146 related articles for article (PubMed ID: 16481432)
1. Connexin29 is highly expressed in cochlear Schwann cells, and it is required for the normal development and function of the auditory nerve of mice.
Tang W; Zhang Y; Chang Q; Ahmad S; Dahlke I; Yi H; Chen P; Paul DL; Lin X
J Neurosci; 2006 Feb; 26(7):1991-9. PubMed ID: 16481432
[TBL] [Abstract][Full Text] [Related]
2. Expression pattern and functional characterization of connexin29 in transgenic mice.
Eiberger J; Kibschull M; Strenzke N; Schober A; Büssow H; Wessig C; Djahed S; Reucher H; Koch DA; Lautermann J; Moser T; Winterhager E; Willecke K
Glia; 2006 Apr; 53(6):601-11. PubMed ID: 16435366
[TBL] [Abstract][Full Text] [Related]
3. Analysis of connexin expression during mouse Schwann cell development identifies connexin29 as a novel marker for the transition of neural crest to precursor cells.
Li J; Habbes HW; Eiberger J; Willecke K; Dermietzel R; Meier C
Glia; 2007 Jan; 55(1):93-103. PubMed ID: 17024657
[TBL] [Abstract][Full Text] [Related]
4. Connexin29 and connexin32 at oligodendrocyte and astrocyte gap junctions and in myelin of the mouse central nervous system.
Nagy JI; Ionescu AV; Lynn BD; Rash JE
J Comp Neurol; 2003 Sep; 464(3):356-70. PubMed ID: 12900929
[TBL] [Abstract][Full Text] [Related]
5. Connexin29 expression, immunocytochemistry and freeze-fracture replica immunogold labelling (FRIL) in sciatic nerve.
Li X; Lynn BD; Olson C; Meier C; Davidson KG; Yasumura T; Rash JE; Nagy JI
Eur J Neurosci; 2002 Sep; 16(5):795-806. PubMed ID: 12372015
[TBL] [Abstract][Full Text] [Related]
6. Connexin29 is uniquely distributed within myelinating glial cells of the central and peripheral nervous systems.
Altevogt BM; Kleopa KA; Postma FR; Scherer SS; Paul DL
J Neurosci; 2002 Aug; 22(15):6458-70. PubMed ID: 12151525
[TBL] [Abstract][Full Text] [Related]
7. Expression patterns of connexin 29 (GJE1) in mouse and rat cochlea.
Yang JJ; Liao PJ; Su CC; Li SY
Biochem Biophys Res Commun; 2005 Dec; 338(2):723-8. PubMed ID: 16236250
[TBL] [Abstract][Full Text] [Related]
8. Early developmental expression of connexin26 in the cochlea contributes to its dominate functional role in the cochlear gap junctions.
Qu Y; Tang W; Zhou B; Ahmad S; Chang Q; Li X; Lin X
Biochem Biophys Res Commun; 2012 Jan; 417(1):245-50. PubMed ID: 22142852
[TBL] [Abstract][Full Text] [Related]
9. A deafness mechanism of digenic Cx26 (GJB2) and Cx30 (GJB6) mutations: Reduction of endocochlear potential by impairment of heterogeneous gap junctional function in the cochlear lateral wall.
Mei L; Chen J; Zong L; Zhu Y; Liang C; Jones RO; Zhao HB
Neurobiol Dis; 2017 Dec; 108():195-203. PubMed ID: 28823936
[TBL] [Abstract][Full Text] [Related]
10. Cx29 and Cx32, two connexins expressed by myelinating glia, do not interact and are functionally distinct.
Ahn M; Lee J; Gustafsson A; Enriquez A; Lancaster E; Sul JY; Haydon PG; Paul DL; Huang Y; Abrams CK; Scherer SS
J Neurosci Res; 2008 Apr; 86(5):992-1006. PubMed ID: 17972320
[TBL] [Abstract][Full Text] [Related]
11. Cochlear gap junctions coassembled from Cx26 and 30 show faster intercellular Ca2+ signaling than homomeric counterparts.
Sun J; Ahmad S; Chen S; Tang W; Zhang Y; Chen P; Lin X
Am J Physiol Cell Physiol; 2005 Mar; 288(3):C613-23. PubMed ID: 15692151
[TBL] [Abstract][Full Text] [Related]
12. Timed conditional null of connexin26 in mice reveals temporary requirements of connexin26 in key cochlear developmental events before the onset of hearing.
Chang Q; Tang W; Kim Y; Lin X
Neurobiol Dis; 2015 Jan; 73():418-27. PubMed ID: 25251605
[TBL] [Abstract][Full Text] [Related]
13. Functional studies reveal new mechanisms for deafness caused by connexin mutations.
Chang Q; Tang W; Ahmad S; Stong B; Leu G; Lin X
Otol Neurotol; 2009 Feb; 30(2):237-40. PubMed ID: 19169135
[TBL] [Abstract][Full Text] [Related]
14. Pannexin 1 deficiency can induce hearing loss.
Zhao HB; Zhu Y; Liang C; Chen J
Biochem Biophys Res Commun; 2015 Jul 17-24; 463(1-2):143-7. PubMed ID: 26002464
[TBL] [Abstract][Full Text] [Related]
15. Cell degeneration is not a primary causer for Connexin26 (GJB2) deficiency associated hearing loss.
Liang C; Zhu Y; Zong L; Lu GJ; Zhao HB
Neurosci Lett; 2012 Oct; 528(1):36-41. PubMed ID: 22975134
[TBL] [Abstract][Full Text] [Related]
16. Gap junction disorders of myelinating cells.
Kleopa KA; Orthmann-Murphy J; Sargiannidou I
Rev Neurosci; 2010; 21(5):397-419. PubMed ID: 21280457
[TBL] [Abstract][Full Text] [Related]
17. Connexin30 null and conditional connexin26 null mice display distinct pattern and time course of cellular degeneration in the cochlea.
Sun Y; Tang W; Chang Q; Wang Y; Kong W; Lin X
J Comp Neurol; 2009 Oct; 516(6):569-79. PubMed ID: 19673007
[TBL] [Abstract][Full Text] [Related]
18. Schwann cells revert to non-myelinating phenotypes in the deafened rat cochlea.
Hurley PA; Crook JM; Shepherd RK
Eur J Neurosci; 2007 Oct; 26(7):1813-21. PubMed ID: 17868369
[TBL] [Abstract][Full Text] [Related]
19. Analysis of connexin subunits required for the survival of vestibular hair cells.
Qu Y; Tang W; Dahlke I; Ding D; Salvi R; Söhl G; Willecke K; Chen P; Lin X
J Comp Neurol; 2007 Oct; 504(5):499-507. PubMed ID: 17702002
[TBL] [Abstract][Full Text] [Related]
20. Progressive age-dependence and frequency difference in the effect of gap junctions on active cochlear amplification and hearing.
Zong L; Chen J; Zhu Y; Zhao HB
Biochem Biophys Res Commun; 2017 Jul; 489(2):223-227. PubMed ID: 28552523
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
