BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

490 related articles for article (PubMed ID: 15983830)

  • 1. Redox system expression in the motor neurons in amyotrophic lateral sclerosis (ALS): immunohistochemical studies on sporadic ALS, superoxide dismutase 1 (SOD1)-mutated familial ALS, and SOD1-mutated ALS animal models.
    Kato S; Kato M; Abe Y; Matsumura T; Nishino T; Aoki M; Itoyama Y; Asayama K; Awaya A; Hirano A; Ohama E
    Acta Neuropathol; 2005 Aug; 110(2):101-12. PubMed ID: 15983830
    [TBL] [Abstract][Full Text] [Related]  

  • 2. Expression of hepatocyte growth factor and c-Met in the anterior horn cells of the spinal cord in the patients with amyotrophic lateral sclerosis (ALS): immunohistochemical studies on sporadic ALS and familial ALS with superoxide dismutase 1 gene mutation.
    Kato S; Funakoshi H; Nakamura T; Kato M; Nakano I; Hirano A; Ohama E
    Acta Neuropathol; 2003 Aug; 106(2):112-20. PubMed ID: 12707786
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Histological evidence of redox system breakdown caused by superoxide dismutase 1 (SOD1) aggregation is common to SOD1-mutated motor neurons in humans and animal models.
    Kato S; Saeki Y; Aoki M; Nagai M; Ishigaki A; Itoyama Y; Kato M; Asayama K; Awaya A; Hirano A; Ohama E
    Acta Neuropathol; 2004 Feb; 107(2):149-58. PubMed ID: 14648077
    [TBL] [Abstract][Full Text] [Related]  

  • 4. Histological recovery of the hepatocytes is based on the redox system upregulation in the animal models of mutant superoxide dismutase (SOD)1-linked amyotrophic lateral sclerosis.
    Kato M; Kato S; Abe Y; Nishino T; Ohama E; Aoki M; Itoyama Y
    Histol Histopathol; 2006 Jul; 21(7):729-42. PubMed ID: 16598672
    [TBL] [Abstract][Full Text] [Related]  

  • 5. TDP-43 immunoreactivity in neuronal inclusions in familial amyotrophic lateral sclerosis with or without SOD1 gene mutation.
    Tan CF; Eguchi H; Tagawa A; Onodera O; Iwasaki T; Tsujino A; Nishizawa M; Kakita A; Takahashi H
    Acta Neuropathol; 2007 May; 113(5):535-42. PubMed ID: 17333220
    [TBL] [Abstract][Full Text] [Related]  

  • 6. Protein-bound crotonaldehyde accumulates in the spinal cord of superoxide dismutase-1 mutation-associated familial amyotrophic lateral sclerosis and its transgenic mouse model.
    Shibata N; Kawaguchi M; Uchida K; Kakita A; Takahashi H; Nakano R; Fujimura H; Sakoda S; Ihara Y; Nobukuni K; Takehisa Y; Kuroda S; Kokubo Y; Kuzuhara S; Honma T; Mochizuki Y; Mizutani T; Yamada S; Toi S; Sasaki S; Iwata M; Hirano A; Yamamoto T; Kato Y; Sawada T; Kobayashi M
    Neuropathology; 2007 Feb; 27(1):49-61. PubMed ID: 17319283
    [TBL] [Abstract][Full Text] [Related]  

  • 7. HtrA2/Omi-immunoreactive intraneuronal inclusions in the anterior horn of patients with sporadic and Cu/Zn superoxide dismutase (SOD1) mutant amyotrophic lateral sclerosis.
    Kawamoto Y; Ito H; Kobayashi Y; Suzuki Y; Akiguchi I; Fujimura H; Sakoda S; Kusaka H; Hirano A; Takahashi R
    Neuropathol Appl Neurobiol; 2010 Jun; 36(4):331-44. PubMed ID: 20202124
    [TBL] [Abstract][Full Text] [Related]  

  • 8. FUS-immunoreactive inclusions are a common feature in sporadic and non-SOD1 familial amyotrophic lateral sclerosis.
    Deng HX; Zhai H; Bigio EH; Yan J; Fecto F; Ajroud K; Mishra M; Ajroud-Driss S; Heller S; Sufit R; Siddique N; Mugnaini E; Siddique T
    Ann Neurol; 2010 Jun; 67(6):739-48. PubMed ID: 20517935
    [TBL] [Abstract][Full Text] [Related]  

  • 9. TDP-43 is consistently co-localized with ubiquitinated inclusions in sporadic and Guam amyotrophic lateral sclerosis but not in familial amyotrophic lateral sclerosis with and without SOD1 mutations.
    Maekawa S; Leigh PN; King A; Jones E; Steele JC; Bodi I; Shaw CE; Hortobagyi T; Al-Sarraj S
    Neuropathology; 2009 Dec; 29(6):672-83. PubMed ID: 19496940
    [TBL] [Abstract][Full Text] [Related]  

  • 10. Colocalization of 14-3-3 proteins with SOD1 in Lewy body-like hyaline inclusions in familial amyotrophic lateral sclerosis cases and the animal model.
    Okamoto Y; Shirakashi Y; Ihara M; Urushitani M; Oono M; Kawamoto Y; Yamashita H; Shimohama S; Kato S; Hirano A; Tomimoto H; Ito H; Takahashi R
    PLoS One; 2011; 6(5):e20427. PubMed ID: 21655264
    [TBL] [Abstract][Full Text] [Related]  

  • 11. S100A6 overexpression within astrocytes associated with impaired axons from both ALS mouse model and human patients.
    Hoyaux D; Boom A; Van den Bosch L; Belot N; Martin JJ; Heizmann CW; Kiss R; Pochet R
    J Neuropathol Exp Neurol; 2002 Aug; 61(8):736-44. PubMed ID: 12152788
    [TBL] [Abstract][Full Text] [Related]  

  • 12. Delayed disease onset and extended survival in the SOD1G93A rat model of amyotrophic lateral sclerosis after suppression of mutant SOD1 in the motor cortex.
    Thomsen GM; Gowing G; Latter J; Chen M; Vit JP; Staggenborg K; Avalos P; Alkaslasi M; Ferraiuolo L; Likhite S; Kaspar BK; Svendsen CN
    J Neurosci; 2014 Nov; 34(47):15587-600. PubMed ID: 25411487
    [TBL] [Abstract][Full Text] [Related]  

  • 13. Recent advances in research on neuropathological aspects of familial amyotrophic lateral sclerosis with superoxide dismutase 1 gene mutations: neuronal Lewy body-like hyaline inclusions and astrocytic hyaline inclusions.
    Kato S; Saito M; Hirano A; Ohama E
    Histol Histopathol; 1999 Jul; 14(3):973-89. PubMed ID: 10425565
    [TBL] [Abstract][Full Text] [Related]  

  • 14. Human Cu/Zn superoxide dismutase (SOD1) overexpression in mice causes mitochondrial vacuolization, axonal degeneration, and premature motoneuron death and accelerates motoneuron disease in mice expressing a familial amyotrophic lateral sclerosis mutant SOD1.
    Jaarsma D; Haasdijk ED; Grashorn JA; Hawkins R; van Duijn W; Verspaget HW; London J; Holstege JC
    Neurobiol Dis; 2000 Dec; 7(6 Pt B):623-43. PubMed ID: 11114261
    [TBL] [Abstract][Full Text] [Related]  

  • 15. The small heat shock protein B8 (HspB8) promotes autophagic removal of misfolded proteins involved in amyotrophic lateral sclerosis (ALS).
    Crippa V; Sau D; Rusmini P; Boncoraglio A; Onesto E; Bolzoni E; Galbiati M; Fontana E; Marino M; Carra S; Bendotti C; De Biasi S; Poletti A
    Hum Mol Genet; 2010 Sep; 19(17):3440-56. PubMed ID: 20570967
    [TBL] [Abstract][Full Text] [Related]  

  • 16. Phosphorylated Smad2/3 immunoreactivity in sporadic and familial amyotrophic lateral sclerosis and its mouse model.
    Nakamura M; Ito H; Wate R; Nakano S; Hirano A; Kusaka H
    Acta Neuropathol; 2008 Mar; 115(3):327-34. PubMed ID: 18210139
    [TBL] [Abstract][Full Text] [Related]  

  • 17. Aberrant localization of FUS and TDP43 is associated with misfolding of SOD1 in amyotrophic lateral sclerosis.
    Pokrishevsky E; Grad LI; Yousefi M; Wang J; Mackenzie IR; Cashman NR
    PLoS One; 2012; 7(4):e35050. PubMed ID: 22493728
    [TBL] [Abstract][Full Text] [Related]  

  • 18. Pathological Modification of TDP-43 in Amyotrophic Lateral Sclerosis with SOD1 Mutations.
    Jeon GS; Shim YM; Lee DY; Kim JS; Kang M; Ahn SH; Shin JY; Geum D; Hong YH; Sung JJ
    Mol Neurobiol; 2019 Mar; 56(3):2007-2021. PubMed ID: 29982983
    [TBL] [Abstract][Full Text] [Related]  

  • 19. Bromocriptine methylate suppresses glial inflammation and moderates disease progression in a mouse model of amyotrophic lateral sclerosis.
    Tanaka K; Kanno T; Yanagisawa Y; Yasutake K; Hadano S; Yoshii F; Ikeda JE
    Exp Neurol; 2011 Nov; 232(1):41-52. PubMed ID: 21867702
    [TBL] [Abstract][Full Text] [Related]  

  • 20. Pathological TDP-43 distinguishes sporadic amyotrophic lateral sclerosis from amyotrophic lateral sclerosis with SOD1 mutations.
    Mackenzie IR; Bigio EH; Ince PG; Geser F; Neumann M; Cairns NJ; Kwong LK; Forman MS; Ravits J; Stewart H; Eisen A; McClusky L; Kretzschmar HA; Monoranu CM; Highley JR; Kirby J; Siddique T; Shaw PJ; Lee VM; Trojanowski JQ
    Ann Neurol; 2007 May; 61(5):427-34. PubMed ID: 17469116
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 25.