175 related articles for article (PubMed ID: 15378446)
1. Phosphorylation-independent effects of CagA during interaction between Helicobacter pylori and T84 polarized monolayers.
El-Etr SH; Mueller A; Tompkins LS; Falkow S; Merrell DS
J Infect Dis; 2004 Oct; 190(8):1516-23. PubMed ID: 15378446
[TBL] [Abstract][Full Text] [Related]
2. Identification of a tyrosine-phosphorylated 35 kDa carboxy-terminal fragment (p35CagA) of the Helicobacter pylori CagA protein in phagocytic cells: processing or breakage?
Moese S; Selbach M; Zimny-Arndt U; Jungblut PR; Meyer TF; Backert S
Proteomics; 2001 Apr; 1(4):618-29. PubMed ID: 11681214
[TBL] [Abstract][Full Text] [Related]
3. Role of type IV secretion in Helicobacter pylori pathogenesis.
Backert S; Selbach M
Cell Microbiol; 2008 Aug; 10(8):1573-81. PubMed ID: 18410539
[TBL] [Abstract][Full Text] [Related]
4. Structural and functional diversity in the PAR1b/MARK2-binding region of Helicobacter pylori CagA.
Lu HS; Saito Y; Umeda M; Murata-Kamiya N; Zhang HM; Higashi H; Hatakeyama M
Cancer Sci; 2008 Oct; 99(10):2004-11. PubMed ID: 19016760
[TBL] [Abstract][Full Text] [Related]
5. Functional antagonism between Helicobacter pylori CagA and vacuolating toxin VacA in control of the NFAT signaling pathway in gastric epithelial cells.
Yokoyama K; Higashi H; Ishikawa S; Fujii Y; Kondo S; Kato H; Azuma T; Wada A; Hirayama T; Aburatani H; Hatakeyama M
Proc Natl Acad Sci U S A; 2005 Jul; 102(27):9661-6. PubMed ID: 15980153
[TBL] [Abstract][Full Text] [Related]
6. Helicobacter pylori CagA targets PAR1/MARK kinase to disrupt epithelial cell polarity.
Saadat I; Higashi H; Obuse C; Umeda M; Murata-Kamiya N; Saito Y; Lu H; Ohnishi N; Azuma T; Suzuki A; Ohno S; Hatakeyama M
Nature; 2007 May; 447(7142):330-3. PubMed ID: 17507984
[TBL] [Abstract][Full Text] [Related]
7. The versatility of Helicobacter pylori CagA effector protein functions: The master key hypothesis.
Backert S; Tegtmeyer N; Selbach M
Helicobacter; 2010 Jun; 15(3):163-76. PubMed ID: 20557357
[TBL] [Abstract][Full Text] [Related]
8. CagA-independent disruption of adherence junction complexes involves E-cadherin shedding and implies multiple steps in Helicobacter pylori pathogenicity.
Weydig C; Starzinski-Powitz A; Carra G; Löwer J; Wessler S
Exp Cell Res; 2007 Oct; 313(16):3459-71. PubMed ID: 17692843
[TBL] [Abstract][Full Text] [Related]
9. Helicobacter pylori HopH (OipA) and bacterial pathogenicity: genetic and functional genomic analysis of hopH gene polymorphisms.
Dossumbekova A; Prinz C; Mages J; Lang R; Kusters JG; Van Vliet AH; Reindl W; Backert S; Saur D; Schmid RM; Rad R
J Infect Dis; 2006 Nov; 194(10):1346-55. PubMed ID: 17054063
[TBL] [Abstract][Full Text] [Related]
10. Interaction of CagA with Crk plays an important role in Helicobacter pylori-induced loss of gastric epithelial cell adhesion.
Suzuki M; Mimuro H; Suzuki T; Park M; Yamamoto T; Sasakawa C
J Exp Med; 2005 Nov; 202(9):1235-47. PubMed ID: 16275761
[TBL] [Abstract][Full Text] [Related]
11. Gene expression and protein profiling of AGS gastric epithelial cells upon infection with Helicobacter pylori.
Backert S; Gressmann H; Kwok T; Zimny-Arndt U; König W; Jungblut PR; Meyer TF
Proteomics; 2005 Oct; 5(15):3902-18. PubMed ID: 16145711
[TBL] [Abstract][Full Text] [Related]
12. The genome-wide expression profile of gastric epithelial cells infected by naturally occurring cagA isogenic strains of Helicobacter pylori.
Sohn SH; Lee YC
Environ Toxicol Pharmacol; 2011 Nov; 32(3):382-9. PubMed ID: 22004957
[TBL] [Abstract][Full Text] [Related]
13. Tyrosine phosphorylation patterns and size modification of the Helicobacter pylori CagA protein after translocation into gastric epithelial cells.
Backert S; Müller EC; Jungblut PR; Meyer TF
Proteomics; 2001 Apr; 1(4):608-17. PubMed ID: 11681213
[TBL] [Abstract][Full Text] [Related]
14. Helicobacter pylori CagA: a new paradigm for bacterial carcinogenesis.
Hatakeyama M; Higashi H
Cancer Sci; 2005 Dec; 96(12):835-43. PubMed ID: 16367902
[TBL] [Abstract][Full Text] [Related]
15. Helicobacter pylori-host cell interactions mediated by type IV secretion.
Bourzac KM; Guillemin K
Cell Microbiol; 2005 Jul; 7(7):911-9. PubMed ID: 15953024
[TBL] [Abstract][Full Text] [Related]
16. Helicobacter pylori CagA protein activates serum response element-driven transcription independently of tyrosine phosphorylation.
Hirata Y; Maeda S; Mitsuno Y; Tateishi K; Yanai A; Akanuma M; Yoshida H; Kawabe T; Shiratori Y; Omata M
Gastroenterology; 2002 Dec; 123(6):1962-71. PubMed ID: 12454853
[TBL] [Abstract][Full Text] [Related]
17. Activation of Abl by Helicobacter pylori: a novel kinase for CagA and crucial mediator of host cell scattering.
Tammer I; Brandt S; Hartig R; König W; Backert S
Gastroenterology; 2007 Apr; 132(4):1309-19. PubMed ID: 17408661
[TBL] [Abstract][Full Text] [Related]
18. Phosphorylation of Helicobacter pylori CagA by c-Abl leads to cell motility.
Poppe M; Feller SM; Römer G; Wessler S
Oncogene; 2007 May; 26(24):3462-72. PubMed ID: 17160020
[TBL] [Abstract][Full Text] [Related]
19. Pathogenesis of Helicobacter pylori infection.
Costa AC; Figueiredo C; Touati E
Helicobacter; 2009 Sep; 14 Suppl 1():15-20. PubMed ID: 19712163
[TBL] [Abstract][Full Text] [Related]
20. [The type IV secretion system encoded by the cag PAI of Helicobacter pylori].
Cui LL; Shao SH
Wei Sheng Wu Xue Bao; 2007 Aug; 47(4):743-5. PubMed ID: 17944386
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]