301 related articles for article (PubMed ID: 16217030)
1. Gap junction-mediated intercellular biochemical coupling in cochlear supporting cells is required for normal cochlear functions.
Zhang Y; Tang W; Ahmad S; Sipp JA; Chen P; Lin X
Proc Natl Acad Sci U S A; 2005 Oct; 102(42):15201-6. PubMed ID: 16217030
[TBL] [Abstract][Full Text] [Related]
2. Gap junction mediated intercellular metabolite transfer in the cochlea is compromised in connexin30 null mice.
Chang Q; Tang W; Ahmad S; Zhou B; Lin X
PLoS One; 2008; 3(12):e4088. PubMed ID: 19116647
[TBL] [Abstract][Full Text] [Related]
3. Cochlear gap junctions coassembled from Cx26 and 30 show faster intercellular Ca2+ signaling than homomeric counterparts.
Sun J; Ahmad S; Chen S; Tang W; Zhang Y; Chen P; Lin X
Am J Physiol Cell Physiol; 2005 Mar; 288(3):C613-23. PubMed ID: 15692151
[TBL] [Abstract][Full Text] [Related]
4. A deafness mechanism of digenic Cx26 (GJB2) and Cx30 (GJB6) mutations: Reduction of endocochlear potential by impairment of heterogeneous gap junctional function in the cochlear lateral wall.
Mei L; Chen J; Zong L; Zhu Y; Liang C; Jones RO; Zhao HB
Neurobiol Dis; 2017 Dec; 108():195-203. PubMed ID: 28823936
[TBL] [Abstract][Full Text] [Related]
5. Functional studies reveal new mechanisms for deafness caused by connexin mutations.
Chang Q; Tang W; Ahmad S; Stong B; Leu G; Lin X
Otol Neurotol; 2009 Feb; 30(2):237-40. PubMed ID: 19169135
[TBL] [Abstract][Full Text] [Related]
6. The human deafness-associated connexin 30 T5M mutation causes mild hearing loss and reduces biochemical coupling among cochlear non-sensory cells in knock-in mice.
Schütz M; Scimemi P; Majumder P; De Siati RD; Crispino G; Rodriguez L; Bortolozzi M; Santarelli R; Seydel A; Sonntag S; Ingham N; Steel KP; Willecke K; Mammano F
Hum Mol Genet; 2010 Dec; 19(24):4759-73. PubMed ID: 20858605
[TBL] [Abstract][Full Text] [Related]
7. Connexin26 gap junction mediates miRNA intercellular genetic communication in the cochlea and is required for inner ear development.
Zhu Y; Zong L; Mei L; Zhao HB
Sci Rep; 2015 Oct; 5():15647. PubMed ID: 26490746
[TBL] [Abstract][Full Text] [Related]
8. Analysis of connexin subunits required for the survival of vestibular hair cells.
Qu Y; Tang W; Dahlke I; Ding D; Salvi R; Söhl G; Willecke K; Chen P; Lin X
J Comp Neurol; 2007 Oct; 504(5):499-507. PubMed ID: 17702002
[TBL] [Abstract][Full Text] [Related]
9. Early developmental expression of connexin26 in the cochlea contributes to its dominate functional role in the cochlear gap junctions.
Qu Y; Tang W; Zhou B; Ahmad S; Chang Q; Li X; Lin X
Biochem Biophys Res Commun; 2012 Jan; 417(1):245-50. PubMed ID: 22142852
[TBL] [Abstract][Full Text] [Related]
10. Deficiency of transcription factor Brn4 disrupts cochlear gap junction plaques in a model of DFN3 non-syndromic deafness.
Kidokoro Y; Karasawa K; Minowa O; Sugitani Y; Noda T; Ikeda K; Kamiya K
PLoS One; 2014; 9(9):e108216. PubMed ID: 25259580
[TBL] [Abstract][Full Text] [Related]
11. Timed conditional null of connexin26 in mice reveals temporary requirements of connexin26 in key cochlear developmental events before the onset of hearing.
Chang Q; Tang W; Kim Y; Lin X
Neurobiol Dis; 2015 Jan; 73():418-27. PubMed ID: 25251605
[TBL] [Abstract][Full Text] [Related]
12. Impaired permeability to Ins(1,4,5)P3 in a mutant connexin underlies recessive hereditary deafness.
Beltramello M; Piazza V; Bukauskas FF; Pozzan T; Mammano F
Nat Cell Biol; 2005 Jan; 7(1):63-9. PubMed ID: 15592461
[TBL] [Abstract][Full Text] [Related]
13. Unique expression of connexins in the human cochlea.
Liu W; Boström M; Kinnefors A; Rask-Andersen H
Hear Res; 2009 Apr; 250(1-2):55-62. PubMed ID: 19450429
[TBL] [Abstract][Full Text] [Related]
14. Functional analysis of connexin-26 mutants associated with hereditary recessive deafness.
Wang HL; Chang WT; Li AH; Yeh TH; Wu CY; Chen MS; Huang PC
J Neurochem; 2003 Feb; 84(4):735-42. PubMed ID: 12562518
[TBL] [Abstract][Full Text] [Related]
15. Gap junctions in the inner ear: comparison of distribution patterns in different vertebrates and assessement of connexin composition in mammals.
Forge A; Becker D; Casalotti S; Edwards J; Marziano N; Nevill G
J Comp Neurol; 2003 Dec; 467(2):207-31. PubMed ID: 14595769
[TBL] [Abstract][Full Text] [Related]
16. Connexin26 is responsible for anionic molecule permeability in the cochlea for intercellular signalling and metabolic communications.
Zhao HB
Eur J Neurosci; 2005 Apr; 21(7):1859-68. PubMed ID: 15869481
[TBL] [Abstract][Full Text] [Related]
17. Virally expressed connexin26 restores gap junction function in the cochlea of conditional Gjb2 knockout mice.
Yu Q; Wang Y; Chang Q; Wang J; Gong S; Li H; Lin X
Gene Ther; 2014 Jan; 21(1):71-80. PubMed ID: 24225640
[TBL] [Abstract][Full Text] [Related]
18. Cisplatin-induced ototoxicity in organotypic cochlear cultures occurs independent of gap junctional intercellular communication.
Abitbol J; Beach R; Barr K; Esseltine J; Allman B; Laird D
Cell Death Dis; 2020 May; 11(5):342. PubMed ID: 32393745
[TBL] [Abstract][Full Text] [Related]
19. Compartmentalized and signal-selective gap junctional coupling in the hearing cochlea.
Jagger DJ; Forge A
J Neurosci; 2006 Jan; 26(4):1260-8. PubMed ID: 16436613
[TBL] [Abstract][Full Text] [Related]
20. Restoration of connexin26 protein level in the cochlea completely rescues hearing in a mouse model of human connexin30-linked deafness.
Ahmad S; Tang W; Chang Q; Qu Y; Hibshman J; Li Y; Söhl G; Willecke K; Chen P; Lin X
Proc Natl Acad Sci U S A; 2007 Jan; 104(4):1337-41. PubMed ID: 17227867
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]