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5. Hypertension does not account for the accelerated atherosclerosis and development of aneurysms in male apolipoprotein e/endothelial nitric oxide synthase double knockout mice. Chen J; Kuhlencordt PJ; Astern J; Gyurko R; Huang PL Circulation; 2001 Nov; 104(20):2391-4. PubMed ID: 11705813 [TBL] [Abstract][Full Text] [Related]
6. TSLPR deficiency attenuates atherosclerotic lesion development associated with the inhibition of TH17 cells and the promotion of regulator T cells in ApoE-deficient mice. Wu C; He S; Peng Y; Kushwaha KK; Lin J; Dong J; Wang B; Lin J; Shan S; Liu J; Huang K; Li D J Mol Cell Cardiol; 2014 Nov; 76():33-45. PubMed ID: 25117469 [TBL] [Abstract][Full Text] [Related]
7. Effects of chronic treatment with L-arginine on atherosclerosis in apoE knockout and apoE/inducible NO synthase double-knockout mice. Chen J; Kuhlencordt P; Urano F; Ichinose H; Astern J; Huang PL Arterioscler Thromb Vasc Biol; 2003 Jan; 23(1):97-103. PubMed ID: 12524231 [TBL] [Abstract][Full Text] [Related]
8. Decreased production of neuronal NOS-derived hydrogen peroxide contributes to endothelial dysfunction in atherosclerosis. Capettini LS; Cortes SF; Silva JF; Alvarez-Leite JI; Lemos VS Br J Pharmacol; 2011 Nov; 164(6):1738-48. PubMed ID: 21615722 [TBL] [Abstract][Full Text] [Related]
9. Chronic intermittent hypoxia exposure induces atherosclerosis in ApoE knockout mice: role of NF-κB p50. Fang G; Song D; Ye X; Mao SZ; Liu G; Liu SF Am J Pathol; 2012 Nov; 181(5):1530-9. PubMed ID: 22940439 [TBL] [Abstract][Full Text] [Related]
10. nNOS splice variants differentially regulate myofilament function but are dispensable for intracellular calcium and force transients in cardiac papillary muscles. Kerrick WGL; Xu Y; Percival JM PLoS One; 2018; 13(7):e0200834. PubMed ID: 30028847 [TBL] [Abstract][Full Text] [Related]
11. Elderly apolipoprotein E‑/‑ mice with advanced atherosclerotic lesions in the aorta do not develop Alzheimer's disease-like pathologies. Shnerb Ganor R; Harats D; Schiby G; Rosenblatt K; Lubitz I; Shaish A; Salomon O Mol Med Rep; 2018 Feb; 17(2):2488-2492. PubMed ID: 29207114 [TBL] [Abstract][Full Text] [Related]
12. A₁ adenosine receptor deficiency or inhibition reduces atherosclerotic lesions in apolipoprotein E deficient mice. Teng B; Smith JD; Rosenfeld ME; Robinet P; Davis ME; Morrison RR; Mustafa SJ Cardiovasc Res; 2014 Apr; 102(1):157-65. PubMed ID: 24525840 [TBL] [Abstract][Full Text] [Related]
14. Attenuated atherosclerotic lesions in apoE-Fcγ-chain-deficient hyperlipidemic mouse model is associated with inhibition of Th17 cells and promotion of regulatory T cells. Ng HP; Burris RL; Nagarajan S J Immunol; 2011 Dec; 187(11):6082-93. PubMed ID: 22043015 [TBL] [Abstract][Full Text] [Related]
15. Urotensin II receptor knockout mice on an ApoE knockout background fed a high-fat diet exhibit an enhanced hyperlipidemic and atherosclerotic phenotype. Bousette N; D'Orleans-Juste P; Kiss RS; You Z; Genest J; Al-Ramli W; Qureshi ST; Gramolini A; Behm D; Ohlstein EH; Harrison SM; Douglas SA; Giaid A Circ Res; 2009 Sep; 105(7):686-95, 19 p following 695. PubMed ID: 19696412 [TBL] [Abstract][Full Text] [Related]
16. Ablation of the bach1 gene leads to the suppression of atherosclerosis in bach1 and apolipoprotein E double knockout mice. Watari Y; Yamamoto Y; Brydun A; Ishida T; Mito S; Yoshizumi M; Igarashi K; Chayama K; Ohshima T; Ozono R Hypertens Res; 2008 Apr; 31(4):783-92. PubMed ID: 18633191 [TBL] [Abstract][Full Text] [Related]
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20. Overexpression of endothelial nitric oxide synthase accelerates atherosclerotic lesion formation in apoE-deficient mice. Ozaki M; Kawashima S; Yamashita T; Hirase T; Namiki M; Inoue N; Hirata K; Yasui H; Sakurai H; Yoshida Y; Masada M; Yokoyama M J Clin Invest; 2002 Aug; 110(3):331-40. PubMed ID: 12163452 [TBL] [Abstract][Full Text] [Related] [Next] [New Search]