BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

416 related articles for article (PubMed ID: 16939419)

  • 1. Copper binding to the N-terminal metal-binding sites or the CPC motif is not essential for copper-induced trafficking of the human Wilson protein (ATP7B).
    Cater MA; La Fontaine S; Mercer JF
    Biochem J; 2007 Jan; 401(1):143-53. PubMed ID: 16939419
    [TBL] [Abstract][Full Text] [Related]  

  • 2. Intracellular trafficking of the human Wilson protein: the role of the six N-terminal metal-binding sites.
    Cater MA; Forbes J; La Fontaine S; Cox D; Mercer JF
    Biochem J; 2004 Jun; 380(Pt 3):805-13. PubMed ID: 14998371
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Molecular events initiating exit of a copper-transporting ATPase ATP7B from the trans-Golgi network.
    Hasan NM; Gupta A; Polishchuk E; Yu CH; Polishchuk R; Dmitriev OY; Lutsenko S
    J Biol Chem; 2012 Oct; 287(43):36041-50. PubMed ID: 22898812
    [TBL] [Abstract][Full Text] [Related]  

  • 4. Communication between the N and C termini is required for copper-stimulated Ser/Thr phosphorylation of Cu(I)-ATPase (ATP7B).
    Braiterman LT; Gupta A; Chaerkady R; Cole RN; Hubbard AL
    J Biol Chem; 2015 Apr; 290(14):8803-19. PubMed ID: 25666620
    [TBL] [Abstract][Full Text] [Related]  

  • 5. Sequence variation in the ATP-binding domain of the Wilson disease transporter, ATP7B, affects copper transport in a yeast model system.
    Hsi G; Cullen LM; Macintyre G; Chen MM; Glerum DM; Cox DW
    Hum Mutat; 2008 Apr; 29(4):491-501. PubMed ID: 18203200
    [TBL] [Abstract][Full Text] [Related]  

  • 6. Critical roles for the COOH terminus of the Cu-ATPase ATP7B in protein stability, trans-Golgi network retention, copper sensing, and retrograde trafficking.
    Braiterman L; Nyasae L; Leves F; Hubbard AL
    Am J Physiol Gastrointest Liver Physiol; 2011 Jul; 301(1):G69-81. PubMed ID: 21454443
    [TBL] [Abstract][Full Text] [Related]  

  • 7. Functional characterization of missense mutations in ATP7B: Wilson disease mutation or normal variant?
    Forbes JR; Cox DW
    Am J Hum Genet; 1998 Dec; 63(6):1663-74. PubMed ID: 9837819
    [TBL] [Abstract][Full Text] [Related]  

  • 8. Hepatic copper-transporting ATPase ATP7B: function and inactivation at the molecular and cellular level.
    Bartee MY; Lutsenko S
    Biometals; 2007 Jun; 20(3-4):627-37. PubMed ID: 17268820
    [TBL] [Abstract][Full Text] [Related]  

  • 9. Distinct phenotype of a Wilson disease mutation reveals a novel trafficking determinant in the copper transporter ATP7B.
    Braiterman LT; Murthy A; Jayakanthan S; Nyasae L; Tzeng E; Gromadzka G; Woolf TB; Lutsenko S; Hubbard AL
    Proc Natl Acad Sci U S A; 2014 Apr; 111(14):E1364-73. PubMed ID: 24706876
    [TBL] [Abstract][Full Text] [Related]  

  • 10. Copper-dependent interaction of dynactin subunit p62 with the N terminus of ATP7B but not ATP7A.
    Lim CM; Cater MA; Mercer JF; La Fontaine S
    J Biol Chem; 2006 May; 281(20):14006-14. PubMed ID: 16554302
    [TBL] [Abstract][Full Text] [Related]  

  • 11. The role of the invariant His-1069 in folding and function of the Wilson's disease protein, the human copper-transporting ATPase ATP7B.
    Tsivkovskii R; Efremov RG; Lutsenko S
    J Biol Chem; 2003 Apr; 278(15):13302-8. PubMed ID: 12551905
    [TBL] [Abstract][Full Text] [Related]  

  • 12. Distinct Wilson's disease mutations in ATP7B are associated with enhanced binding to COMMD1 and reduced stability of ATP7B.
    de Bie P; van de Sluis B; Burstein E; van de Berghe PV; Muller P; Berger R; Gitlin JD; Wijmenga C; Klomp LW
    Gastroenterology; 2007 Oct; 133(4):1316-26. PubMed ID: 17919502
    [TBL] [Abstract][Full Text] [Related]  

  • 13. Interactions between copper-binding sites determine the redox status and conformation of the regulatory N-terminal domain of ATP7B.
    LeShane ES; Shinde U; Walker JM; Barry AN; Blackburn NJ; Ralle M; Lutsenko S
    J Biol Chem; 2010 Feb; 285(9):6327-36. PubMed ID: 20032459
    [TBL] [Abstract][Full Text] [Related]  

  • 14. ATP7B mediates vesicular sequestration of copper: insight into biliary copper excretion.
    Cater MA; La Fontaine S; Shield K; Deal Y; Mercer JF
    Gastroenterology; 2006 Feb; 130(2):493-506. PubMed ID: 16472602
    [TBL] [Abstract][Full Text] [Related]  

  • 15. Polarized trafficking and copper transport activity of ATP7B: A mutational approach to establish genotype-phenotype correlation in Wilson disease.
    Das S; Mohammed A; Mandal T; Maji S; Verma J; Ruturaj ; Gupta A
    Hum Mutat; 2022 Oct; 43(10):1408-1429. PubMed ID: 35762218
    [TBL] [Abstract][Full Text] [Related]  

  • 16. Apical targeting and Golgi retention signals reside within a 9-amino acid sequence in the copper-ATPase, ATP7B.
    Braiterman L; Nyasae L; Guo Y; Bustos R; Lutsenko S; Hubbard A
    Am J Physiol Gastrointest Liver Physiol; 2009 Feb; 296(2):G433-44. PubMed ID: 19033537
    [TBL] [Abstract][Full Text] [Related]  

  • 17. Defective cellular localization of mutant ATP7B in Wilson's disease patients and hepatoma cell lines.
    Huster D; Hoppert M; Lutsenko S; Zinke J; Lehmann C; Mössner J; Berr F; Caca K
    Gastroenterology; 2003 Feb; 124(2):335-45. PubMed ID: 12557139
    [TBL] [Abstract][Full Text] [Related]  

  • 18. Role of the copper-binding domain in the copper transport function of ATP7B, the P-type ATPase defective in Wilson disease.
    Forbes JR; Hsi G; Cox DW
    J Biol Chem; 1999 Apr; 274(18):12408-13. PubMed ID: 10212214
    [TBL] [Abstract][Full Text] [Related]  

  • 19. Copper transport and its defect in Wilson disease: characterization of the copper-binding domain of Wilson disease ATPase.
    Sarkar B
    J Inorg Biochem; 2000 Apr; 79(1-4):187-91. PubMed ID: 10830865
    [TBL] [Abstract][Full Text] [Related]  

  • 20. Copper-induced translocation of the Wilson disease protein ATP7B independent of Murr1/COMMD1 and Rab7.
    Weiss KH; Lozoya JC; Tuma S; Gotthardt D; Reichert J; Ehehalt R; Stremmel W; Füllekrug J
    Am J Pathol; 2008 Dec; 173(6):1783-94. PubMed ID: 18974300
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 21.