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5. Ca2+ influx in human T lymphocytes is induced independently of inositol phosphate production by mobilization of intracellular Ca2+ stores. A study with the Ca2+ endoplasmic reticulum-ATPase inhibitor thapsigargin. Gouy H; Cefai D; Christensen SB; Debré P; Bismuth G Eur J Immunol; 1990 Oct; 20(10):2269-75. PubMed ID: 1700752 [TBL] [Abstract][Full Text] [Related]
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7. Inhibition by nicardipine of endothelin-mediated inositol phosphate formation and Ca2+ mobilization in smooth muscle cell. Xuan YT; Whorton AR; Watkins WD Biochem Biophys Res Commun; 1989 Apr; 160(2):758-64. PubMed ID: 2541709 [TBL] [Abstract][Full Text] [Related]
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10. Inhibition of L-type calcium-channel activity by thapsigargin and 2,5-t-butylhydroquinone, but not by cyclopiazonic acid. Nelson EJ; Li CC; Bangalore R; Benson T; Kass RS; Hinkle PM Biochem J; 1994 Aug; 302 ( Pt 1)(Pt 1):147-54. PubMed ID: 7520693 [TBL] [Abstract][Full Text] [Related]
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12. Regulation of endothelin-mediated calcium mobilization in vascular smooth muscle cells by isoproterenol. Xuan YT; Watkins WD; Whorton AR Am J Physiol; 1991 Mar; 260(3 Pt 1):C492-502. PubMed ID: 1706144 [TBL] [Abstract][Full Text] [Related]
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15. Activation of calcium entry by the tumor promoter thapsigargin in parotid acinar cells. Evidence that an intracellular calcium pool and not an inositol phosphate regulates calcium fluxes at the plasma membrane. Takemura H; Hughes AR; Thastrup O; Putney JW J Biol Chem; 1989 Jul; 264(21):12266-71. PubMed ID: 2663854 [TBL] [Abstract][Full Text] [Related]
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