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4. Molecular genetic basis for inherited human disorders of branched-chain alpha-keto acid dehydrogenase complex. Danner DJ; Litwer S; Herring WJ; Elsas LJ Ann N Y Acad Sci; 1989; 573():369-77. PubMed ID: 2699404 [No Abstract] [Full Text] [Related]
5. Diagnosis and mutational analysis of maple syrup urine disease using cell cultures. Chuang JL; Chuang DT Methods Enzymol; 2000; 324():413-23. PubMed ID: 10989449 [No Abstract] [Full Text] [Related]
6. Complementation analysis in lymphoid cells from five patients with different forms of maple syrup urine disease. Jinno Y; Akaboshi I; Matsuda I Hum Genet; 1984; 68(1):54-6. PubMed ID: 6500555 [TBL] [Abstract][Full Text] [Related]
7. Maple syrup urine disease 1954 to 1993. Peinemann F; Danner DJ J Inherit Metab Dis; 1994; 17(1):3-15. PubMed ID: 8051937 [No Abstract] [Full Text] [Related]
8. Genetic defects in E3 component of alpha-keto acid dehydrogenase complexes. Patel MS; Hong YS; Kerr DS Methods Enzymol; 2000; 324():453-64. PubMed ID: 10989452 [No Abstract] [Full Text] [Related]
9. Targeting E3 component of alpha-keto acid dehydrogenase complexes. Johnson MT; Yang HS; Patel MS Methods Enzymol; 2000; 324():465-76. PubMed ID: 10989453 [No Abstract] [Full Text] [Related]
10. Maple syrup urine disease. Complete defect of the E1 beta subunit of the branched chain alpha-ketoacid dehydrogenase complex due to a deletion of an 11-bp repeat sequence which encodes a mitochondrial targeting leader peptide in a family with the disease. Nobukuni Y; Mitsubuchi H; Akaboshi I; Indo Y; Endo F; Yoshioka A; Matsuda I J Clin Invest; 1991 May; 87(5):1862-6. PubMed ID: 2022752 [TBL] [Abstract][Full Text] [Related]
11. Maple syrup urine disease: the E1beta gene of human branched-chain alpha-ketoacid dehydrogenase complex has 11 rather than 10 exons, and the 3' UTR in one of the two E1beta mRNAs arises from intronic sequences. Chuang JL; Cox RP; Chuang DT Am J Hum Genet; 1996 Jun; 58(6):1373-7. PubMed ID: 8651316 [No Abstract] [Full Text] [Related]
12. Maple syrup urine disease caused by a partial deletion in the inner E2 core domain of the branched chain alpha-keto acid dehydrogenase complex due to aberrant splicing. A single base deletion at a 5'-splice donor site of an intron of the E2 gene disrupts the consensus sequence in this region. Mitsubuchi H; Nobukuni Y; Akaboshi I; Indo Y; Endo F; Matsuda I J Clin Invest; 1991 Apr; 87(4):1207-11. PubMed ID: 2010537 [TBL] [Abstract][Full Text] [Related]
13. Molecular heterogeneity for bovine maple syrup urine disease. Healy PJ; Dennis JA Anim Genet; 1994 Oct; 25(5):329-32. PubMed ID: 7818167 [TBL] [Abstract][Full Text] [Related]
14. Maple syrup urine disease: domain structure, mutations and exon skipping in the dihydrolipoyl transacylase (E2) component of the branched-chain alpha-keto acid dehydrogenase complex. Chuang DT; Fisher CW; Lau KS; Griffin TA; Wynn RM; Cox RP Mol Biol Med; 1991 Feb; 8(1):49-63. PubMed ID: 1943690 [TBL] [Abstract][Full Text] [Related]
15. A nonsense mutation (R242X) in the branched-chain alpha-keto acid dehydrogenase E1alpha subunit gene (BCKDHA) as a cause of maple syrup urine disease. Mutations in brief no. 160. Online. Chinsky J; Appel M; Almashanu S; Costeas P; Ambulos N; Carmi R Hum Mutat; 1998; 12(2):136. PubMed ID: 10694918 [TBL] [Abstract][Full Text] [Related]
16. Molecular genetic characterization of maple syrup urine disease in European families. Peinemann F; Wendel U; Danner DJ Biochem Med Metab Biol; 1993 Dec; 50(3):338-45. PubMed ID: 8123297 [TBL] [Abstract][Full Text] [Related]
17. Heterogeneity of mutations in maple syrup urine disease (MSUD): screening and identification of affected E1 alpha and E1 beta subunits of the branched-chain alpha-keto-acid dehydrogenase multienzyme complex. Nobukuni Y; Mitsubuchi H; Hayashida Y; Ohta K; Indo Y; Ichiba Y; Endo F; Matsuda I Biochim Biophys Acta; 1993 Nov; 1225(1):64-70. PubMed ID: 8161368 [TBL] [Abstract][Full Text] [Related]
18. Occurrence of a 2-bp (AT) deletion allele and a nonsense (G-to-T) mutant allele at the E2 (DBT) locus of six patients with maple syrup urine disease: multiple-exon skipping as a secondary effect of the mutations. Fisher CW; Fisher CR; Chuang JL; Lau KS; Chuang DT; Cox RP Am J Hum Genet; 1993 Feb; 52(2):414-24. PubMed ID: 8430702 [TBL] [Abstract][Full Text] [Related]