250 related articles for article (PubMed ID: 17468500)
1. Interplay between DNA N-glycosylases/AP lyases at multiply damaged sites and biological consequences.
Eot-Houllier G; Gonera M; Gasparutto D; Giustranti C; Sage E
Nucleic Acids Res; 2007; 35(10):3355-66. PubMed ID: 17468500
[TBL] [Abstract][Full Text] [Related]
2. Processing of a complex multiply damaged DNA site by human cell extracts and purified repair proteins.
Eot-Houllier G; Eon-Marchais S; Gasparutto D; Sage E
Nucleic Acids Res; 2005; 33(1):260-71. PubMed ID: 15647508
[TBL] [Abstract][Full Text] [Related]
3. Attempted base excision repair of ionizing radiation damage in human lymphoblastoid cells produces lethal and mutagenic double strand breaks.
Yang N; Galick H; Wallace SS
DNA Repair (Amst); 2004 Oct; 3(10):1323-34. PubMed ID: 15336627
[TBL] [Abstract][Full Text] [Related]
4. XRCC1 interactions with base excision repair DNA intermediates.
Nazarkina ZK; Khodyreva SN; Marsin S; Lavrik OI; Radicella JP
DNA Repair (Amst); 2007 Feb; 6(2):254-64. PubMed ID: 17118717
[TBL] [Abstract][Full Text] [Related]
5. Base excision repair by hNTH1 and hOGG1: a two edged sword in the processing of DNA damage in gamma-irradiated human cells.
Yang N; Chaudhry MA; Wallace SS
DNA Repair (Amst); 2006 Jan; 5(1):43-51. PubMed ID: 16111924
[TBL] [Abstract][Full Text] [Related]
6. 8-OxoG retards the activity of the ligase III/XRCC1 complex during the repair of a single-strand break, when present within a clustered DNA damage site.
Lomax ME; Cunniffe S; O'Neill P
DNA Repair (Amst); 2004 Mar; 3(3):289-99. PubMed ID: 15177044
[TBL] [Abstract][Full Text] [Related]
7. APE1-dependent repair of DNA single-strand breaks containing 3'-end 8-oxoguanine.
Parsons JL; Dianova II; Dianov GL
Nucleic Acids Res; 2005; 33(7):2204-9. PubMed ID: 15831793
[TBL] [Abstract][Full Text] [Related]
8. Slow base excision by human alkyladenine DNA glycosylase limits the rate of formation of AP sites and AP endonuclease 1 does not stimulate base excision.
Maher RL; Vallur AC; Feller JA; Bloom LB
DNA Repair (Amst); 2007 Jan; 6(1):71-81. PubMed ID: 17018265
[TBL] [Abstract][Full Text] [Related]
9. Major oxidative products of cytosine are substrates for the nucleotide incision repair pathway.
Daviet S; Couvé-Privat S; Gros L; Shinozuka K; Ide H; Saparbaev M; Ishchenko AA
DNA Repair (Amst); 2007 Jan; 6(1):8-18. PubMed ID: 16978929
[TBL] [Abstract][Full Text] [Related]
10. Replication fork collapse is a major cause of the high mutation frequency at three-base lesion clusters.
Sedletska Y; Radicella JP; Sage E
Nucleic Acids Res; 2013 Nov; 41(20):9339-48. PubMed ID: 23945941
[TBL] [Abstract][Full Text] [Related]
11. Repair of tandem base lesions in DNA by human cell extracts generates persisting single-strand breaks.
Budworth H; Matthewman G; O'Neill P; Dianov GL
J Mol Biol; 2005 Sep; 351(5):1020-9. PubMed ID: 16054643
[TBL] [Abstract][Full Text] [Related]
12. DNA polymerase beta is the major dRP lyase involved in repair of oxidative base lesions in DNA by mammalian cell extracts.
Allinson SL; Dianova II; Dianov GL
EMBO J; 2001 Dec; 20(23):6919-26. PubMed ID: 11726527
[TBL] [Abstract][Full Text] [Related]
13. Different organization of base excision repair of uracil in DNA in nuclei and mitochondria and selective upregulation of mitochondrial uracil-DNA glycosylase after oxidative stress.
Akbari M; Otterlei M; Peña-Diaz J; Krokan HE
Neuroscience; 2007 Apr; 145(4):1201-12. PubMed ID: 17101234
[TBL] [Abstract][Full Text] [Related]
14. Recognition of the oxidized lesions spiroiminodihydantoin and guanidinohydantoin in DNA by the mammalian base excision repair glycosylases NEIL1 and NEIL2.
Hailer MK; Slade PG; Martin BD; Rosenquist TA; Sugden KD
DNA Repair (Amst); 2005 Jan; 4(1):41-50. PubMed ID: 15533836
[TBL] [Abstract][Full Text] [Related]
15. Product inhibition and magnesium modulate the dual reaction mode of hOgg1.
Morland I; Luna L; Gustad E; Seeberg E; Bjørås M
DNA Repair (Amst); 2005 Mar; 4(3):381-7. PubMed ID: 15661661
[TBL] [Abstract][Full Text] [Related]
16. Securing genome stability by orchestrating DNA repair: removal of radiation-induced clustered lesions in DNA.
Dianov GL; O'Neill P; Goodhead DT
Bioessays; 2001 Aug; 23(8):745-9. PubMed ID: 11494323
[TBL] [Abstract][Full Text] [Related]
17. The formation of double-strand breaks at multiply damaged sites is driven by the kinetics of excision/incision at base damage in eukaryotic cells.
Kozmin SG; Sedletska Y; Reynaud-Angelin A; Gasparutto D; Sage E
Nucleic Acids Res; 2009 Apr; 37(6):1767-77. PubMed ID: 19174565
[TBL] [Abstract][Full Text] [Related]
18. Enzymatic processing of radiation-induced free radical damage in DNA.
Wallace SS
Radiat Res; 1998 Nov; 150(5 Suppl):S60-79. PubMed ID: 9806610
[TBL] [Abstract][Full Text] [Related]
19. DNA damage levels and biochemical repair capacities associated with XRCC1 deficiency.
Wong HK; Kim D; Hogue BA; McNeill DR; Wilson DM
Biochemistry; 2005 Nov; 44(43):14335-43. PubMed ID: 16245950
[TBL] [Abstract][Full Text] [Related]
20. Base-excision repair of oxidative DNA damage by DNA glycosylases.
Dizdaroglu M
Mutat Res; 2005 Dec; 591(1-2):45-59. PubMed ID: 16054172
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]