491 related articles for article (PubMed ID: 17599064)
1. BLM is required for faithful chromosome segregation and its localization defines a class of ultrafine anaphase bridges.
Chan KL; North PS; Hickson ID
EMBO J; 2007 Jul; 26(14):3397-409. PubMed ID: 17599064
[TBL] [Abstract][Full Text] [Related]
2. Bloom's syndrome and PICH helicases cooperate with topoisomerase IIα in centromere disjunction before anaphase.
Rouzeau S; Cordelières FP; Buhagiar-Labarchède G; Hurbain I; Onclercq-Delic R; Gemble S; Magnaghi-Jaulin L; Jaulin C; Amor-Guéret M
PLoS One; 2012; 7(4):e33905. PubMed ID: 22563370
[TBL] [Abstract][Full Text] [Related]
3. PICH and BLM limit histone association with anaphase centromeric DNA threads and promote their resolution.
Ke Y; Huh JW; Warrington R; Li B; Wu N; Leng M; Zhang J; Ball HL; Li B; Yu H
EMBO J; 2011 Jul; 30(16):3309-21. PubMed ID: 21743438
[TBL] [Abstract][Full Text] [Related]
4. BLAP75/RMI1 promotes the BLM-dependent dissolution of homologous recombination intermediates.
Wu L; Bachrati CZ; Ou J; Xu C; Yin J; Chang M; Wang W; Li L; Brown GW; Hickson ID
Proc Natl Acad Sci U S A; 2006 Mar; 103(11):4068-73. PubMed ID: 16537486
[TBL] [Abstract][Full Text] [Related]
5. Bloom helicase and DNA topoisomerase IIIalpha are involved in the dissolution of sister chromatids.
Seki M; Nakagawa T; Seki T; Kato G; Tada S; Takahashi Y; Yoshimura A; Kobayashi T; Aoki A; Otsuki M; Habermann FA; Tanabe H; Ishii Y; Enomoto T
Mol Cell Biol; 2006 Aug; 26(16):6299-307. PubMed ID: 16880537
[TBL] [Abstract][Full Text] [Related]
6. MPS1-dependent mitotic BLM phosphorylation is important for chromosome stability.
Leng M; Chan DW; Luo H; Zhu C; Qin J; Wang Y
Proc Natl Acad Sci U S A; 2006 Aug; 103(31):11485-90. PubMed ID: 16864798
[TBL] [Abstract][Full Text] [Related]
7. BLAP75, an essential component of Bloom's syndrome protein complexes that maintain genome integrity.
Yin J; Sobeck A; Xu C; Meetei AR; Hoatlin M; Li L; Wang W
EMBO J; 2005 Apr; 24(7):1465-76. PubMed ID: 15775963
[TBL] [Abstract][Full Text] [Related]
8. Rif1 Is Required for Resolution of Ultrafine DNA Bridges in Anaphase to Ensure Genomic Stability.
Hengeveld RC; de Boer HR; Schoonen PM; de Vries EG; Lens SM; van Vugt MA
Dev Cell; 2015 Aug; 34(4):466-74. PubMed ID: 26256213
[TBL] [Abstract][Full Text] [Related]
9. The Bloom's syndrome helicase suppresses crossing over during homologous recombination.
Wu L; Hickson ID
Nature; 2003 Dec; 426(6968):870-4. PubMed ID: 14685245
[TBL] [Abstract][Full Text] [Related]
10. Evidence for BLM and Topoisomerase IIIalpha interaction in genomic stability.
Hu P; Beresten SF; van Brabant AJ; Ye TZ; Pandolfi PP; Johnson FB; Guarente L; Ellis NA
Hum Mol Genet; 2001 Jun; 10(12):1287-98. PubMed ID: 11406610
[TBL] [Abstract][Full Text] [Related]
11. Holliday junction processing activity of the BLM-Topo IIIalpha-BLAP75 complex.
Bussen W; Raynard S; Busygina V; Singh AK; Sung P
J Biol Chem; 2007 Oct; 282(43):31484-92. PubMed ID: 17728255
[TBL] [Abstract][Full Text] [Related]
12. A double Holliday junction dissolvasome comprising BLM, topoisomerase IIIalpha, and BLAP75.
Raynard S; Bussen W; Sung P
J Biol Chem; 2006 May; 281(20):13861-4. PubMed ID: 16595695
[TBL] [Abstract][Full Text] [Related]
13. Bloom's syndrome protein response to ultraviolet-C radiation and hydroxyurea-mediated DNA synthesis inhibition.
Ababou M; Dumaire V; Lécluse Y; Amor-Guéret M
Oncogene; 2002 Mar; 21(13):2079-88. PubMed ID: 11960380
[TBL] [Abstract][Full Text] [Related]
14. Absence of BLM leads to accumulation of chromosomal DNA breaks during both unperturbed and disrupted S phases.
Li W; Kim SM; Lee J; Dunphy WG
J Cell Biol; 2004 Jun; 165(6):801-12. PubMed ID: 15197177
[TBL] [Abstract][Full Text] [Related]
15. Cell cycle regulation of the endogenous wild type Bloom's syndrome DNA helicase.
Dutertre S; Ababou M; Onclercq R; Delic J; Chatton B; Jaulin C; Amor-Guéret M
Oncogene; 2000 May; 19(23):2731-8. PubMed ID: 10851073
[TBL] [Abstract][Full Text] [Related]
16. Physical and functional interaction between the Bloom's syndrome gene product and the largest subunit of chromatin assembly factor 1.
Jiao R; Bachrati CZ; Pedrazzi G; Kuster P; Petkovic M; Li JL; Egli D; Hickson ID; Stagljar I
Mol Cell Biol; 2004 Jun; 24(11):4710-9. PubMed ID: 15143166
[TBL] [Abstract][Full Text] [Related]
17. Cleavage of the Bloom's syndrome gene product during apoptosis by caspase-3 results in an impaired interaction with topoisomerase IIIalpha.
Freire R; d'Adda Di Fagagna F; Wu L; Pedrazzi G; Stagljar I; Hickson ID; Jackson SP
Nucleic Acids Res; 2001 Aug; 29(15):3172-80. PubMed ID: 11470874
[TBL] [Abstract][Full Text] [Related]
18. BLAP18/RMI2, a novel OB-fold-containing protein, is an essential component of the Bloom helicase-double Holliday junction dissolvasome.
Singh TR; Ali AM; Busygina V; Raynard S; Fan Q; Du CH; Andreassen PR; Sung P; Meetei AR
Genes Dev; 2008 Oct; 22(20):2856-68. PubMed ID: 18923083
[TBL] [Abstract][Full Text] [Related]
19. The Bloom syndrome protein limits the lethality associated with RAD51 deficiency.
Lahkim Bennani-Belhaj K; Rouzeau S; Buhagiar-Labarchède G; Chabosseau P; Onclercq-Delic R; Bayart E; Cordelières F; Couturier J; Amor-Guéret M
Mol Cancer Res; 2010 Mar; 8(3):385-94. PubMed ID: 20215422
[TBL] [Abstract][Full Text] [Related]
20. The Bloom's syndrome helicase interacts directly with the human DNA mismatch repair protein hMSH6.
Pedrazzi G; Bachrati CZ; Selak N; Studer I; Petkovic M; Hickson ID; Jiricny J; Stagljar I
Biol Chem; 2003 Aug; 384(8):1155-64. PubMed ID: 12974384
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
