252 related articles for article (PubMed ID: 17610901)
1. Down syndrome candidate region 1-like 1 (DSCR1-L1) mimics the inhibitory effects of DSCR1 on calcineurin signaling in endothelial cells and inhibits angiogenesis.
Gollogly LK; Ryeom SW; Yoon SS
J Surg Res; 2007 Sep; 142(1):129-36. PubMed ID: 17610901
[TBL] [Abstract][Full Text] [Related]
2. Down syndrome candidate region 1 isoform 1 mediates angiogenesis through the calcineurin-NFAT pathway.
Qin L; Zhao D; Liu X; Nagy JA; Hoang MV; Brown LF; Dvorak HF; Zeng H
Mol Cancer Res; 2006 Nov; 4(11):811-20. PubMed ID: 17114339
[TBL] [Abstract][Full Text] [Related]
3. VEGF selectively induces Down syndrome critical region 1 gene expression in endothelial cells: a mechanism for feedback regulation of angiogenesis?
Yao YG; Duh EJ
Biochem Biophys Res Commun; 2004 Aug; 321(3):648-56. PubMed ID: 15358155
[TBL] [Abstract][Full Text] [Related]
4. Down syndrome candidate region 1,a downstream target of VEGF, participates in endothelial cell migration and angiogenesis.
Iizuka M; Abe M; Shiiba K; Sasaki I; Sato Y
J Vasc Res; 2004; 41(4):334-44. PubMed ID: 15263820
[TBL] [Abstract][Full Text] [Related]
5. Requirement of the nuclear localization of transcription enhancer factor 3 for proliferation, migration, tube formation, and angiogenesis induced by vascular endothelial growth factor.
Liu X; Zhao D; James L; Li J; Zeng H
FASEB J; 2011 Apr; 25(4):1188-97. PubMed ID: 21169383
[TBL] [Abstract][Full Text] [Related]
6. Down syndrome critical region protein 1 (DSCR1), a novel VEGF target gene that regulates expression of inflammatory markers on activated endothelial cells.
Hesser BA; Liang XH; Camenisch G; Yang S; Lewin DA; Scheller R; Ferrara N; Gerber HP
Blood; 2004 Jul; 104(1):149-58. PubMed ID: 15016650
[TBL] [Abstract][Full Text] [Related]
7. Targeted deletion of the calcineurin inhibitor DSCR1 suppresses tumor growth.
Ryeom S; Baek KH; Rioth MJ; Lynch RC; Zaslavsky A; Birsner A; Yoon SS; McKeon F
Cancer Cell; 2008 May; 13(5):420-31. PubMed ID: 18455125
[TBL] [Abstract][Full Text] [Related]
8. The novel axis of YAP1, transcription enhancer factor 3 and Down Syndrome Candidate Region 1 isoform 1L is a common signaling pathway downstream of several angiogenic factors.
Cui P; Liu X; Zhao K; Hou S; Chen C; Zhao D; Zeng H
Microvasc Res; 2020 May; 129():103955. PubMed ID: 31733305
[TBL] [Abstract][Full Text] [Related]
9. Secretion of Down Syndrome Critical Region 1 Isoform 4 in Ischemic Retinal Ganglion Cells Displays Anti-Angiogenic Properties Via NFATc1-Dependent Pathway.
Xu Y; Yang B; Hu Y; Lu L; Lu X; Wang J; Shu Q; Cheng Q; Yu S; Xu F; Huang J; Liang X
Mol Neurobiol; 2017 Oct; 54(8):6556-6571. PubMed ID: 27734335
[TBL] [Abstract][Full Text] [Related]
10. A novel transcriptional complex on the VE-cadherin promoter regulated the downregulation of VE-cadherin in the Down Syndrome Candidate Region 1 isoform 1L-mediated angiogenesis.
Hou S; Niu G; Liu X; Bourbon PM; Zhang D; Cui P; Zhao K; Zhao D; Zeng H
Microvasc Res; 2021 Nov; 138():104209. PubMed ID: 34146582
[TBL] [Abstract][Full Text] [Related]
11. Arsenic Trioxide Inhibits the Metastasis of Small Cell Lung Cancer by Blocking Calcineurin-Nuclear Factor of Activated T Cells (NFAT) Signaling.
Zheng JC; Chang KJ; Jin YX; Zhao XW; Li B; Yang MH
Med Sci Monit; 2019 Mar; 25():2228-2237. PubMed ID: 30913205
[TBL] [Abstract][Full Text] [Related]
12. A single extra copy of Dscr1 improves survival of mice developing spontaneous lung tumors through suppression of tumor angiogenesis.
Shin J; Lee JC; Baek KH
Cancer Lett; 2014 Jan; 342(1):70-81. PubMed ID: 24051307
[TBL] [Abstract][Full Text] [Related]
13. DSCR1, overexpressed in Down syndrome, is an inhibitor of calcineurin-mediated signaling pathways.
Fuentes JJ; Genescà L; Kingsbury TJ; Cunningham KW; Pérez-Riba M; Estivill X; de la Luna S
Hum Mol Genet; 2000 Jul; 9(11):1681-90. PubMed ID: 10861295
[TBL] [Abstract][Full Text] [Related]
14. Depolarization of neural cells induces transcription of the Down syndrome critical region 1 isoform 4 via a calcineurin/nuclear factor of activated T cells-dependent pathway.
Cano E; Canellada A; Minami T; Iglesias T; Redondo JM
J Biol Chem; 2005 Aug; 280(33):29435-43. PubMed ID: 15975916
[TBL] [Abstract][Full Text] [Related]
15. Necl-5/poliovirus receptor interacts with VEGFR2 and regulates VEGF-induced angiogenesis.
Kinugasa M; Amano H; Satomi-Kobayashi S; Nakayama K; Miyata M; Kubo Y; Nagamatsu Y; Kurogane Y; Kureha F; Yamana S; Hirata K; Miyoshi J; Takai Y; Rikitake Y
Circ Res; 2012 Mar; 110(5):716-26. PubMed ID: 22282193
[TBL] [Abstract][Full Text] [Related]
16. All-trans retinoic acid induces in vitro angiogenesis via retinoic acid receptor: possible involvement of paracrine effects of endogenous vascular endothelial growth factor signaling.
Saito A; Sugawara A; Uruno A; Kudo M; Kagechika H; Sato Y; Owada Y; Kondo H; Sato M; Kurabayashi M; Imaizumi M; Tsuchiya S; Ito S
Endocrinology; 2007 Mar; 148(3):1412-23. PubMed ID: 17170094
[TBL] [Abstract][Full Text] [Related]
17. Induction of tube formation by angiopoietin-1 in endothelial cell/fibroblast co-culture is dependent on endogenous VEGF.
Saito M; Hamasaki M; Shibuya M
Cancer Sci; 2003 Sep; 94(9):782-90. PubMed ID: 12967476
[TBL] [Abstract][Full Text] [Related]
18. Down syndrome candidate region 1 increases the stability of the IkappaBalpha protein: implications for its anti-inflammatory effects.
Kim YS; Cho KO; Lee HJ; Kim SY; Sato Y; Cho YJ
J Biol Chem; 2006 Dec; 281(51):39051-61. PubMed ID: 17062574
[TBL] [Abstract][Full Text] [Related]
19. Down syndrome candidate region-1 protein interacts with Tollip and positively modulates interleukin-1 receptor-mediated signaling.
Lee JY; Lee HJ; Lee EJ; Jang SH; Kim H; Yoon JH; Chung KC
Biochim Biophys Acta; 2009 Dec; 1790(12):1673-80. PubMed ID: 19716405
[TBL] [Abstract][Full Text] [Related]
20. Transcription enhancer factor 3 (TEF3) mediates the expression of Down syndrome candidate region 1 isoform 1 (DSCR1-1L) in endothelial cells.
Liu X; Zhao D; Qin L; Li J; Zeng H
J Biol Chem; 2008 Dec; 283(49):34159-67. PubMed ID: 18840614
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
