LG
    Am J Physiol Renal Physiol; 2005 Nov; 289(5):F1144-52. PubMed ID: 15956778
    [TBL] [Abstract][Full Text] [Related]  

  • 15. BCR-ABL stimulates mutagenic homologous DNA double-strand break repair via the DNA-end-processing factor CtIP.
    Salles D; Mencalha AL; Ireno IC; Wiesmüller L; Abdelhay E
    Carcinogenesis; 2011 Jan; 32(1):27-34. PubMed ID: 20974687
    [TBL] [Abstract][Full Text] [Related]  

  • 16. BCR-ABL1 kinase facilitates localization of acetylated histones 3 and 4 on DNA double-strand breaks.
    Falinski R; Nieborowska-Skorska M; Skorski T
    Leuk Res; 2012 Feb; 36(2):241-4. PubMed ID: 22036635
    [TBL] [Abstract][Full Text] [Related]  

  • 17. BLM helicase is activated in BCR/ABL leukemia cells to modulate responses to cisplatin.
    Slupianek A; Gurdek E; Koptyra M; Nowicki MO; Siddiqui KM; Groden J; Skorski T
    Oncogene; 2005 Jun; 24(24):3914-22. PubMed ID: 15750625
    [TBL] [Abstract][Full Text] [Related]  

  • 18. Leukemia patient-derived lymphoblastoid cell lines exhibit increased induction of leukemia-associated transcripts following high-dose irradiation.
    Spencer A; Granter N
    Exp Hematol; 1999 Sep; 27(9):1397-401. PubMed ID: 10480430
    [TBL] [Abstract][Full Text] [Related]  

  • 19. ABL-fusion oncoproteins activate multi-pathway of DNA repair: role in drug resistance?
    Majsterek I; Slupianek A; Hoser G; Skórski T; Blasiak J
    Biochimie; 2004 Jan; 86(1):53-65. PubMed ID: 14987801
    [TBL] [Abstract][Full Text] [Related]  

  • 20. BCR/ABL induces chromosomal instability after genotoxic stress and alters the cell death threshold.
    Dierov J; Sanchez PV; Burke BA; Padilla-Nash H; Putt ME; Ried T; Carroll M
    Leukemia; 2009 Feb; 23(2):279-86. PubMed ID: 19020542
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 11.