195 related articles for article (PubMed ID: 17339354)
1. Shigella flexneri inhibits staurosporine-induced apoptosis in epithelial cells.
Clark CS; Maurelli AT
Infect Immun; 2007 May; 75(5):2531-9. PubMed ID: 17339354
[TBL] [Abstract][Full Text] [Related]
2. Spa15 of Shigella flexneri is secreted through the type III secretion system and prevents staurosporine-induced apoptosis.
Faherty CS; Maurelli AT
Infect Immun; 2009 Dec; 77(12):5281-90. PubMed ID: 19805534
[TBL] [Abstract][Full Text] [Related]
3. Microarray analysis of Shigella flexneri-infected epithelial cells identifies host factors important for apoptosis inhibition.
Faherty CS; Merrell DS; Semino-Mora C; Dubois A; Ramaswamy AV; Maurelli AT
BMC Genomics; 2010 Apr; 11():272. PubMed ID: 20429941
[TBL] [Abstract][Full Text] [Related]
4. Intracellular type III secretion by cytoplasmic Shigella flexneri promotes caspase-1-dependent macrophage cell death.
Schroeder GN; Jann NJ; Hilbi H
Microbiology (Reading); 2007 Sep; 153(Pt 9):2862-2876. PubMed ID: 17768231
[TBL] [Abstract][Full Text] [Related]
5. Transcriptional adaptation of Shigella flexneri during infection of macrophages and epithelial cells: insights into the strategies of a cytosolic bacterial pathogen.
Lucchini S; Liu H; Jin Q; Hinton JC; Yu J
Infect Immun; 2005 Jan; 73(1):88-102. PubMed ID: 15618144
[TBL] [Abstract][Full Text] [Related]
6. A bacterial invasin induces macrophage apoptosis by binding directly to ICE.
Chen Y; Smith MR; Thirumalai K; Zychlinsky A
EMBO J; 1996 Aug; 15(15):3853-60. PubMed ID: 8670890
[TBL] [Abstract][Full Text] [Related]
7. Structure-function analysis of the Shigella virulence factor IpaB.
Guichon A; Hersh D; Smith MR; Zychlinsky A
J Bacteriol; 2001 Feb; 183(4):1269-76. PubMed ID: 11157939
[TBL] [Abstract][Full Text] [Related]
8. Analysis of virulence plasmid gene expression defines three classes of effectors in the type III secretion system of Shigella flexneri.
Gall TL; Mavris M; Martino MC; Bernardini ML; Denamur E; Parsot C
Microbiology (Reading); 2005 Mar; 151(Pt 3):951-962. PubMed ID: 15758240
[TBL] [Abstract][Full Text] [Related]
9. Tripeptidyl peptidase II promotes maturation of caspase-1 in Shigella flexneri-induced macrophage apoptosis.
Hilbi H; Puro RJ; Zychlinsky A
Infect Immun; 2000 Oct; 68(10):5502-8. PubMed ID: 10992446
[TBL] [Abstract][Full Text] [Related]
10. Shigella flexneri-HeLa cell interactions: a putative role for host cell protein kinases.
Collaco C; Dyer RB; Doan R; Herzog NK; Niesel DW
FEMS Immunol Med Microbiol; 1995 Jan; 10(2):93-100. PubMed ID: 7719286
[TBL] [Abstract][Full Text] [Related]
11. Cholesterol is required to trigger caspase-1 activation and macrophage apoptosis after phagosomal escape of Shigella.
Schroeder GN; Hilbi H
Cell Microbiol; 2007 Jan; 9(1):265-78. PubMed ID: 16925787
[TBL] [Abstract][Full Text] [Related]
12. MxiE regulates intracellular expression of factors secreted by the Shigella flexneri 2a type III secretion system.
Kane CD; Schuch R; Day WA; Maurelli AT
J Bacteriol; 2002 Aug; 184(16):4409-19. PubMed ID: 12142411
[TBL] [Abstract][Full Text] [Related]
13. Two novel virulence loci, mxiA and mxiB, in Shigella flexneri 2a facilitate excretion of invasion plasmid antigens.
Andrews GP; Hromockyj AE; Coker C; Maurelli AT
Infect Immun; 1991 Jun; 59(6):1997-2005. PubMed ID: 2037361
[TBL] [Abstract][Full Text] [Related]
14. Identification of functional regions within invasion plasmid antigen C (IpaC) of Shigella flexneri.
Picking WL; Coye L; Osiecki JC; Barnoski Serfis A; Schaper E; Picking WD
Mol Microbiol; 2001 Jan; 39(1):100-11. PubMed ID: 11123692
[TBL] [Abstract][Full Text] [Related]
15. Transcriptional adaptation of Shigella flexneri during adherence to epithelial cells.
Ni Z; Jiang L; Feng L; Wang L; Liu B
J Basic Microbiol; 2015 Feb; 55(2):186-94. PubMed ID: 25291620
[TBL] [Abstract][Full Text] [Related]
16. IpaB of Shigella flexneri causes entry into epithelial cells and escape from the phagocytic vacuole.
High N; Mounier J; Prévost MC; Sansonetti PJ
EMBO J; 1992 May; 11(5):1991-9. PubMed ID: 1582426
[TBL] [Abstract][Full Text] [Related]
17. Analysis of epithelial cell stress response during infection by Shigella flexneri.
Mantis N; Prévost MC; Sansonetti P
Infect Immun; 1996 Jul; 64(7):2474-82. PubMed ID: 8698469
[TBL] [Abstract][Full Text] [Related]
18. The Shigella OspC3 effector inhibits caspase-4, antagonizes inflammatory cell death, and promotes epithelial infection.
Kobayashi T; Ogawa M; Sanada T; Mimuro H; Kim M; Ashida H; Akakura R; Yoshida M; Kawalec M; Reichhart JM; Mizushima T; Sasakawa C
Cell Host Microbe; 2013 May; 13(5):570-583. PubMed ID: 23684308
[TBL] [Abstract][Full Text] [Related]
19. OspF and OspC1 are Shigella flexneri type III secretion system effectors that are required for postinvasion aspects of virulence.
Zurawski DV; Mitsuhata C; Mumy KL; McCormick BA; Maurelli AT
Infect Immun; 2006 Oct; 74(10):5964-76. PubMed ID: 16988276
[TBL] [Abstract][Full Text] [Related]
20. Invasive Shigella flexneri activates NF-kappa B through a lipopolysaccharide-dependent innate intracellular response and leads to IL-8 expression in epithelial cells.
Philpott DJ; Yamaoka S; Israël A; Sansonetti PJ
J Immunol; 2000 Jul; 165(2):903-14. PubMed ID: 10878365
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
