306 related articles for article (PubMed ID: 16208517)
1. Correction of cellular phenotypes of Hutchinson-Gilford Progeria cells by RNA interference.
Huang S; Chen L; Libina N; Janes J; Martin GM; Campisi J; Oshima J
Hum Genet; 2005 Dec; 118(3-4):444-50. PubMed ID: 16208517
[TBL] [Abstract][Full Text] [Related]
2. Dermal fibroblasts in Hutchinson-Gilford progeria syndrome with the lamin A G608G mutation have dysmorphic nuclei and are hypersensitive to heat stress.
Paradisi M; McClintock D; Boguslavsky RL; Pedicelli C; Worman HJ; Djabali K
BMC Cell Biol; 2005 Jun; 6():27. PubMed ID: 15982412
[TBL] [Abstract][Full Text] [Related]
3. Incomplete processing of mutant lamin A in Hutchinson-Gilford progeria leads to nuclear abnormalities, which are reversed by farnesyltransferase inhibition.
Glynn MW; Glover TW
Hum Mol Genet; 2005 Oct; 14(20):2959-69. PubMed ID: 16126733
[TBL] [Abstract][Full Text] [Related]
4. Reversal of the cellular phenotype in the premature aging disease Hutchinson-Gilford progeria syndrome.
Scaffidi P; Misteli T
Nat Med; 2005 Apr; 11(4):440-5. PubMed ID: 15750600
[TBL] [Abstract][Full Text] [Related]
5. Hutchinson-Gilford progeria syndrome through the lens of transcription.
Prokocimer M; Barkan R; Gruenbaum Y
Aging Cell; 2013 Aug; 12(4):533-43. PubMed ID: 23496208
[TBL] [Abstract][Full Text] [Related]
6. Identification of novel RNA isoforms of LMNA.
DeBoy E; Puttaraju M; Jailwala P; Kasoji M; Cam M; Misteli T
Nucleus; 2017 Sep; 8(5):573-582. PubMed ID: 28857661
[TBL] [Abstract][Full Text] [Related]
7. Eliminating the synthesis of mature lamin A reduces disease phenotypes in mice carrying a Hutchinson-Gilford progeria syndrome allele.
Yang SH; Qiao X; Farber E; Chang SY; Fong LG; Young SG
J Biol Chem; 2008 Mar; 283(11):7094-9. PubMed ID: 18178963
[TBL] [Abstract][Full Text] [Related]
8. Hutchinson-Gilford progeria mutant lamin A primarily targets human vascular cells as detected by an anti-Lamin A G608G antibody.
McClintock D; Gordon LB; Djabali K
Proc Natl Acad Sci U S A; 2006 Feb; 103(7):2154-9. PubMed ID: 16461887
[TBL] [Abstract][Full Text] [Related]
9. Vitamin D receptor signaling improves Hutchinson-Gilford progeria syndrome cellular phenotypes.
Kreienkamp R; Croke M; Neumann MA; Bedia-Diaz G; Graziano S; Dusso A; Dorsett D; Carlberg C; Gonzalo S
Oncotarget; 2016 May; 7(21):30018-31. PubMed ID: 27145372
[TBL] [Abstract][Full Text] [Related]
10. Epigenetic involvement in Hutchinson-Gilford progeria syndrome: a mini-review.
Arancio W; Pizzolanti G; Genovese SI; Pitrone M; Giordano C
Gerontology; 2014; 60(3):197-203. PubMed ID: 24603298
[TBL] [Abstract][Full Text] [Related]
11. Targeted transgenic expression of the mutation causing Hutchinson-Gilford progeria syndrome leads to proliferative and degenerative epidermal disease.
Sagelius H; Rosengardten Y; Hanif M; Erdos MR; Rozell B; Collins FS; Eriksson M
J Cell Sci; 2008 Apr; 121(Pt 7):969-78. PubMed ID: 18334552
[TBL] [Abstract][Full Text] [Related]
12. In vivo base editing rescues Hutchinson-Gilford progeria syndrome in mice.
Koblan LW; Erdos MR; Wilson C; Cabral WA; Levy JM; Xiong ZM; Tavarez UL; Davison LM; Gete YG; Mao X; Newby GA; Doherty SP; Narisu N; Sheng Q; Krilow C; Lin CY; Gordon LB; Cao K; Collins FS; Brown JD; Liu DR
Nature; 2021 Jan; 589(7843):608-614. PubMed ID: 33408413
[TBL] [Abstract][Full Text] [Related]
13. Increased expression of the Hutchinson-Gilford progeria syndrome truncated lamin A transcript during cell aging.
Rodriguez S; Coppedè F; Sagelius H; Eriksson M
Eur J Hum Genet; 2009 Jul; 17(7):928-37. PubMed ID: 19172989
[TBL] [Abstract][Full Text] [Related]
14. Accumulation of mutant lamin A causes progressive changes in nuclear architecture in Hutchinson-Gilford progeria syndrome.
Goldman RD; Shumaker DK; Erdos MR; Eriksson M; Goldman AE; Gordon LB; Gruenbaum Y; Khuon S; Mendez M; Varga R; Collins FS
Proc Natl Acad Sci U S A; 2004 Jun; 101(24):8963-8. PubMed ID: 15184648
[TBL] [Abstract][Full Text] [Related]
15. A progeroid syndrome in mice is caused by defects in A-type lamins.
Mounkes LC; Kozlov S; Hernandez L; Sullivan T; Stewart CL
Nature; 2003 May; 423(6937):298-301. PubMed ID: 12748643
[TBL] [Abstract][Full Text] [Related]
16. Accelerated telomere shortening and replicative senescence in human fibroblasts overexpressing mutant and wild-type lamin A.
Huang S; Risques RA; Martin GM; Rabinovitch PS; Oshima J
Exp Cell Res; 2008 Jan; 314(1):82-91. PubMed ID: 17870066
[TBL] [Abstract][Full Text] [Related]
17. Aging of Hutchinson-Gilford progeria syndrome fibroblasts is characterised by hyperproliferation and increased apoptosis.
Bridger JM; Kill IR
Exp Gerontol; 2004 May; 39(5):717-24. PubMed ID: 15130666
[TBL] [Abstract][Full Text] [Related]
18. LMNA-associated cardiocutaneous progeria: an inherited autosomal dominant premature aging syndrome with late onset.
Kane MS; Lindsay ME; Judge DP; Barrowman J; Ap Rhys C; Simonson L; Dietz HC; Michaelis S
Am J Med Genet A; 2013 Jul; 161A(7):1599-611. PubMed ID: 23666920
[TBL] [Abstract][Full Text] [Related]
19. A conserved splicing mechanism of the LMNA gene controls premature aging.
Lopez-Mejia IC; Vautrot V; De Toledo M; Behm-Ansmant I; Bourgeois CF; Navarro CL; Osorio FG; Freije JM; Stévenin J; De Sandre-Giovannoli A; Lopez-Otin C; Lévy N; Branlant C; Tazi J
Hum Mol Genet; 2011 Dec; 20(23):4540-55. PubMed ID: 21875900
[TBL] [Abstract][Full Text] [Related]
20. Genome-scale expression profiling of Hutchinson-Gilford progeria syndrome reveals widespread transcriptional misregulation leading to mesodermal/mesenchymal defects and accelerated atherosclerosis.
Csoka AB; English SB; Simkevich CP; Ginzinger DG; Butte AJ; Schatten GP; Rothman FG; Sedivy JM
Aging Cell; 2004 Aug; 3(4):235-43. PubMed ID: 15268757
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
