190 related articles for article (PubMed ID: 1576739)
1. Relation between glycolysis and calcium homeostasis in postischemic myocardium.
Jeremy RW; Koretsune Y; Marban E; Becker LC
Circ Res; 1992 Jun; 70(6):1180-90. PubMed ID: 1576739
[TBL] [Abstract][Full Text] [Related]
2. The functional recovery of post-ischemic myocardium requires glycolysis during early reperfusion.
Jeremy RW; Ambrosio G; Pike MM; Jacobus WE; Becker LC
J Mol Cell Cardiol; 1993 Mar; 25(3):261-76. PubMed ID: 8510169
[TBL] [Abstract][Full Text] [Related]
3. New Na(+)-H+ exchange inhibitor HOE 694 improves postischemic function and high-energy phosphate resynthesis and reduces Ca2+ overload in isolated perfused rabbit heart.
Hendrikx M; Mubagwa K; Verdonck F; Overloop K; Van Hecke P; Vanstapel F; Van Lommel A; Verbeken E; Lauweryns J; Flameng W
Circulation; 1994 Jun; 89(6):2787-98. PubMed ID: 8205693
[TBL] [Abstract][Full Text] [Related]
4. Pyruvate dehydrogenase influences postischemic heart function.
Lewandowski ED; White LT
Circulation; 1995 Apr; 91(7):2071-9. PubMed ID: 7895366
[TBL] [Abstract][Full Text] [Related]
5. Glycolytic inhibition and calcium overload as consequences of exogenously generated free radicals in rabbit hearts.
Corretti MC; Koretsune Y; Kusuoka H; Chacko VP; Zweier JL; Marban E
J Clin Invest; 1991 Sep; 88(3):1014-25. PubMed ID: 1653271
[TBL] [Abstract][Full Text] [Related]
6. Protective effects of dimethyl amiloride against postischemic myocardial dysfunction in rabbit hearts: phosphorus 31-nuclear magnetic resonance measurements of intracellular pH and cellular energy.
Koike A; Akita T; Hotta Y; Takeya K; Kodama I; Murase M; Abe T; Toyama J
J Thorac Cardiovasc Surg; 1996 Sep; 112(3):765-75. PubMed ID: 8800166
[TBL] [Abstract][Full Text] [Related]
7. Na(+)/H(+) exchange inhibition with HOE642 improves postischemic recovery due to attenuation of Ca(2+) overload and prolonged acidosis on reperfusion.
Strömer H; de Groot MC; Horn M; Faul C; Leupold A; Morgan JP; Scholz W; Neubauer S
Circulation; 2000 Jun; 101(23):2749-55. PubMed ID: 10851214
[TBL] [Abstract][Full Text] [Related]
8. Glycolysis is necessary to preserve myocardial Ca2+ homeostasis during beta-adrenergic stimulation.
Nakamura K; Kusuoka H; Ambrosio G; Becker LC
Am J Physiol; 1993 Mar; 264(3 Pt 2):H670-8. PubMed ID: 8384419
[TBL] [Abstract][Full Text] [Related]
9. Overexpression of the cardiac Na+/Ca2+ exchanger increases susceptibility to ischemia/reperfusion injury in male, but not female, transgenic mice.
Cross HR; Lu L; Steenbergen C; Philipson KD; Murphy E
Circ Res; 1998 Dec 14-28; 83(12):1215-23. PubMed ID: 9851938
[TBL] [Abstract][Full Text] [Related]
10. Quantification of [Ca2+]i in perfused hearts. Critical evaluation of the 5F-BAPTA and nuclear magnetic resonance method as applied to the study of ischemia and reperfusion.
Marban E; Kitakaze M; Koretsune Y; Yue DT; Chacko VP; Pike MM
Circ Res; 1990 May; 66(5):1255-67. PubMed ID: 2110515
[TBL] [Abstract][Full Text] [Related]
11. Intracellular sodium accumulation during ischemia as the substrate for reperfusion injury.
Imahashi K; Kusuoka H; Hashimoto K; Yoshioka J; Yamaguchi H; Nishimura T
Circ Res; 1999 Jun; 84(12):1401-6. PubMed ID: 10381892
[TBL] [Abstract][Full Text] [Related]
12. Expression of SERCA isoform with faster Ca2+ transport properties improves postischemic cardiac function and Ca2+ handling and decreases myocardial infarction.
Talukder MA; Kalyanasundaram A; Zhao X; Zuo L; Bhupathy P; Babu GJ; Cardounel AJ; Periasamy M; Zweier JL
Am J Physiol Heart Circ Physiol; 2007 Oct; 293(4):H2418-28. PubMed ID: 17630344
[TBL] [Abstract][Full Text] [Related]
13. Administration of fructose 1,6-diphosphate during early reperfusion significantly improves recovery of contractile function in the postischemic heart.
Takeuchi K; Cao-Danh H; Friehs I; Glynn P; D'Agostino D; Simplaceanu E; McGowan FX; del Nido PJ
J Thorac Cardiovasc Surg; 1998 Aug; 116(2):335-43. PubMed ID: 9699588
[TBL] [Abstract][Full Text] [Related]
14. Mechanism of preconditioning. Ionic alterations.
Steenbergen C; Perlman ME; London RE; Murphy E
Circ Res; 1993 Jan; 72(1):112-25. PubMed ID: 8380259
[TBL] [Abstract][Full Text] [Related]
15. Effect of aging on intracellular Ca2+, pHi, and contractility during ischemia and reperfusion.
Ataka K; Chen D; Levitsky S; Jimenez E; Feinberg H
Circulation; 1992 Nov; 86(5 Suppl):II371-6. PubMed ID: 1424026
[TBL] [Abstract][Full Text] [Related]
16. Substrate competition in postischemic myocardium. Effect of substrate availability during reperfusion on metabolic and contractile recovery in isolated rat hearts.
Tamm C; Benzi R; Papageorgiou I; Tardy I; Lerch R
Circ Res; 1994 Dec; 75(6):1103-12. PubMed ID: 7955147
[TBL] [Abstract][Full Text] [Related]
17. Low-dose calcium antagonists reduce energy demand and cellular damage of isolated hearts during both ischemia and reperfusion.
Becker BF; Möbert J
Naunyn Schmiedebergs Arch Pharmacol; 1999 Sep; 360(3):287-94. PubMed ID: 10543430
[TBL] [Abstract][Full Text] [Related]
18. Effects of TA-3090, a new calcium channel blocker, on myocardial substrate utilization in ischemic and nonischemic isolated working fatty acid-perfused rat hearts.
Davies NJ; McVeigh JJ; Lopaschuk GD
Circ Res; 1991 Mar; 68(3):807-17. PubMed ID: 1742868
[TBL] [Abstract][Full Text] [Related]
19. Soluble complement receptor type 1 inhibits the complement pathway and prevents contractile failure in the postischemic heart. Evidence that complement activation is required for neutrophil-mediated reperfusion injury.
Shandelya SM; Kuppusamy P; Herskowitz A; Weisfeldt ML; Zweier JL
Circulation; 1993 Dec; 88(6):2812-26. PubMed ID: 8252695
[TBL] [Abstract][Full Text] [Related]
20. R56865, a Na(+)- and Ca(2+)-overload inhibitor, reduces myocardial ischemia-reperfusion injury in blood-perfused rabbit hearts.
Chen CC; Morishige N; Masuda M; Lin W; Wieland W; Thoné F; Mubagwa K; Borgers M; Flameng W
J Mol Cell Cardiol; 1993 Dec; 25(12):1445-59. PubMed ID: 8158664
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
