245 related articles for article (PubMed ID: 17531925)
1. Matrix metalloprotease activity is enhanced in the compensated but not in the decompensated phase of pressure overload hypertrophy.
Tozzi R; Palladini G; Fallarini S; Nano R; Gatti C; Presotto C; Schiavone A; Micheletti R; Ferrari P; Fogari R; Perlini S
Am J Hypertens; 2007 Jun; 20(6):663-9. PubMed ID: 17531925
[TBL] [Abstract][Full Text] [Related]
2. Matrix metalloproteinases and their tissue inhibitors in pressure-overloaded human myocardium during heart failure progression.
Polyakova V; Hein S; Kostin S; Ziegelhoeffer T; Schaper J
J Am Coll Cardiol; 2004 Oct; 44(8):1609-18. PubMed ID: 15489093
[TBL] [Abstract][Full Text] [Related]
3. Matrix metalloproteinases/tissue inhibitors of metalloproteinases: relationship between changes in proteolytic determinants of matrix composition and structural, functional, and clinical manifestations of hypertensive heart disease.
Ahmed SH; Clark LL; Pennington WR; Webb CS; Bonnema DD; Leonardi AH; McClure CD; Spinale FG; Zile MR
Circulation; 2006 May; 113(17):2089-96. PubMed ID: 16636176
[TBL] [Abstract][Full Text] [Related]
4. Concentric left ventricular remodeling in endothelial nitric oxide synthase knockout mice by chronic pressure overload.
Ruetten H; Dimmeler S; Gehring D; Ihling C; Zeiher AM
Cardiovasc Res; 2005 Jun; 66(3):444-53. PubMed ID: 15914109
[TBL] [Abstract][Full Text] [Related]
5. Extracellular matrix remodeling attenuated after experimental postinfarct left ventricular aneurysm repair.
Hsu CP; Huang CY; Wang JS; Sun PC; Shih CC
Ann Thorac Surg; 2008 Oct; 86(4):1243-9. PubMed ID: 18805169
[TBL] [Abstract][Full Text] [Related]
6. Tumor necrosis factor-alpha mediates cardiac remodeling and ventricular dysfunction after pressure overload state.
Sun M; Chen M; Dawood F; Zurawska U; Li JY; Parker T; Kassiri Z; Kirshenbaum LA; Arnold M; Khokha R; Liu PP
Circulation; 2007 Mar; 115(11):1398-407. PubMed ID: 17353445
[TBL] [Abstract][Full Text] [Related]
7. Increased cardiac expression of tissue inhibitor of metalloproteinase-1 and tissue inhibitor of metalloproteinase-2 is related to cardiac fibrosis and dysfunction in the chronic pressure-overloaded human heart.
Heymans S; Schroen B; Vermeersch P; Milting H; Gao F; Kassner A; Gillijns H; Herijgers P; Flameng W; Carmeliet P; Van de Werf F; Pinto YM; Janssens S
Circulation; 2005 Aug; 112(8):1136-44. PubMed ID: 16103240
[TBL] [Abstract][Full Text] [Related]
8. Chronic pressure overload cardiac hypertrophy and failure in guinea pigs: I. Regional hemodynamics and myocyte remodeling.
Wang X; Li F; Gerdes AM
J Mol Cell Cardiol; 1999 Feb; 31(2):307-17. PubMed ID: 10093044
[TBL] [Abstract][Full Text] [Related]
9. Lack of tissue inhibitor of metalloproteinases 2 leads to exacerbated left ventricular dysfunction and adverse extracellular matrix remodeling in response to biomechanical stress.
Kandalam V; Basu R; Moore L; Fan D; Wang X; Jaworski DM; Oudit GY; Kassiri Z
Circulation; 2011 Nov; 124(19):2094-105. PubMed ID: 21986284
[TBL] [Abstract][Full Text] [Related]
10. Activation of apoptotic caspase cascade during the transition to pressure overload-induced heart failure.
Moorjani N; Ahmad M; Catarino P; Brittin R; Trabzuni D; Al-Mohanna F; Narula N; Narula J; Westaby S
J Am Coll Cardiol; 2006 Oct; 48(7):1451-8. PubMed ID: 17010810
[TBL] [Abstract][Full Text] [Related]
11. Adenosine A1 receptor expression during the transition from compensated pressure overload hypertrophy to heart failure.
Perlini S; Arosio B; Parmeggiani L; Santambrogio D; Palladini G; Tozzi R; Gatti C; Annoni G; Meyer TE; Ferrari AU
J Hypertens; 2007 Feb; 25(2):449-54. PubMed ID: 17211253
[TBL] [Abstract][Full Text] [Related]
12. Pravastatin increases survival and suppresses an increase in myocardial matrix metalloproteinase activity in a rat model of heart failure.
Ichihara S; Noda A; Nagata K; Obata K; Xu J; Ichihara G; Oikawa S; Kawanishi S; Yamada Y; Yokota M
Cardiovasc Res; 2006 Feb; 69(3):726-35. PubMed ID: 16165109
[TBL] [Abstract][Full Text] [Related]
13. Excessive activation of matrix metalloproteinases coincides with left ventricular remodeling during transition from hypertrophy to heart failure in hypertensive rats.
Iwanaga Y; Aoyama T; Kihara Y; Onozawa Y; Yoneda T; Sasayama S
J Am Coll Cardiol; 2002 Apr; 39(8):1384-91. PubMed ID: 11955860
[TBL] [Abstract][Full Text] [Related]
14. The plasminogen-MMP system is more activated in the scar than in viable myocardium 3 months post-MI in the rat.
Gaertner R; Jacob MP; Prunier F; Angles-Cano E; Mercadier JJ; Michel JB
J Mol Cell Cardiol; 2005 Jan; 38(1):193-204. PubMed ID: 15623436
[TBL] [Abstract][Full Text] [Related]
15. Effects of left ventricular volume overload on mitochondrial and death-receptor-mediated apoptotic pathways in the transition to heart failure.
Moorjani N; Westaby S; Narula J; Catarino PA; Brittin R; Kemp TJ; Narula N; Sugden PH
Am J Cardiol; 2009 May; 103(9):1261-8. PubMed ID: 19406269
[TBL] [Abstract][Full Text] [Related]
16. Differential regulation of extracellular matrix constituents in myocardial remodeling with and without heart failure following pressure overload.
Skrbic B; Bjørnstad JL; Marstein HS; Carlson CR; Sjaastad I; Nygård S; Bjørnstad S; Christensen G; Tønnessen T
Matrix Biol; 2013 Mar; 32(2):133-42. PubMed ID: 23220517
[TBL] [Abstract][Full Text] [Related]
17. Selective induction of matrix metalloproteinases and tissue inhibitor of metalloproteinases in atrial and ventricular myocardium in patients with atrial fibrillation.
Mukherjee R; Herron AR; Lowry AS; Stroud RE; Stroud MR; Wharton JM; Ikonomidis JS; Crumbley AJ; Spinale FG; Gold MR
Am J Cardiol; 2006 Feb; 97(4):532-7. PubMed ID: 16461051
[TBL] [Abstract][Full Text] [Related]
18. Calcineurin and matrix protein expression in cardiac hypertrophy: evidence for calcineurin B to control excessive hypertrophic signaling.
Grammer JB; Bleiziffer S; Monticelli F; Lange R; Bauernschmitt R
Basic Res Cardiol; 2006 Jul; 101(4):292-300. PubMed ID: 16688406
[TBL] [Abstract][Full Text] [Related]
19. K(ATP) activation prevents progression of cardiac hypertrophy to failure induced by pressure overload via protecting endothelial function.
Gao S; Long CL; Wang RH; Wang H
Cardiovasc Res; 2009 Aug; 83(3):444-56. PubMed ID: 19304734
[TBL] [Abstract][Full Text] [Related]
20. Involvement of the nicotinamide adenosine dinucleotide phosphate oxidase isoform Nox2 in cardiac contractile dysfunction occurring in response to pressure overload.
Grieve DJ; Byrne JA; Siva A; Layland J; Johar S; Cave AC; Shah AM
J Am Coll Cardiol; 2006 Feb; 47(4):817-26. PubMed ID: 16487851
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
