BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

181 related articles for article (PubMed ID: 17159817)

  • 1. Chronic fatigue syndrome is accompanied by an IgM-related immune response directed against neopitopes formed by oxidative or nitrosative damage to lipids and proteins.
    Maes M; Mihaylova I; Leunis JC
    Neuro Endocrinol Lett; 2006 Oct; 27(5):615-21. PubMed ID: 17159817
    [TBL] [Abstract][Full Text] [Related]  

  • 2. An IgM-mediated immune response directed against nitro-bovine serum albumin (nitro-BSA) in chronic fatigue syndrome (CFS) and major depression: evidence that nitrosative stress is another factor underpinning the comorbidity between major depression and CFS.
    Maes M; Mihaylova I; Kubera M; Leunis JC
    Neuro Endocrinol Lett; 2008 Jun; 29(3):313-9. PubMed ID: 18580855
    [TBL] [Abstract][Full Text] [Related]  

  • 3. IgM-mediated autoimmune responses directed against multiple neoepitopes in depression: new pathways that underpin the inflammatory and neuroprogressive pathophysiology.
    Maes M; Mihaylova I; Kubera M; Leunis JC; Geffard M
    J Affect Disord; 2011 Dec; 135(1-3):414-8. PubMed ID: 21930301
    [TBL] [Abstract][Full Text] [Related]  

  • 4. IgM-mediated autoimmune responses directed against anchorage epitopes are greater in Myalgic Encephalomyelitis/Chronic Fatigue Syndrome (ME/CFS) than in major depression.
    Maes M; Mihaylova I; Kubera M; Leunis JC; Twisk FN; Geffard M
    Metab Brain Dis; 2012 Dec; 27(4):415-23. PubMed ID: 22614823
    [TBL] [Abstract][Full Text] [Related]  

  • 5. Increased serum IgM antibodies directed against phosphatidyl inositol (Pi) in chronic fatigue syndrome (CFS) and major depression: evidence that an IgM-mediated immune response against Pi is one factor underpinning the comorbidity between both CFS and depression.
    Maes M; Mihaylova I; Leunis JC
    Neuro Endocrinol Lett; 2007 Dec; 28(6):861-7. PubMed ID: 18063934
    [TBL] [Abstract][Full Text] [Related]  

  • 6. Attenuation of autoimmune responses to oxidative specific epitopes, but not nitroso-adducts, is associated with a better clinical outcome in Myalgic Encephalomyelitis/chronic fatigue syndrome.
    Maes M; Leunis JC
    Neuro Endocrinol Lett; 2014; 35(7):577-85. PubMed ID: 25617880
    [TBL] [Abstract][Full Text] [Related]  

  • 7. Increased autoimmune responses against auto-epitopes modified by oxidative and nitrosative damage in depression: implications for the pathways to chronic depression and neuroprogression.
    Maes M; Kubera M; Mihaylova I; Geffard M; Galecki P; Leunis JC; Berk M
    J Affect Disord; 2013 Jul; 149(1-3):23-9. PubMed ID: 22898471
    [TBL] [Abstract][Full Text] [Related]  

  • 8. Construction of a nitro-oxidative stress-driven, mechanistic model of mood disorders: A nomothetic network approach.
    Simeonova D; Stoyanov D; Leunis JC; Murdjeva M; Maes M
    Nitric Oxide; 2021 Jan; 106():45-54. PubMed ID: 33186727
    [TBL] [Abstract][Full Text] [Related]  

  • 9. Normalization of leaky gut in chronic fatigue syndrome (CFS) is accompanied by a clinical improvement: effects of age, duration of illness and the translocation of LPS from gram-negative bacteria.
    Maes M; Leunis JC
    Neuro Endocrinol Lett; 2008 Dec; 29(6):902-10. PubMed ID: 19112401
    [TBL] [Abstract][Full Text] [Related]  

  • 10. In chronic fatigue syndrome, the decreased levels of omega-3 poly-unsaturated fatty acids are related to lowered serum zinc and defects in T cell activation.
    Maes M; Mihaylova I; Leunis JC
    Neuro Endocrinol Lett; 2005 Dec; 26(6):745-51. PubMed ID: 16380690
    [TBL] [Abstract][Full Text] [Related]  

  • 11. Increased serum IgA and IgM against LPS of enterobacteria in chronic fatigue syndrome (CFS): indication for the involvement of gram-negative enterobacteria in the etiology of CFS and for the presence of an increased gut-intestinal permeability.
    Maes M; Mihaylova I; Leunis JC
    J Affect Disord; 2007 Apr; 99(1-3):237-40. PubMed ID: 17007934
    [TBL] [Abstract][Full Text] [Related]  

  • 12. A review on the oxidative and nitrosative stress (O&NS) pathways in major depression and their possible contribution to the (neuro)degenerative processes in that illness.
    Maes M; Galecki P; Chang YS; Berk M
    Prog Neuropsychopharmacol Biol Psychiatry; 2011 Apr; 35(3):676-92. PubMed ID: 20471444
    [TBL] [Abstract][Full Text] [Related]  

  • 13. Natural regulatory IgM-mediated autoimmune responses directed against malondialdehyde regulate oxidative and nitrosative pathways and coupled with IgM responses to nitroso adducts attenuate depressive and physiosomatic symptoms at the end of term pregnancy.
    Roomruangwong C; Barbosa DS; de Farias CC; Matsumoto AK; Baltus THL; Morelli NR; Kanchanatawan B; Duleu S; Geffard M; Maes M
    Psychiatry Clin Neurosci; 2018 Feb; 72(2):116-130. PubMed ID: 29194848
    [TBL] [Abstract][Full Text] [Related]  

  • 14. Increased expression of activation antigens on CD8+ T lymphocytes in Myalgic Encephalomyelitis/chronic fatigue syndrome: inverse associations with lowered CD19+ expression and CD4+/CD8+ ratio, but no associations with (auto)immune, leaky gut, oxidative and nitrosative stress biomarkers.
    Maes M; Bosmans E; Kubera M
    Neuro Endocrinol Lett; 2015; 36(5):439-46. PubMed ID: 26707044
    [TBL] [Abstract][Full Text] [Related]  

  • 15. Mitochondrial dysfunctions in myalgic encephalomyelitis/chronic fatigue syndrome explained by activated immuno-inflammatory, oxidative and nitrosative stress pathways.
    Morris G; Maes M
    Metab Brain Dis; 2014 Mar; 29(1):19-36. PubMed ID: 24557875
    [TBL] [Abstract][Full Text] [Related]  

  • 16. Increased 8-hydroxy-deoxyguanosine, a marker of oxidative damage to DNA, in major depression and myalgic encephalomyelitis / chronic fatigue syndrome.
    Maes M; Mihaylova I; Kubera M; Uytterhoeven M; Vrydags N; Bosmans E
    Neuro Endocrinol Lett; 2009; 30(6):715-22. PubMed ID: 20035260
    [TBL] [Abstract][Full Text] [Related]  

  • 17. Not in the mind of neurasthenic lazybones but in the cell nucleus: patients with chronic fatigue syndrome have increased production of nuclear factor kappa beta.
    Maes M; Mihaylova I; Bosmans E
    Neuro Endocrinol Lett; 2007 Aug; 28(4):456-62. PubMed ID: 17693979
    [TBL] [Abstract][Full Text] [Related]  

  • 18. In depression, bacterial translocation may drive inflammatory responses, oxidative and nitrosative stress (O&NS), and autoimmune responses directed against O&NS-damaged neoepitopes.
    Maes M; Kubera M; Leunis JC; Berk M; Geffard M; Bosmans E
    Acta Psychiatr Scand; 2013 May; 127(5):344-54. PubMed ID: 22900942
    [TBL] [Abstract][Full Text] [Related]  

  • 19. In Schizophrenia, Deficits in Natural IgM Isotype Antibodies Including those Directed to Malondialdehyde and Azelaic Acid Strongly Predict Negative Symptoms, Neurocognitive Impairments, and the Deficit Syndrome.
    Maes M; Kanchanatawan B; Sirivichayakul S; Carvalho AF
    Mol Neurobiol; 2019 Jul; 56(7):5122-5135. PubMed ID: 30484113
    [TBL] [Abstract][Full Text] [Related]  

  • 20. Why myalgic encephalomyelitis/chronic fatigue syndrome (ME/CFS) may kill you: disorders in the inflammatory and oxidative and nitrosative stress (IO&NS) pathways may explain cardiovascular disorders in ME/CFS.
    Maes M; Twisk FN
    Neuro Endocrinol Lett; 2009; 30(6):677-93. PubMed ID: 20038921
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 10.