230 related articles for article (PubMed ID: 17059562)
1. A ubiquitin ligase HRD1 promotes the degradation of Pael receptor, a substrate of Parkin.
Omura T; Kaneko M; Okuma Y; Orba Y; Nagashima K; Takahashi R; Fujitani N; Matsumura S; Hata A; Kubota K; Murahashi K; Uehara T; Nomura Y
J Neurochem; 2006 Dec; 99(6):1456-69. PubMed ID: 17059562
[TBL] [Abstract][Full Text] [Related]
2. Immunohistochemical localization of a ubiquitin ligase HRD1 in murine brain.
Omura T; Kaneko M; Tabei N; Okuma Y; Nomura Y
J Neurosci Res; 2008 May; 86(7):1577-87. PubMed ID: 18241051
[TBL] [Abstract][Full Text] [Related]
3. Pael receptor, endoplasmic reticulum stress, and Parkinson's disease.
Takahashi R; Imai Y
J Neurol; 2003 Oct; 250 Suppl 3():III25-9. PubMed ID: 14579121
[TBL] [Abstract][Full Text] [Related]
4. [Physiological Roles of Ubiquitin Ligases Related to the Endoplasmic Reticulum].
Kaneko M
Yakugaku Zasshi; 2016; 136(6):805-9. PubMed ID: 27252059
[TBL] [Abstract][Full Text] [Related]
5. [Protective effects of HRD1 and 4-phenylbutyric acid against neuronal cell death].
Kaneko M; Nomura Y
Nihon Yakurigaku Zasshi; 2004 Dec; 124(6):391-8. PubMed ID: 15572843
[TBL] [Abstract][Full Text] [Related]
6. Novel functions of ubiquitin ligase HRD1 with transmembrane and proline-rich domains.
Omura T; Kaneko M; Onoguchi M; Koizumi S; Itami M; Ueyama M; Okuma Y; Nomura Y
J Pharmacol Sci; 2008 Mar; 106(3):512-9. PubMed ID: 18344614
[TBL] [Abstract][Full Text] [Related]
7. Pael-R transgenic mice crossed with parkin deficient mice displayed progressive and selective catecholaminergic neuronal loss.
Wang HQ; Imai Y; Inoue H; Kataoka A; Iita S; Nukina N; Takahashi R
J Neurochem; 2008 Oct; 107(1):171-85. PubMed ID: 18691389
[TBL] [Abstract][Full Text] [Related]
8. [Molecular pharmacological studies on the protection mechanism against endoplasmic reticulum stress-induced neurodegenerative disease].
Kaneko M
Yakugaku Zasshi; 2012; 132(12):1437-42. PubMed ID: 23208051
[TBL] [Abstract][Full Text] [Related]
9. Pael receptor induces death of dopaminergic neurons in the substantia nigra via endoplasmic reticulum stress and dopamine toxicity, which is enhanced under condition of parkin inactivation.
Kitao Y; Imai Y; Ozawa K; Kataoka A; Ikeda T; Soda M; Nakimawa K; Kiyama H; Stern DM; Hori O; Wakamatsu K; Ito S; Itohara S; Takahashi R; Ogawa S
Hum Mol Genet; 2007 Jan; 16(1):50-60. PubMed ID: 17116640
[TBL] [Abstract][Full Text] [Related]
10. [Neurodegeneration caused by ER stress?--the pathogenetic mechanisms underlying AR-JP].
Takahashi R
Nihon Yakurigaku Zasshi; 2004 Dec; 124(6):375-82. PubMed ID: 15572841
[TBL] [Abstract][Full Text] [Related]
11. Parkin and endoplasmic reticulum stress.
Takahashi R; Imai Y; Hattori N; Mizuno Y
Ann N Y Acad Sci; 2003 Jun; 991():101-6. PubMed ID: 12846978
[TBL] [Abstract][Full Text] [Related]
12. Pael receptor is involved in dopamine metabolism in the nigrostriatal system.
Imai Y; Inoue H; Kataoka A; Hua-Qin W; Masuda M; Ikeda T; Tsukita K; Soda M; Kodama T; Fuwa T; Honda Y; Kaneko S; Matsumoto S; Wakamatsu K; Ito S; Miura M; Aosaki T; Itohara S; Takahashi R
Neurosci Res; 2007 Dec; 59(4):413-25. PubMed ID: 17889953
[TBL] [Abstract][Full Text] [Related]
13. CHIP is associated with Parkin, a gene responsible for familial Parkinson's disease, and enhances its ubiquitin ligase activity.
Imai Y; Soda M; Hatakeyama S; Akagi T; Hashikawa T; Nakayama KI; Takahashi R
Mol Cell; 2002 Jul; 10(1):55-67. PubMed ID: 12150907
[TBL] [Abstract][Full Text] [Related]
14. Parkin suppresses dopaminergic neuron-selective neurotoxicity induced by Pael-R in Drosophila.
Yang Y; Nishimura I; Imai Y; Takahashi R; Lu B
Neuron; 2003 Mar; 37(6):911-24. PubMed ID: 12670421
[TBL] [Abstract][Full Text] [Related]
15. Suppressive effects of 4-phenylbutyrate on the aggregation of Pael receptors and endoplasmic reticulum stress.
Kubota K; Niinuma Y; Kaneko M; Okuma Y; Sugai M; Omura T; Uesugi M; Uehara T; Hosoi T; Nomura Y
J Neurochem; 2006 Jun; 97(5):1259-68. PubMed ID: 16539653
[TBL] [Abstract][Full Text] [Related]
16. [Pharmacological studies on neurodegenerative diseases focusing on refolding and degradation of unfolded proteins in the endoplasmic reticulum].
Nomura Y
Yakugaku Zasshi; 2014; 134(4):537-43. PubMed ID: 24694815
[TBL] [Abstract][Full Text] [Related]
17. Targeted overexpression of the parkin substrate Pael-R in the nigrostriatal system of adult rats to model Parkinson's disease.
Dusonchet J; Bensadoun JC; Schneider BL; Aebischer P
Neurobiol Dis; 2009 Jul; 35(1):32-41. PubMed ID: 19348945
[TBL] [Abstract][Full Text] [Related]
18. An unfolded putative transmembrane polypeptide, which can lead to endoplasmic reticulum stress, is a substrate of Parkin.
Imai Y; Soda M; Inoue H; Hattori N; Mizuno Y; Takahashi R
Cell; 2001 Jun; 105(7):891-902. PubMed ID: 11439185
[TBL] [Abstract][Full Text] [Related]
19. Functional modulation of parkin through physical interaction with SUMO-1.
Um JW; Chung KC
J Neurosci Res; 2006 Nov; 84(7):1543-54. PubMed ID: 16955485
[TBL] [Abstract][Full Text] [Related]
20. Pael-R is accumulated in Lewy bodies of Parkinson's disease.
Murakami T; Shoji M; Imai Y; Inoue H; Kawarabayashi T; Matsubara E; Harigaya Y; Sasaki A; Takahashi R; Abe K
Ann Neurol; 2004 Mar; 55(3):439-42. PubMed ID: 14991825
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
