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6. Cardiolipin remodeling: a regulatory hub for modulating cardiolipin metabolism and function. Ye C; Shen Z; Greenberg ML J Bioenerg Biomembr; 2016 Apr; 48(2):113-23. PubMed ID: 25432572 [TBL] [Abstract][Full Text] [Related]
7. Cardiolipin and monolysocardiolipin analysis in fibroblasts, lymphocytes, and tissues using high-performance liquid chromatography-mass spectrometry as a diagnostic test for Barth syndrome. Houtkooper RH; Rodenburg RJ; Thiels C; van Lenthe H; Stet F; Poll-The BT; Stone JE; Steward CG; Wanders RJ; Smeitink J; Kulik W; Vaz FM Anal Biochem; 2009 Apr; 387(2):230-7. PubMed ID: 19454236 [TBL] [Abstract][Full Text] [Related]
8. Only one splice variant of the human TAZ gene encodes a functional protein with a role in cardiolipin metabolism. Vaz FM; Houtkooper RH; Valianpour F; Barth PG; Wanders RJ J Biol Chem; 2003 Oct; 278(44):43089-94. PubMed ID: 12930833 [TBL] [Abstract][Full Text] [Related]
9. Aberrant cardiolipin metabolism is associated with cognitive deficiency and hippocampal alteration in tafazzin knockdown mice. Cole LK; Kim JH; Amoscato AA; Tyurina YY; Bay R H; Karimi B; Siddiqui TJ; Kagan VE; Hatch GM; Kauppinen TM Biochim Biophys Acta Mol Basis Dis; 2018 Oct; 1864(10):3353-3367. PubMed ID: 30055293 [TBL] [Abstract][Full Text] [Related]
10. Loss of tafazzin results in decreased myoblast differentiation in C2C12 cells: A myoblast model of Barth syndrome and cardiolipin deficiency. Lou W; Reynolds CA; Li Y; Liu J; Hüttemann M; Schlame M; Stevenson D; Strathdee D; Greenberg ML Biochim Biophys Acta Mol Cell Biol Lipids; 2018 Aug; 1863(8):857-865. PubMed ID: 29694924 [TBL] [Abstract][Full Text] [Related]
11. Promotion of plasmalogen biosynthesis reverse lipid changes in a Barth Syndrome cell model. Bozelli JC; Lu D; Atilla-Gokcumen GE; Epand RM Biochim Biophys Acta Mol Cell Biol Lipids; 2020 Jun; 1865(6):158677. PubMed ID: 32126285 [TBL] [Abstract][Full Text] [Related]
12. Proapoptotic Bid binds to monolysocardiolipin, a new molecular connection between mitochondrial membranes and cell death. Esposti MD; Cristea IM; Gaskell SJ; Nakao Y; Dive C Cell Death Differ; 2003 Dec; 10(12):1300-9. PubMed ID: 12894218 [TBL] [Abstract][Full Text] [Related]
13. Anomalous peroxidase activity of cytochrome c is the primary pathogenic target in Barth syndrome. Kagan VE; Tyurina YY; Mikulska-Ruminska K; Damschroder D; Vieira Neto E; Lasorsa A; Kapralov AA; Tyurin VA; Amoscato AA; Samovich SN; Souryavong AB; Dar HH; Ramim A; Liang Z; Lazcano P; Ji J; Schmidtke MW; Kiselyov K; Korkmaz A; Vladimirov GK; Artyukhova MA; Rampratap P; Cole LK; Niyatie A; Baker EK; Peterson J; Hatch GM; Atkinson J; Vockley J; Kühn B; Wessells R; van der Wel PCA; Bahar I; Bayir H; Greenberg ML Nat Metab; 2023 Dec; 5(12):2184-2205. PubMed ID: 37996701 [TBL] [Abstract][Full Text] [Related]
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18. An improved functional assay in blood spot to diagnose Barth syndrome using the monolysocardiolipin/cardiolipin ratio. Vaz FM; van Lenthe H; Vervaart MAT; Stet FS; Klinkspoor JH; Vernon HJ; Goorden SMI; Houtkooper RH; Kulik W; Wanders RJA J Inherit Metab Dis; 2022 Jan; 45(1):29-37. PubMed ID: 34382226 [TBL] [Abstract][Full Text] [Related]
19. The interaction between tBid and cardiolipin or monolysocardiolipin. Liu J; Durrant D; Yang HS; He Y; Whitby FG; Myszka DG; Lee RM Biochem Biophys Res Commun; 2005 May; 330(3):865-70. PubMed ID: 15809076 [TBL] [Abstract][Full Text] [Related]
20. Bid-cardiolipin interaction at mitochondrial contact site contributes to mitochondrial cristae reorganization and cytochrome C release. Kim TH; Zhao Y; Ding WX; Shin JN; He X; Seo YW; Chen J; Rabinowich H; Amoscato AA; Yin XM Mol Biol Cell; 2004 Jul; 15(7):3061-72. PubMed ID: 15107464 [TBL] [Abstract][Full Text] [Related] [Next] [New Search]