BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

561 related articles for article (PubMed ID: 15652988)

  • 1. The effect of peripheral nerve injury on disease progression in the SOD1(G93A) mouse model of amyotrophic lateral sclerosis.
    Sharp PS; Dick JR; Greensmith L
    Neuroscience; 2005; 130(4):897-910. PubMed ID: 15652988
    [TBL] [Abstract][Full Text] [Related]  

  • 2. Functional over-load saves motor units in the SOD1-G93A transgenic mouse model of amyotrophic lateral sclerosis.
    Gordon T; Tyreman N; Li S; Putman CT; Hegedus J
    Neurobiol Dis; 2010 Feb; 37(2):412-22. PubMed ID: 19879358
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Time course of preferential motor unit loss in the SOD1 G93A mouse model of amyotrophic lateral sclerosis.
    Hegedus J; Putman CT; Gordon T
    Neurobiol Dis; 2007 Nov; 28(2):154-64. PubMed ID: 17766128
    [TBL] [Abstract][Full Text] [Related]  

  • 4. Crush injury to motor nerves in the G93A transgenic mouse model of amyotrophic lateral sclerosis promotes muscle reinnervation and survival of functionally intact nerve-muscle contacts.
    Sharp PS; Tyreman N; Jones KE; Gordon T
    Neurobiol Dis; 2018 May; 113():33-44. PubMed ID: 29409912
    [TBL] [Abstract][Full Text] [Related]  

  • 5. Progressive impairment of CaV1.1 function in the skeletal muscle of mice expressing a mutant type 1 Cu/Zn superoxide dismutase (G93A) linked to amyotrophic lateral sclerosis.
    Beqollari D; Romberg CF; Dobrowolny G; Martini M; Voss AA; Musarò A; Bannister RA
    Skelet Muscle; 2016; 6():24. PubMed ID: 27340545
    [TBL] [Abstract][Full Text] [Related]  

  • 6. Amyloid precursor protein (APP) contributes to pathology in the SOD1(G93A) mouse model of amyotrophic lateral sclerosis.
    Bryson JB; Hobbs C; Parsons MJ; Bosch KD; Pandraud A; Walsh FS; Doherty P; Greensmith L
    Hum Mol Genet; 2012 Sep; 21(17):3871-82. PubMed ID: 22678056
    [TBL] [Abstract][Full Text] [Related]  

  • 7. Human Cu/Zn superoxide dismutase (SOD1) overexpression in mice causes mitochondrial vacuolization, axonal degeneration, and premature motoneuron death and accelerates motoneuron disease in mice expressing a familial amyotrophic lateral sclerosis mutant SOD1.
    Jaarsma D; Haasdijk ED; Grashorn JA; Hawkins R; van Duijn W; Verspaget HW; London J; Holstege JC
    Neurobiol Dis; 2000 Dec; 7(6 Pt B):623-43. PubMed ID: 11114261
    [TBL] [Abstract][Full Text] [Related]  

  • 8. The expression of the chemorepellent Semaphorin 3A is selectively induced in terminal Schwann cells of a subset of neuromuscular synapses that display limited anatomical plasticity and enhanced vulnerability in motor neuron disease.
    De Winter F; Vo T; Stam FJ; Wisman LA; Bär PR; Niclou SP; van Muiswinkel FL; Verhaagen J
    Mol Cell Neurosci; 2006; 32(1-2):102-17. PubMed ID: 16677822
    [TBL] [Abstract][Full Text] [Related]  

  • 9. Early vulnerability to ischemia/reperfusion injury in motor terminals innervating fast muscles of SOD1-G93A mice.
    David G; Nguyen K; Barrett EF
    Exp Neurol; 2007 Mar; 204(1):411-20. PubMed ID: 17292357
    [TBL] [Abstract][Full Text] [Related]  

  • 10. Intact single muscle fibres from SOD1
    Cheng AJ; Allodi I; Chaillou T; Schlittler M; Ivarsson N; Lanner JT; Thams S; Hedlund E; Andersson DC
    J Physiol; 2019 Jun; 597(12):3133-3146. PubMed ID: 31074054
    [TBL] [Abstract][Full Text] [Related]  

  • 11. Transcriptomic indices of fast and slow disease progression in two mouse models of amyotrophic lateral sclerosis.
    Nardo G; Iennaco R; Fusi N; Heath PR; Marino M; Trolese MC; Ferraiuolo L; Lawrence N; Shaw PJ; Bendotti C
    Brain; 2013 Nov; 136(Pt 11):3305-32. PubMed ID: 24065725
    [TBL] [Abstract][Full Text] [Related]  

  • 12. Preferential motor unit loss in the SOD1 G93A transgenic mouse model of amyotrophic lateral sclerosis.
    Hegedus J; Putman CT; Tyreman N; Gordon T
    J Physiol; 2008 Jul; 586(14):3337-51. PubMed ID: 18467368
    [TBL] [Abstract][Full Text] [Related]  

  • 13. Mutant SOD1(G93A) microglia are more neurotoxic relative to wild-type microglia.
    Xiao Q; Zhao W; Beers DR; Yen AA; Xie W; Henkel JS; Appel SH
    J Neurochem; 2007 Sep; 102(6):2008-2019. PubMed ID: 17555556
    [TBL] [Abstract][Full Text] [Related]  

  • 14. Deficiency in the ALS2 gene does not affect the motor neuron degeneration in SOD1(G93A) transgenic mice.
    Lin X; Shim H; Cai H
    Neurobiol Aging; 2007 Oct; 28(10):1628-30. PubMed ID: 16973244
    [TBL] [Abstract][Full Text] [Related]  

  • 15. Preservation of neuromuscular function in symptomatic SOD1-G93A mice by peripheral infusion of methylene blue.
    Talbot JD; Barrett JN; Nonner D; Zhang Z; Wicomb K; Barrett EF
    Exp Neurol; 2016 Nov; 285(Pt A):96-107. PubMed ID: 27567739
    [TBL] [Abstract][Full Text] [Related]  

  • 16. Coincident thresholds of mutant protein for paralytic disease and protein aggregation caused by restrictively expressed superoxide dismutase cDNA.
    Wang J; Xu G; Slunt HH; Gonzales V; Coonfield M; Fromholt D; Copeland NG; Jenkins NA; Borchelt DR
    Neurobiol Dis; 2005 Dec; 20(3):943-52. PubMed ID: 16046140
    [TBL] [Abstract][Full Text] [Related]  

  • 17. Pre-symptomatic detection of chronic motor deficits and genotype prediction in congenic B6.SOD1(G93A) ALS mouse model.
    Hayworth CR; Gonzalez-Lima F
    Neuroscience; 2009 Dec; 164(3):975-85. PubMed ID: 19699279
    [TBL] [Abstract][Full Text] [Related]  

  • 18. MRI detects early hindlimb muscle atrophy in Gly93Ala superoxide dismutase-1 (G93A SOD1) transgenic mice, an animal model of familial amyotrophic lateral sclerosis.
    Brooks KJ; Hill MD; Hockings PD; Reid DG
    NMR Biomed; 2004 Feb; 17(1):28-32. PubMed ID: 15011248
    [TBL] [Abstract][Full Text] [Related]  

  • 19. Progressive Degeneration and Inhibition of Peripheral Nerve Regeneration in the SOD1-G93A Mouse Model of Amyotrophic Lateral Sclerosis.
    Deng B; Lv W; Duan W; Liu Y; Li Z; Ma Y; Zhang G; Song X; Cui C; Qi X; Li Y; Li C
    Cell Physiol Biochem; 2018; 46(6):2358-2372. PubMed ID: 29742495
    [TBL] [Abstract][Full Text] [Related]  

  • 20. Over-expression of Hsp27 does not influence disease in the mutant SOD1(G93A) mouse model of amyotrophic lateral sclerosis.
    Krishnan J; Vannuvel K; Andries M; Waelkens E; Robberecht W; Van Den Bosch L
    J Neurochem; 2008 Sep; 106(5):2170-83. PubMed ID: 18624915
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 29.