149 related articles for article (PubMed ID: 16940437)
1. Transgenic expression of beta-APP in fast-twitch skeletal muscle leads to calcium dyshomeostasis and IBM-like pathology.
Moussa CE; Fu Q; Kumar P; Shtifman A; Lopez JR; Allen PD; LaFerla F; Weinberg D; Magrane J; Aprahamian T; Walsh K; Rosen KM; Querfurth HW
FASEB J; 2006 Oct; 20(12):2165-7. PubMed ID: 16940437
[TBL] [Abstract][Full Text] [Related]
2. Pathogenic accumulation of APP in fast twitch muscle of IBM patients and a transgenic model.
Sugarman MC; Kitazawa M; Baker M; Caiozzo VJ; Querfurth HW; LaFerla FM
Neurobiol Aging; 2006 Mar; 27(3):423-32. PubMed ID: 15950323
[TBL] [Abstract][Full Text] [Related]
3. Inclusion body myositis-like phenotype induced by transgenic overexpression of beta APP in skeletal muscle.
Sugarman MC; Yamasaki TR; Oddo S; Echegoyen JC; Murphy MP; Golde TE; Jannatipour M; Leissring MA; LaFerla FM
Proc Natl Acad Sci U S A; 2002 Apr; 99(9):6334-9. PubMed ID: 11972038
[TBL] [Abstract][Full Text] [Related]
4. Intracellular β-amyloid accumulation leads to age-dependent progression of Ca2+ dysregulation in skeletal muscle.
Lopez JR; Shtifman A
Muscle Nerve; 2010 Nov; 42(5):731-8. PubMed ID: 20665519
[TBL] [Abstract][Full Text] [Related]
5. Mitochondrial dysfunction in skeletal muscle of amyloid precursor protein-overexpressing mice.
Boncompagni S; Moussa CE; Levy E; Pezone MJ; Lopez JR; Protasi F; Shtifman A
J Biol Chem; 2012 Jun; 287(24):20534-44. PubMed ID: 22518836
[TBL] [Abstract][Full Text] [Related]
6. Rabbits fed cholesterol-enriched diets exhibit pathological features of inclusion body myositis.
Chen X; Ghribi O; Geiger JD
Am J Physiol Regul Integr Comp Physiol; 2008 Mar; 294(3):R829-35. PubMed ID: 18216139
[TBL] [Abstract][Full Text] [Related]
7. Immunization with amyloid-beta attenuates inclusion body myositis-like myopathology and motor impairment in a transgenic mouse model.
Kitazawa M; Vasilevko V; Cribbs DH; LaFerla FM
J Neurosci; 2009 May; 29(19):6132-41. PubMed ID: 19439591
[TBL] [Abstract][Full Text] [Related]
8. Amyloid-beta42 is preferentially accumulated in muscle fibers of patients with sporadic inclusion-body myositis.
Vattemi G; Nogalska A; King Engel W; D'Agostino C; Checler F; Askanas V
Acta Neuropathol; 2009 May; 117(5):569-74. PubMed ID: 19280202
[TBL] [Abstract][Full Text] [Related]
9. Amyloid-β protein impairs Ca2+ release and contractility in skeletal muscle.
Shtifman A; Ward CW; Laver DR; Bannister ML; Lopez JR; Kitazawa M; LaFerla FM; Ikemoto N; Querfurth HW
Neurobiol Aging; 2010 Dec; 31(12):2080-90. PubMed ID: 19108934
[TBL] [Abstract][Full Text] [Related]
10. Transgenic mice over-expressing the C-99 fragment of betaPP with an alpha-secretase site mutation develop a myopathy similar to human inclusion body myositis.
Jin LW; Hearn MG; Ogburn CE; Dang N; Nochlin D; Ladiges WC; Martin GM
Am J Pathol; 1998 Dec; 153(6):1679-86. PubMed ID: 9846957
[TBL] [Abstract][Full Text] [Related]
11. Cystatin C colocalizes with amyloid-beta and coimmunoprecipitates with amyloid-beta precursor protein in sporadic inclusion-body myositis muscles.
Vattemi G; Engel WK; McFerrin J; Askanas V
J Neurochem; 2003 Jun; 85(6):1539-46. PubMed ID: 12787072
[TBL] [Abstract][Full Text] [Related]
12. Genetically augmenting Abeta42 levels in skeletal muscle exacerbates inclusion body myositis-like pathology and motor deficits in transgenic mice.
Kitazawa M; Green KN; Caccamo A; LaFerla FM
Am J Pathol; 2006 Jun; 168(6):1986-97. PubMed ID: 16723713
[TBL] [Abstract][Full Text] [Related]
13. Calcium dyshomeostasis in beta-amyloid and tau-bearing skeletal myotubes.
Christensen RA; Shtifman A; Allen PD; Lopez JR; Querfurth HW
J Biol Chem; 2004 Dec; 279(51):53524-32. PubMed ID: 15385569
[TBL] [Abstract][Full Text] [Related]
14. In AbetaPP-overexpressing cultured human muscle fibers proteasome inhibition enhances phosphorylation of AbetaPP751 and GSK3beta activation: effects mitigated by lithium and apparently relevant to sporadic inclusion-body myositis.
Terracciano C; Nogalska A; Engel WK; Askanas V
J Neurochem; 2010 Jan; 112(2):389-96. PubMed ID: 19878439
[TBL] [Abstract][Full Text] [Related]
15. Cultured inclusion-body myositis muscle fibers do not accumulate beta-amyloid precursor protein and can be innervated.
McFerrin J; Engel WK; Askanas V
Neurology; 1999 Dec; 53(9):2184-7. PubMed ID: 10599804
[TBL] [Abstract][Full Text] [Related]
16. Proteasome inhibition and aggresome formation in sporadic inclusion-body myositis and in amyloid-beta precursor protein-overexpressing cultured human muscle fibers.
Fratta P; Engel WK; McFerrin J; Davies KJ; Lin SW; Askanas V
Am J Pathol; 2005 Aug; 167(2):517-26. PubMed ID: 16049336
[TBL] [Abstract][Full Text] [Related]
17. Novel demonstration of amyloid-β oligomers in sporadic inclusion-body myositis muscle fibers.
Nogalska A; D'Agostino C; Engel WK; Klein WL; Askanas V
Acta Neuropathol; 2010 Nov; 120(5):661-6. PubMed ID: 20711838
[TBL] [Abstract][Full Text] [Related]
18. Three lipoprotein receptors and cholesterol in inclusion-body myositis muscle.
Jaworska-Wilczynska M; Wilczynski GM; Engel WK; Strickland DK; Weisgraber KH; Askanas V
Neurology; 2002 Feb; 58(3):438-45. PubMed ID: 11839845
[TBL] [Abstract][Full Text] [Related]
19. Primary over-expression of AβPP in muscle does not lead to the development of inclusion body myositis in a new lineage of the MCK-AβPP transgenic mouse.
Luo YB; Johnsen RD; Griffiths L; Needham M; Fabian VA; Fletcher S; Wilton SD; Mastaglia FL
Int J Exp Pathol; 2013 Dec; 94(6):418-25. PubMed ID: 24205796
[TBL] [Abstract][Full Text] [Related]
20. BACE1 and BACE2 in pathologic and normal human muscle.
Vattemi G; Engel WK; McFerrin J; Pastorino L; Buxbaum JD; Askanas V
Exp Neurol; 2003 Feb; 179(2):150-8. PubMed ID: 12618121
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
