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8. Role of p300-family proteins in E1A oncogene induction of cytolytic susceptibility and tumor cell rejection. Cook JL; Krantz CK; Routes BA Proc Natl Acad Sci U S A; 1996 Nov; 93(24):13985-90. PubMed ID: 8943047 [TBL] [Abstract][Full Text] [Related]
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13. E1A gene expression induces susceptibility to killing by NK cells following immortalization but not adenovirus infection of human cells. Routes JM; Cook JL Virology; 1995 Jul; 210(2):421-8. PubMed ID: 7618277 [TBL] [Abstract][Full Text] [Related]
14. E1A second exon requirements for induction of target cell susceptibility to lysis by natural killer cells: implications for the mechanism of action. Krantz CK; Routes BA; Quinlan MP; Cook JL Virology; 1996 Mar; 217(1):23-32. PubMed ID: 8599207 [TBL] [Abstract][Full Text] [Related]
15. E1A oncogene expression in target cells induces cytolytic susceptibility at a post-recognition stage in the interaction with killer lymphocytes. Cook JL; Potter TA; Bellgrau D; Routes BA Oncogene; 1996 Aug; 13(4):833-42. PubMed ID: 8761305 [TBL] [Abstract][Full Text] [Related]
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17. p202, an interferon-inducible negative regulator of cell growth, is a target of the adenovirus E1A protein. Xin H; D'Souza S; Fang L; Lengyel P; Choubey D Oncogene; 2001 Oct; 20(47):6828-39. PubMed ID: 11687962 [TBL] [Abstract][Full Text] [Related]
18. Dissimilar immunogenicities of human papillomavirus E7 and adenovirus E1A proteins influence primary tumor development. Routes JM; Ryan S; Li H; Steinke J; Cook JL Virology; 2000 Nov; 277(1):48-57. PubMed ID: 11062035 [TBL] [Abstract][Full Text] [Related]
19. Role of the E1A Rb-binding domain in repression of the NF-kappa B-dependent defense against tumor necrosis factor-alpha. Cook JL; Walker TA; Worthen GS; Radke JR Proc Natl Acad Sci U S A; 2002 Jul; 99(15):9966-71. PubMed ID: 12119420 [TBL] [Abstract][Full Text] [Related]
20. Adenovirus genes that modulate the sensitivity of virus-infected cells to lysis by TNF. Wold WS J Cell Biochem; 1993 Dec; 53(4):329-35. PubMed ID: 8300750 [TBL] [Abstract][Full Text] [Related] [Next] [New Search]