197 related articles for article (PubMed ID: 15364958)
1. Functional interaction between BLM helicase and 53BP1 in a Chk1-mediated pathway during S-phase arrest.
Sengupta S; Robles AI; Linke SP; Sinogeeva NI; Zhang R; Pedeux R; Ward IM; Celeste A; Nussenzweig A; Chen J; Halazonetis TD; Harris CC
J Cell Biol; 2004 Sep; 166(6):801-13. PubMed ID: 15364958
[TBL] [Abstract][Full Text] [Related]
2. BLM helicase-dependent and -independent roles of 53BP1 during replication stress-mediated homologous recombination.
Tripathi V; Nagarjuna T; Sengupta S
J Cell Biol; 2007 Jul; 178(1):9-14. PubMed ID: 17591918
[TBL] [Abstract][Full Text] [Related]
3. Phosphorylation of the Bloom's syndrome helicase and its role in recovery from S-phase arrest.
Davies SL; North PS; Dart A; Lakin ND; Hickson ID
Mol Cell Biol; 2004 Feb; 24(3):1279-91. PubMed ID: 14729972
[TBL] [Abstract][Full Text] [Related]
4. Phosphorylation-dependent interactions of BLM and 53BP1 are required for their anti-recombinogenic roles during homologous recombination.
Tripathi V; Kaur S; Sengupta S
Carcinogenesis; 2008 Jan; 29(1):52-61. PubMed ID: 17984114
[TBL] [Abstract][Full Text] [Related]
5. BLM helicase-dependent transport of p53 to sites of stalled DNA replication forks modulates homologous recombination.
Sengupta S; Linke SP; Pedeux R; Yang Q; Farnsworth J; Garfield SH; Valerie K; Shay JW; Ellis NA; Wasylyk B; Harris CC
EMBO J; 2003 Mar; 22(5):1210-22. PubMed ID: 12606585
[TBL] [Abstract][Full Text] [Related]
6. ATR and ATM-dependent movement of BLM helicase during replication stress ensures optimal ATM activation and 53BP1 focus formation.
Davalos AR; Kaminker P; Hansen RK; Campisi J
Cell Cycle; 2004 Dec; 3(12):1579-86. PubMed ID: 15539948
[TBL] [Abstract][Full Text] [Related]
7. Phosphorylation of BLM, dissociation from topoisomerase IIIalpha, and colocalization with gamma-H2AX after topoisomerase I-induced replication damage.
Rao VA; Fan AM; Meng L; Doe CF; North PS; Hickson ID; Pommier Y
Mol Cell Biol; 2005 Oct; 25(20):8925-37. PubMed ID: 16199871
[TBL] [Abstract][Full Text] [Related]
8. 53BP1, a mediator of the DNA damage checkpoint.
Wang B; Matsuoka S; Carpenter PB; Elledge SJ
Science; 2002 Nov; 298(5597):1435-8. PubMed ID: 12364621
[TBL] [Abstract][Full Text] [Related]
9. Bloom's syndrome protein response to ultraviolet-C radiation and hydroxyurea-mediated DNA synthesis inhibition.
Ababou M; Dumaire V; Lécluse Y; Amor-Guéret M
Oncogene; 2002 Mar; 21(13):2079-88. PubMed ID: 11960380
[TBL] [Abstract][Full Text] [Related]
10. ATM-dependent phosphorylation and accumulation of endogenous BLM protein in response to ionizing radiation.
Ababou M; Dutertre S; Lécluse Y; Onclercq R; Chatton B; Amor-Guéret M
Oncogene; 2000 Dec; 19(52):5955-63. PubMed ID: 11146546
[TBL] [Abstract][Full Text] [Related]
11. Bloom syndrome cells undergo p53-dependent apoptosis and delayed assembly of BRCA1 and NBS1 repair complexes at stalled replication forks.
Davalos AR; Campisi J
J Cell Biol; 2003 Sep; 162(7):1197-209. PubMed ID: 14517203
[TBL] [Abstract][Full Text] [Related]
12. Bloom's syndrome protein is required for correct relocalization of RAD50/MRE11/NBS1 complex after replication fork arrest.
Franchitto A; Pichierri P
J Cell Biol; 2002 Apr; 157(1):19-30. PubMed ID: 11916980
[TBL] [Abstract][Full Text] [Related]
13. Analysis of Rad3 and Chk1 protein kinases defines different checkpoint responses.
Martinho RG; Lindsay HD; Flaggs G; DeMaggio AJ; Hoekstra MF; Carr AM; Bentley NJ
EMBO J; 1998 Dec; 17(24):7239-49. PubMed ID: 9857181
[TBL] [Abstract][Full Text] [Related]
14. Phosphorylation and rapid relocalization of 53BP1 to nuclear foci upon DNA damage.
Anderson L; Henderson C; Adachi Y
Mol Cell Biol; 2001 Mar; 21(5):1719-29. PubMed ID: 11238909
[TBL] [Abstract][Full Text] [Related]
15. Functional interaction of p53 and BLM DNA helicase in apoptosis.
Wang XW; Tseng A; Ellis NA; Spillare EA; Linke SP; Robles AI; Seker H; Yang Q; Hu P; Beresten S; Bemmels NA; Garfield S; Harris CC
J Biol Chem; 2001 Aug; 276(35):32948-55. PubMed ID: 11399766
[TBL] [Abstract][Full Text] [Related]
16. MPS1-dependent mitotic BLM phosphorylation is important for chromosome stability.
Leng M; Chan DW; Luo H; Zhu C; Qin J; Wang Y
Proc Natl Acad Sci U S A; 2006 Aug; 103(31):11485-90. PubMed ID: 16864798
[TBL] [Abstract][Full Text] [Related]
17. Chk1-dependent constitutive phosphorylation of BLM helicase at serine 646 decreases after DNA damage.
Kaur S; Modi P; Srivastava V; Mudgal R; Tikoo S; Arora P; Mohanty D; Sengupta S
Mol Cancer Res; 2010 Sep; 8(9):1234-47. PubMed ID: 20719863
[TBL] [Abstract][Full Text] [Related]
18. BLM helicase facilitates Mus81 endonuclease activity in human cells.
Zhang R; Sengupta S; Yang Q; Linke SP; Yanaihara N; Bradsher J; Blais V; McGowan CH; Harris CC
Cancer Res; 2005 Apr; 65(7):2526-31. PubMed ID: 15805243
[TBL] [Abstract][Full Text] [Related]
19. Cell cycle regulation of the endogenous wild type Bloom's syndrome DNA helicase.
Dutertre S; Ababou M; Onclercq R; Delic J; Chatton B; Jaulin C; Amor-Guéret M
Oncogene; 2000 May; 19(23):2731-8. PubMed ID: 10851073
[TBL] [Abstract][Full Text] [Related]
20. Dephosphorylation and subcellular compartment change of the mitotic Bloom's syndrome DNA helicase in response to ionizing radiation.
Dutertre S; Sekhri R; Tintignac LA; Onclercq-Delic R; Chatton B; Jaulin C; Amor-Guéret M
J Biol Chem; 2002 Feb; 277(8):6280-6. PubMed ID: 11741924
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]