238 related articles for article (PubMed ID: 15649979)
1. Increased persistent Na(+) current and its effect on excitability in motoneurones cultured from mutant SOD1 mice.
Kuo JJ; Siddique T; Fu R; Heckman CJ
J Physiol; 2005 Mar; 563(Pt 3):843-54. PubMed ID: 15649979
[TBL] [Abstract][Full Text] [Related]
2. Spinal motoneurones are intrinsically more responsive in the adult G93A SOD1 mouse model of amyotrophic lateral sclerosis.
Jensen DB; Kadlecova M; Allodi I; Meehan CF
J Physiol; 2020 Oct; 598(19):4385-4403. PubMed ID: 32716521
[TBL] [Abstract][Full Text] [Related]
3. Increased persistent sodium current determines cortical hyperexcitability in a genetic model of amyotrophic lateral sclerosis.
Pieri M; Carunchio I; Curcio L; Mercuri NB; Zona C
Exp Neurol; 2009 Feb; 215(2):368-79. PubMed ID: 19071115
[TBL] [Abstract][Full Text] [Related]
4. Hyperexcitability of cultured spinal motoneurons from presymptomatic ALS mice.
Kuo JJ; Schonewille M; Siddique T; Schults AN; Fu R; Bär PR; Anelli R; Heckman CJ; Kroese AB
J Neurophysiol; 2004 Jan; 91(1):571-5. PubMed ID: 14523070
[TBL] [Abstract][Full Text] [Related]
5. Intrinsic properties of lumbar motor neurones in the adult G127insTGGG superoxide dismutase-1 mutant mouse in vivo: evidence for increased persistent inward currents.
Meehan CF; Moldovan M; Marklund SL; Graffmo KS; Nielsen JB; Hultborn H
Acta Physiol (Oxf); 2010 Dec; 200(4):361-76. PubMed ID: 20874803
[TBL] [Abstract][Full Text] [Related]
6. Postnatal electrical and morphological abnormalities in lumbar motoneurons from transgenic mouse models of amyotrophic lateral sclerosis.
Amendola J; Gueritaud JP; Lamotte d'Incamps B; Bories C; Liabeuf S; Allene C; Pambo-Pambo A; Durand J
Arch Ital Biol; 2007 Nov; 145(3-4):311-23. PubMed ID: 18075124
[TBL] [Abstract][Full Text] [Related]
7. Increased Axon Initial Segment Length Results in Increased Na
Jørgensen HS; Jensen DB; Dimintiyanova KP; Bonnevie VS; Hedegaard A; Lehnhoff J; Moldovan M; Grondahl L; Meehan CF
Neuroscience; 2021 Aug; 468():247-264. PubMed ID: 33246068
[TBL] [Abstract][Full Text] [Related]
8. Voltage-dependent sodium channels in spinal cord motor neurons display rapid recovery from fast inactivation in a mouse model of amyotrophic lateral sclerosis.
Zona C; Pieri M; Carunchio I
J Neurophysiol; 2006 Dec; 96(6):3314-22. PubMed ID: 16899637
[TBL] [Abstract][Full Text] [Related]
9. Mutant SOD1-expressing astrocytes release toxic factors that trigger motoneuron death by inducing hyperexcitability.
Fritz E; Izaurieta P; Weiss A; Mir FR; Rojas P; Gonzalez D; Rojas F; Brown RH; Madrid R; van Zundert B
J Neurophysiol; 2013 Jun; 109(11):2803-14. PubMed ID: 23486205
[TBL] [Abstract][Full Text] [Related]
10. Altered long-term corticostriatal synaptic plasticity in transgenic mice overexpressing human CU/ZN superoxide dismutase (GLY(93)-->ALA) mutation.
Geracitano R; Paolucci E; Prisco S; Guatteo E; Zona C; Longone P; Ammassari-Teule M; Bernardi G; Berretta N; Mercuri NB
Neuroscience; 2003; 118(2):399-408. PubMed ID: 12699776
[TBL] [Abstract][Full Text] [Related]
11. Human Cu/Zn superoxide dismutase (SOD1) overexpression in mice causes mitochondrial vacuolization, axonal degeneration, and premature motoneuron death and accelerates motoneuron disease in mice expressing a familial amyotrophic lateral sclerosis mutant SOD1.
Jaarsma D; Haasdijk ED; Grashorn JA; Hawkins R; van Duijn W; Verspaget HW; London J; Holstege JC
Neurobiol Dis; 2000 Dec; 7(6 Pt B):623-43. PubMed ID: 11114261
[TBL] [Abstract][Full Text] [Related]
12. Differential Effects of Invasive Anodal Trans-spinal Direct Current Stimulation on Monosynaptic Excitatory Postsynaptic Potentials, Ia Afferents Excitability, and Motoneuron Intrinsic Properties Between Superoxide Dismutase Type-1 Glycine to Alanine Substitution at Position 93 and Wildtype Mice.
Jankowiak T; Cholewiński M; Bączyk M
Neuroscience; 2022 Aug; 498():125-143. PubMed ID: 35792195
[TBL] [Abstract][Full Text] [Related]
13. Ultrastructural study of mitochondria in the spinal cord of transgenic mice with a G93A mutant SOD1 gene.
Sasaki S; Warita H; Murakami T; Abe K; Iwata M
Acta Neuropathol; 2004 May; 107(5):461-74. PubMed ID: 15029445
[TBL] [Abstract][Full Text] [Related]
14. An ALS-Associated Mutant SOD1 Rapidly Suppresses KCNT1 (Slack) Na
Zhang Y; Ni W; Horwich AL; Kaczmarek LK
J Neurosci; 2017 Feb; 37(8):2258-2265. PubMed ID: 28119399
[TBL] [Abstract][Full Text] [Related]
15. Progressive loss of a glial potassium channel (KCNJ10) in the spinal cord of the SOD1 (G93A) transgenic mouse model of amyotrophic lateral sclerosis.
Kaiser M; Maletzki I; Hülsmann S; Holtmann B; Schulz-Schaeffer W; Kirchhoff F; Bähr M; Neusch C
J Neurochem; 2006 Nov; 99(3):900-12. PubMed ID: 16925593
[TBL] [Abstract][Full Text] [Related]
16. Wild-type nonneuronal cells extend survival of SOD1 mutant motor neurons in ALS mice.
Clement AM; Nguyen MD; Roberts EA; Garcia ML; Boillée S; Rule M; McMahon AP; Doucette W; Siwek D; Ferrante RJ; Brown RH; Julien JP; Goldstein LS; Cleveland DW
Science; 2003 Oct; 302(5642):113-7. PubMed ID: 14526083
[TBL] [Abstract][Full Text] [Related]
17. Increased expression of the beta3 subunit of voltage-gated Na+ channels in the spinal cord of the SOD1G93A mouse.
Nutini M; Spalloni A; Florenzano F; Westenbroek RE; Marini C; Catterall WA; Bernardi G; Longone P
Mol Cell Neurosci; 2011 Jun; 47(2):108-18. PubMed ID: 21458573
[TBL] [Abstract][Full Text] [Related]
18. Adult spinal motoneurones are not hyperexcitable in a mouse model of inherited amyotrophic lateral sclerosis.
Delestrée N; Manuel M; Iglesias C; Elbasiouny SM; Heckman CJ; Zytnicki D
J Physiol; 2014 Apr; 592(7):1687-703. PubMed ID: 24445319
[TBL] [Abstract][Full Text] [Related]
19. HoxB2 binds mutant SOD1 and is altered in transgenic model of ALS.
Zhai J; Lin H; Canete-Soler R; Schlaepfer WW
Hum Mol Genet; 2005 Sep; 14(18):2629-40. PubMed ID: 16079151
[TBL] [Abstract][Full Text] [Related]
20. Essential role of the persistent sodium current in spike initiation during slowly rising inputs in mouse spinal neurones.
Kuo JJ; Lee RH; Zhang L; Heckman CJ
J Physiol; 2006 Aug; 574(Pt 3):819-34. PubMed ID: 16728453
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]