123 related articles for article (PubMed ID: 1649081)
1. The effect of free radicals induced by paraquat and copper on the in vitro development of Plasmodium falciparum.
Marva E; Chevion M; Golenser J
Free Radic Res Commun; 1991; 12-13 Pt 1():137-46. PubMed ID: 1649081
[TBL] [Abstract][Full Text] [Related]
2. Deleterious synergistic effects of ascorbate and copper on the development of Plasmodium falciparum: an in vitro study in normal and in G6PD-deficient erythrocytes.
Marva E; Cohen A; Saltman P; Chevion M; Golenser J
Int J Parasitol; 1989 Nov; 19(7):779-85. PubMed ID: 2687184
[TBL] [Abstract][Full Text] [Related]
3. Synergistic enhancement of a copper chelator, bathocuproine disulphonate, and cysteine on in vitro growth of Plasmodium falciparum in glucose-6-phosphate dehydrogenase-deficient erythrocytes.
Tantular IS; Jalloh A; Pusarawati S; Azuno Y; Lin K; Kerong H; Dachlan YP; Ishii A; Kawamoto F
Southeast Asian J Trop Med Public Health; 2003 Jun; 34(2):301-9. PubMed ID: 12971554
[TBL] [Abstract][Full Text] [Related]
4. Susceptibility to hydrogen peroxide of Plasmodium falciparum infecting glucose-6-phosphate dehydrogenase-deficient erythrocytes.
Kamchonwongpaisan S; Bunyaratvej A; Wanachiwanawin W; Yuthavong Y
Parasitology; 1989 Oct; 99 Pt 2():171-4. PubMed ID: 2687776
[TBL] [Abstract][Full Text] [Related]
5. Inhibition of the intraerythrocytic development of Plasmodium falciparum in glucose-6-phosphate dehydrogenase deficient erythrocytes is enhanced by oxidants and by crisis form factor.
Golenser J; Miller J; Spira DT; Kosower NS; Vande Waa JA; Jensen JB
Trop Med Parasitol; 1988 Dec; 39(4):273-6. PubMed ID: 3067318
[TBL] [Abstract][Full Text] [Related]
6. Early phagocytosis of glucose-6-phosphate dehydrogenase (G6PD)-deficient erythrocytes parasitized by Plasmodium falciparum may explain malaria protection in G6PD deficiency.
Cappadoro M; Giribaldi G; O'Brien E; Turrini F; Mannu F; Ulliers D; Simula G; Luzzatto L; Arese P
Blood; 1998 Oct; 92(7):2527-34. PubMed ID: 9746794
[TBL] [Abstract][Full Text] [Related]
7. Plasmodium falciparum: thiol status and growth in normal and glucose-6-phosphate dehydrogenase deficient human erythrocytes.
Miller J; Golenser J; Spira DT; Kosower NS
Exp Parasitol; 1984 Jun; 57(3):239-47. PubMed ID: 6373352
[TBL] [Abstract][Full Text] [Related]
8. Redox metabolism in glucose-6-phosphate dehydrogenase deficient erythrocytes and its relation to antimalarial chemotherapy.
Ginsburg H; Golenser J
Parassitologia; 1999 Sep; 41(1-3):309-11. PubMed ID: 10697873
[TBL] [Abstract][Full Text] [Related]
9. The adaptation of Plasmodium falciparum to oxidative stress in G6PD deficient human erythrocytes.
Roth E; Schulman S
Br J Haematol; 1988 Nov; 70(3):363-7. PubMed ID: 3061444
[TBL] [Abstract][Full Text] [Related]
10. The effect of purified aminoaldehydes produced by polyamine oxidation on the development in vitro of Plasmodium falciparum in normal and glucose-6-phosphate-dehydrogenase-deficient erythrocytes.
Morgan DM; Bachrach U; Assaraf YG; Harari E; Golenser J
Biochem J; 1986 May; 236(1):97-101. PubMed ID: 3539102
[TBL] [Abstract][Full Text] [Related]
11. Iron mediates paraquat toxicity in Escherichia coli.
Korbashi P; Kohen R; Katzhendler J; Chevion M
J Biol Chem; 1986 Sep; 261(27):12472-6. PubMed ID: 3017976
[TBL] [Abstract][Full Text] [Related]
12. Biosynthesis of superoxide dismutase in Saccharomyces cerevisiae: effects of paraquat and copper.
Lee FJ; Hassan HM
J Free Radic Biol Med; 1985; 1(4):319-25. PubMed ID: 3013982
[TBL] [Abstract][Full Text] [Related]
13. Transition metals potentiate paraquat toxicity.
Kohen R; Chevion M
Free Radic Res Commun; 1985; 1(2):79-88. PubMed ID: 3916875
[TBL] [Abstract][Full Text] [Related]
14. Plasmodium falciparum glutathione metabolism and growth are independent of glutathione system of host erythrocyte.
Ayi K; Cappadoro M; Branca M; Turrini F; Arese P
FEBS Lett; 1998 Mar; 424(3):257-61. PubMed ID: 9539162
[TBL] [Abstract][Full Text] [Related]
15. Membrane protein carbonylation of Plasmodium falciparum infected erythrocytes under conditions of sickle cell trait and G6PD deficiency.
Contreras-Puentes N; Rodríguez-Cavallo E; Méndez-Cuadro D
Mol Biochem Parasitol; 2019 Jan; 227():5-14. PubMed ID: 30472238
[TBL] [Abstract][Full Text] [Related]
16. The effect and mechanism of inhibiting glucose-6-phosphate dehydrogenase activity on the proliferation of Plasmodium falciparum.
Zhang Z; Chen X; Jiang C; Fang Z; Feng Y; Jiang W
Biochim Biophys Acta Mol Cell Res; 2017 May; 1864(5):771-781. PubMed ID: 28214533
[TBL] [Abstract][Full Text] [Related]
17. Ribose metabolism and nucleic acid synthesis in normal and glucose-6-phosphate dehydrogenase-deficient human erythrocytes infected with Plasmodium falciparum.
Roth EF; Ruprecht RM; Schulman S; Vanderberg J; Olson JA
J Clin Invest; 1986 Apr; 77(4):1129-35. PubMed ID: 2420826
[TBL] [Abstract][Full Text] [Related]
18. Inhibitory effect of a fava bean component on the in vitro development of Plasmodium falciparum in normal and glucose-6-phosphate dehydrogenase deficient erythrocytes.
Golenser J; Miller J; Spira DT; Navok T; Chevion M
Blood; 1983 Mar; 61(3):507-10. PubMed ID: 6337653
[TBL] [Abstract][Full Text] [Related]
19. 14C-desferrioxamine B: uptake into erythrocytes infected with Plasmodium falciparum.
Fritsch G; Jung A
Z Parasitenkd; 1986; 72(6):709-13. PubMed ID: 3541423
[TBL] [Abstract][Full Text] [Related]
20. Copper pathways in Plasmodium falciparum infected erythrocytes indicate an efflux role for the copper P-ATPase.
Rasoloson D; Shi L; Chong CR; Kafsack BF; Sullivan DJ
Biochem J; 2004 Aug; 381(Pt 3):803-11. PubMed ID: 15125686
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]