387 related articles for article (PubMed ID: 16924481)
1. Hyperinsulinism in mice with heterozygous loss of K(ATP) channels.
Remedi MS; Rocheleau JV; Tong A; Patton BL; McDaniel ML; Piston DW; Koster JC; Nichols CG
Diabetologia; 2006 Oct; 49(10):2368-78. PubMed ID: 16924481
[TBL] [Abstract][Full Text] [Related]
2. Carriers of an inactivating beta-cell ATP-sensitive K(+) channel mutation have normal glucose tolerance and insulin sensitivity and appropriate insulin secretion.
Huopio H; Vauhkonen I; Komulainen J; Niskanen L; Otonkoski T; Laakso M
Diabetes Care; 2002 Jan; 25(1):101-6. PubMed ID: 11772909
[TBL] [Abstract][Full Text] [Related]
3. beta-cell hyperexcitability: from hyperinsulinism to diabetes.
Nichols CG; Koster JC; Remedi MS
Diabetes Obes Metab; 2007 Nov; 9 Suppl 2():81-8. PubMed ID: 17919182
[TBL] [Abstract][Full Text] [Related]
4. Mutations in the genes encoding the pancreatic beta-cell KATP channel subunits Kir6.2 (KCNJ11) and SUR1 (ABCC8) in diabetes mellitus and hyperinsulinism.
Gloyn AL; Siddiqui J; Ellard S
Hum Mutat; 2006 Mar; 27(3):220-31. PubMed ID: 16416420
[TBL] [Abstract][Full Text] [Related]
5. Crosstalk between membrane potential and cytosolic Ca2+ concentration in beta cells from Sur1-/- mice.
Haspel D; Krippeit-Drews P; Aguilar-Bryan L; Bryan J; Drews G; Düfer M
Diabetologia; 2005 May; 48(5):913-21. PubMed ID: 15830184
[TBL] [Abstract][Full Text] [Related]
6. Congenital hyperinsulinism associated ABCC8 mutations that cause defective trafficking of ATP-sensitive K+ channels: identification and rescue.
Yan FF; Lin YW; MacMullen C; Ganguly A; Stanley CA; Shyng SL
Diabetes; 2007 Sep; 56(9):2339-48. PubMed ID: 17575084
[TBL] [Abstract][Full Text] [Related]
7. Genotype-phenotype correlations in children with congenital hyperinsulinism due to recessive mutations of the adenosine triphosphate-sensitive potassium channel genes.
Henwood MJ; Kelly A; Macmullen C; Bhatia P; Ganguly A; Thornton PS; Stanley CA
J Clin Endocrinol Metab; 2005 Feb; 90(2):789-94. PubMed ID: 15562009
[TBL] [Abstract][Full Text] [Related]
8. Defective trafficking and function of KATP channels caused by a sulfonylurea receptor 1 mutation associated with persistent hyperinsulinemic hypoglycemia of infancy.
Cartier EA; Conti LR; Vandenberg CA; Shyng SL
Proc Natl Acad Sci U S A; 2001 Feb; 98(5):2882-7. PubMed ID: 11226335
[TBL] [Abstract][Full Text] [Related]
9. Molecular mechanisms of neonatal hyperinsulinism.
Giurgea I; Bellanné-Chantelot C; Ribeiro M; Hubert L; Sempoux C; Robert JJ; Blankenstein O; Hussain K; Brunelle F; Nihoul-Fékété C; Rahier J; Jaubert F; de Lonlay P
Horm Res; 2006; 66(6):289-96. PubMed ID: 17003566
[TBL] [Abstract][Full Text] [Related]
10. ABCC8 (SUR1) and KCNJ11 (KIR6.2) mutations in persistent hyperinsulinemic hypoglycemia of infancy and evaluation of different therapeutic measures.
Darendeliler F; Fournet JC; Baş F; Junien C; Gross MS; Bundak R; Saka N; Günöz H
J Pediatr Endocrinol Metab; 2002; 15(7):993-1000. PubMed ID: 12199344
[TBL] [Abstract][Full Text] [Related]
11. Destabilization of ATP-sensitive potassium channel activity by novel KCNJ11 mutations identified in congenital hyperinsulinism.
Lin YW; Bushman JD; Yan FF; Haidar S; MacMullen C; Ganguly A; Stanley CA; Shyng SL
J Biol Chem; 2008 Apr; 283(14):9146-56. PubMed ID: 18250167
[TBL] [Abstract][Full Text] [Related]
12. Update of mutations in the genes encoding the pancreatic beta-cell K(ATP) channel subunits Kir6.2 (KCNJ11) and sulfonylurea receptor 1 (ABCC8) in diabetes mellitus and hyperinsulinism.
Flanagan SE; Clauin S; Bellanné-Chantelot C; de Lonlay P; Harries LW; Gloyn AL; Ellard S
Hum Mutat; 2009 Feb; 30(2):170-80. PubMed ID: 18767144
[TBL] [Abstract][Full Text] [Related]
13. Regulated expression of adenosine triphosphate-sensitive potassium channel subunits in pancreatic beta-cells.
Moritz W; Leech CA; Ferrer J; Habener JF
Endocrinology; 2001 Jan; 142(1):129-38. PubMed ID: 11145575
[TBL] [Abstract][Full Text] [Related]
14. Diverse roles of K(ATP) channels learned from Kir6.2 genetically engineered mice.
Seino S; Iwanaga T; Nagashima K; Miki T
Diabetes; 2000 Mar; 49(3):311-8. PubMed ID: 10868950
[TBL] [Abstract][Full Text] [Related]
15. Restitution of defective glucose-stimulated insulin release of sulfonylurea type 1 receptor knockout mice by acetylcholine.
Doliba NM; Qin W; Vatamaniuk MZ; Li C; Zelent D; Najafi H; Buettger CW; Collins HW; Carr RD; Magnuson MA; Matschinsky FM
Am J Physiol Endocrinol Metab; 2004 May; 286(5):E834-43. PubMed ID: 14736703
[TBL] [Abstract][Full Text] [Related]
16. Impact of Sur1 gene inactivation on the morphology of mouse pancreatic endocrine tissue.
Marhfour I; Moulin P; Marchandise J; Rahier J; Sempoux C; Guiot Y
Cell Tissue Res; 2009 Mar; 335(3):505-15. PubMed ID: 19142666
[TBL] [Abstract][Full Text] [Related]
17. Regulation of glucagon secretion at low glucose concentrations: evidence for adenosine triphosphate-sensitive potassium channel involvement.
Muñoz A; Hu M; Hussain K; Bryan J; Aguilar-Bryan L; Rajan AS
Endocrinology; 2005 Dec; 146(12):5514-21. PubMed ID: 16123162
[TBL] [Abstract][Full Text] [Related]
18. Sulfonylurea receptor 1 mutations that cause opposite insulin secretion defects with chemical chaperone exposure.
Pratt EB; Yan FF; Gay JW; Stanley CA; Shyng SL
J Biol Chem; 2009 Mar; 284(12):7951-9. PubMed ID: 19151370
[TBL] [Abstract][Full Text] [Related]
19. The structure and function of the ATP-sensitive K+ channel in insulin-secreting pancreatic beta-cells.
Miki T; Nagashima K; Seino S
J Mol Endocrinol; 1999 Apr; 22(2):113-23. PubMed ID: 10194514
[TBL] [Abstract][Full Text] [Related]
20. Clinical and molecular characterization of a dominant form of congenital hyperinsulinism caused by a mutation in the high-affinity sulfonylurea receptor.
Thornton PS; MacMullen C; Ganguly A; Ruchelli E; Steinkrauss L; Crane A; Aguilar-Bryan L; Stanley CA
Diabetes; 2003 Sep; 52(9):2403-10. PubMed ID: 12941782
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
