133 related articles for article (PubMed ID: 16962048)
1. [Defect in lytic granule exocytosis: several causes, a same effect].
Ménasché G; Ménager M; Le Deist F; Fischer A; de Saint Basile G
Med Sci (Paris); 2006; 22(8-9):733-8. PubMed ID: 16962048
[TBL] [Abstract][Full Text] [Related]
2. Primary hemophagocytic syndromes point to a direct link between lymphocyte cytotoxicity and homeostasis.
Ménasché G; Feldmann J; Fischer A; de Saint Basile G
Immunol Rev; 2005 Feb; 203():165-79. PubMed ID: 15661029
[TBL] [Abstract][Full Text] [Related]
3. Loss of cytotoxic T lymphocyte function in Chediak-Higashi syndrome arises from a secretory defect that prevents lytic granule exocytosis.
Baetz K; Isaaz S; Griffiths GM
J Immunol; 1995 Jun; 154(11):6122-31. PubMed ID: 7751653
[TBL] [Abstract][Full Text] [Related]
4. Inherited defects causing hemophagocytic lymphohistiocytic syndrome.
de Saint Basile G; Ménasché G; Latour S
Ann N Y Acad Sci; 2011 Dec; 1246():64-76. PubMed ID: 22236431
[TBL] [Abstract][Full Text] [Related]
5. Angeborene hämophagozytische Lymphohistiozytose (HLH).
Pachlopnik Schmid J; de Saint Basile G
Klin Padiatr; 2010 Nov; 222(6):345-50. PubMed ID: 20458667
[TBL] [Abstract][Full Text] [Related]
6. Activation of primary T lymphocytes results in lysosome development and polarized granule exocytosis in CD4+ and CD8+ subsets, whereas expression of lytic molecules confers cytotoxicity to CD8+ T cells.
Shen DT; Ma JS; Mather J; Vukmanovic S; Radoja S
J Leukoc Biol; 2006 Oct; 80(4):827-37. PubMed ID: 16891618
[TBL] [Abstract][Full Text] [Related]
7. LYST controls the biogenesis of the endosomal compartment required for secretory lysosome function.
Sepulveda FE; Burgess A; Heiligenstein X; Goudin N; Ménager MM; Romao M; Côte M; Mahlaoui N; Fischer A; Raposo G; Ménasché G; de Saint Basile G
Traffic; 2015 Feb; 16(2):191-203. PubMed ID: 25425525
[TBL] [Abstract][Full Text] [Related]
8. Comparison of primary human cytotoxic T-cell and natural killer cell responses reveal similar molecular requirements for lytic granule exocytosis but differences in cytokine production.
Chiang SC; Theorell J; Entesarian M; Meeths M; Mastafa M; Al-Herz W; Frisk P; Gilmour KC; Ifversen M; Langenskiöld C; Machaczka M; Naqvi A; Payne J; Perez-Martinez A; Sabel M; Unal E; Unal S; Winiarski J; Nordenskjöld M; Ljunggren HG; Henter JI; Bryceson YT
Blood; 2013 Feb; 121(8):1345-56. PubMed ID: 23287865
[TBL] [Abstract][Full Text] [Related]
9. Calcium influx and signaling in cytotoxic T-lymphocyte lytic granule exocytosis.
Pores-Fernando AT; Zweifach A
Immunol Rev; 2009 Sep; 231(1):160-73. PubMed ID: 19754896
[TBL] [Abstract][Full Text] [Related]
10. Inherited defects in lymphocyte cytotoxic activity.
Pachlopnik Schmid J; Côte M; Ménager MM; Burgess A; Nehme N; Ménasché G; Fischer A; de Saint Basile G
Immunol Rev; 2010 May; 235(1):10-23. PubMed ID: 20536552
[TBL] [Abstract][Full Text] [Related]
11. An actin cytoskeletal barrier inhibits lytic granule release from natural killer cells in patients with Chediak-Higashi syndrome.
Gil-Krzewska A; Saeed MB; Oszmiana A; Fischer ER; Lagrue K; Gahl WA; Introne WJ; Coligan JE; Davis DM; Krzewski K
J Allergy Clin Immunol; 2018 Sep; 142(3):914-927.e6. PubMed ID: 29241728
[TBL] [Abstract][Full Text] [Related]
12. An anergic cytotoxic T lymphocyte clone exhibits granule exocytosis-mediated cytotoxicity.
Kuwano K; Akashi A; Arai S
Cell Immunol; 1998 May; 185(2):114-22. PubMed ID: 9636689
[TBL] [Abstract][Full Text] [Related]
13. The exocytosis of lytic granules is impaired in Vti1b- or Vamp8-deficient CTL leading to a reduced cytotoxic activity following antigen-specific activation.
Dressel R; Elsner L; Novota P; Kanwar N; Fischer von Mollard G
J Immunol; 2010 Jul; 185(2):1005-14. PubMed ID: 20543108
[TBL] [Abstract][Full Text] [Related]
14. Different Munc13 isoforms function as priming factors in lytic granule release from murine cytotoxic T lymphocytes.
Dudenhöffer-Pfeifer M; Schirra C; Pattu V; Halimani M; Maier-Peuschel M; Marshall MR; Matti U; Becherer U; Dirks J; Jung M; Lipp P; Hoth M; Sester M; Krause E; Rettig J
Traffic; 2013 Jul; 14(7):798-809. PubMed ID: 23590328
[TBL] [Abstract][Full Text] [Related]
15. Cytotoxic T lymphocyte exocytosis: bring on the SNAREs!
Hong W
Trends Cell Biol; 2005 Dec; 15(12):644-50. PubMed ID: 16260137
[TBL] [Abstract][Full Text] [Related]
16. Defective cytotoxic granule-mediated cell death pathway impairs T lymphocyte homeostasis.
de Saint Basile G; Fischer A
Curr Opin Rheumatol; 2003 Jul; 15(4):436-45. PubMed ID: 12819472
[TBL] [Abstract][Full Text] [Related]
17. Highly lytic in vivo primed cytolytic T lymphocytes devoid of lytic granules and BLT-esterase activity acquire these constituents in the presence of T cell growth factors upon blast transformation in vitro.
Berke G; Rosen D
J Immunol; 1988 Sep; 141(5):1429-36. PubMed ID: 3261748
[TBL] [Abstract][Full Text] [Related]
18. [Secretory lysosome disorders in the immune synapse and other tissues].
García IJ; Miñarro AG; Riestra EL; Cortés MB; Miguélez SA; Soler JL
An Pediatr (Barc); 2012 Feb; 76(2):92-7. PubMed ID: 22032885
[TBL] [Abstract][Full Text] [Related]
19. A unique SNARE machinery for exocytosis of cytotoxic granules and platelets granules.
Tang BL
Mol Membr Biol; 2015; 32(4):120-6. PubMed ID: 26508555
[TBL] [Abstract][Full Text] [Related]
20. Different NK cell-activating receptors preferentially recruit Rab27a or Munc13-4 to perforin-containing granules for cytotoxicity.
Wood SM; Meeths M; Chiang SC; Bechensteen AG; Boelens JJ; Heilmann C; Horiuchi H; Rosthøj S; Rutynowska O; Winiarski J; Stow JL; Nordenskjöld M; Henter JI; Ljunggren HG; Bryceson YT
Blood; 2009 Nov; 114(19):4117-27. PubMed ID: 19704116
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]