BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

228 related articles for article (PubMed ID: 17074758)

  • 1. The vaccinia virus protein F1L interacts with Bim and inhibits activation of the pro-apoptotic protein Bax.
    Taylor JM; Quilty D; Banadyga L; Barry M
    J Biol Chem; 2006 Dec; 281(51):39728-39. PubMed ID: 17074758
    [TBL] [Abstract][Full Text] [Related]  

  • 2. Structural insight into BH3 domain binding of vaccinia virus antiapoptotic F1L.
    Campbell S; Thibault J; Mehta N; Colman PM; Barry M; Kvansakul M
    J Virol; 2014 Aug; 88(15):8667-77. PubMed ID: 24850748
    [TBL] [Abstract][Full Text] [Related]  

  • 3. The vaccinia virus F1L protein interacts with the proapoptotic protein Bak and inhibits Bak activation.
    Wasilenko ST; Banadyga L; Bond D; Barry M
    J Virol; 2005 Nov; 79(22):14031-43. PubMed ID: 16254338
    [TBL] [Abstract][Full Text] [Related]  

  • 4. Vaccinia virus F1L interacts with Bak using highly divergent Bcl-2 homology domains and replaces the function of Mcl-1.
    Campbell S; Hazes B; Kvansakul M; Colman P; Barry M
    J Biol Chem; 2010 Feb; 285(7):4695-708. PubMed ID: 19955184
    [TBL] [Abstract][Full Text] [Related]  

  • 5. Variola virus F1L is a Bcl-2-like protein that unlike its vaccinia virus counterpart inhibits apoptosis independent of Bim.
    Marshall B; Puthalakath H; Caria S; Chugh S; Doerflinger M; Colman PM; Kvansakul M
    Cell Death Dis; 2015 Mar; 6(3):e1680. PubMed ID: 25766319
    [TBL] [Abstract][Full Text] [Related]  

  • 6. Interaction of F1L with the BH3 domain of Bak is responsible for inhibiting vaccinia-induced apoptosis.
    Postigo A; Cross JR; Downward J; Way M
    Cell Death Differ; 2006 Oct; 13(10):1651-62. PubMed ID: 16439990
    [TBL] [Abstract][Full Text] [Related]  

  • 7. Bcl-XL protects BimEL-induced Bax conformational change and cytochrome C release independent of interacting with Bax or BimEL.
    Yamaguchi H; Wang HG
    J Biol Chem; 2002 Nov; 277(44):41604-12. PubMed ID: 12198137
    [TBL] [Abstract][Full Text] [Related]  

  • 8. BimS-induced apoptosis requires mitochondrial localization but not interaction with anti-apoptotic Bcl-2 proteins.
    Weber A; Paschen SA; Heger K; Wilfling F; Frankenberg T; Bauerschmitt H; Seiffert BM; Kirschnek S; Wagner H; Häcker G
    J Cell Biol; 2007 May; 177(4):625-36. PubMed ID: 17517961
    [TBL] [Abstract][Full Text] [Related]  

  • 9. Toxoplasma gondii infection confers resistance against BimS-induced apoptosis by preventing the activation and mitochondrial targeting of pro-apoptotic Bax.
    Hippe D; Weber A; Zhou L; Chang DC; Häcker G; Lüder CG
    J Cell Sci; 2009 Oct; 122(Pt 19):3511-21. PubMed ID: 19737817
    [TBL] [Abstract][Full Text] [Related]  

  • 10. Bax/Bak activation in the absence of Bid, Bim, Puma, and p53.
    Zhang J; Huang K; O'Neill KL; Pang X; Luo X
    Cell Death Dis; 2016 Jun; 7(6):e2266. PubMed ID: 27310874
    [TBL] [Abstract][Full Text] [Related]  

  • 11. Involvement of proapoptotic molecules Bax and Bak in tumor necrosis factor-related apoptosis-inducing ligand (TRAIL)-induced mitochondrial disruption and apoptosis: differential regulation of cytochrome c and Smac/DIABLO release.
    Kandasamy K; Srinivasula SM; Alnemri ES; Thompson CB; Korsmeyer SJ; Bryant JL; Srivastava RK
    Cancer Res; 2003 Apr; 63(7):1712-21. PubMed ID: 12670926
    [TBL] [Abstract][Full Text] [Related]  

  • 12. Direct addition of BimL to mitochondria does not lead to cytochrome c release.
    Terradillos O; Montessuit S; Huang DC; Martinou JC
    FEBS Lett; 2002 Jul; 522(1-3):29-34. PubMed ID: 12095614
    [TBL] [Abstract][Full Text] [Related]  

  • 13. Vaccinia virus anti-apoptotic F1L is a novel Bcl-2-like domain-swapped dimer that binds a highly selective subset of BH3-containing death ligands.
    Kvansakul M; Yang H; Fairlie WD; Czabotar PE; Fischer SF; Perugini MA; Huang DC; Colman PM
    Cell Death Differ; 2008 Oct; 15(10):1564-71. PubMed ID: 18551131
    [TBL] [Abstract][Full Text] [Related]  

  • 14. The vaccinia virus-encoded Bcl-2 homologues do not act as direct Bax inhibitors.
    Postigo A; Way M
    J Virol; 2012 Jan; 86(1):203-13. PubMed ID: 22013032
    [TBL] [Abstract][Full Text] [Related]  

  • 15. Vaccinia virus protein F1L is a caspase-9 inhibitor.
    Zhai D; Yu E; Jin C; Welsh K; Shiau CW; Chen L; Salvesen GS; Liddington R; Reed JC
    J Biol Chem; 2010 Feb; 285(8):5569-80. PubMed ID: 20022954
    [TBL] [Abstract][Full Text] [Related]  

  • 16. Bcl-2 family member Bfl-1/A1 sequesters truncated bid to inhibit is collaboration with pro-apoptotic Bak or Bax.
    Werner AB; de Vries E; Tait SW; Bontjer I; Borst J
    J Biol Chem; 2002 Jun; 277(25):22781-8. PubMed ID: 11929871
    [TBL] [Abstract][Full Text] [Related]  

  • 17. Tumor necrosis factor-alpha induces Bax-Bak interaction and apoptosis, which is inhibited by adenovirus E1B 19K.
    Sundararajan R; Cuconati A; Nelson D; White E
    J Biol Chem; 2001 Nov; 276(48):45120-7. PubMed ID: 11571294
    [TBL] [Abstract][Full Text] [Related]  

  • 18. Induction of Noxa-mediated apoptosis by modified vaccinia virus Ankara depends on viral recognition by cytosolic helicases, leading to IRF-3/IFN-β-dependent induction of pro-apoptotic Noxa.
    Eitz Ferrer P; Potthoff S; Kirschnek S; Gasteiger G; Kastenmüller W; Ludwig H; Paschen SA; Villunger A; Sutter G; Drexler I; Häcker G
    PLoS Pathog; 2011 Jun; 7(6):e1002083. PubMed ID: 21698224
    [TBL] [Abstract][Full Text] [Related]  

  • 19. BimL directly neutralizes Bcl-xL to promote Bax activation during UV-induced apoptosis.
    Wang X; Xing D; Liu L; Chen WR
    FEBS Lett; 2009 Jun; 583(12):1873-9. PubMed ID: 19427863
    [TBL] [Abstract][Full Text] [Related]  

  • 20. Mitochondrial membrane permeabilisation by Bax/Bak.
    Degli Esposti M; Dive C
    Biochem Biophys Res Commun; 2003 May; 304(3):455-61. PubMed ID: 12729579
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 12.