These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.
252 related articles for article (PubMed ID: 17611735)
1. Ablation of the gene encoding p66Shc protects mice against AGE-induced glomerulopathy by preventing oxidant-dependent tissue injury and further AGE accumulation. Menini S; Iacobini C; Ricci C; Oddi G; Pesce C; Pugliese F; Block K; Abboud HE; Giorgio M; Migliaccio E; Pelicci PG; Pugliese G Diabetologia; 2007 Sep; 50(9):1997-2007. PubMed ID: 17611735 [TBL] [Abstract][Full Text] [Related]
2. Deletion of p66Shc longevity gene protects against experimental diabetic glomerulopathy by preventing diabetes-induced oxidative stress. Menini S; Amadio L; Oddi G; Ricci C; Pesce C; Pugliese F; Giorgio M; Migliaccio E; Pelicci P; Iacobini C; Pugliese G Diabetes; 2006 Jun; 55(6):1642-50. PubMed ID: 16731826 [TBL] [Abstract][Full Text] [Related]
3. Galectin-3/AGE-receptor 3 knockout mice show accelerated AGE-induced glomerular injury: evidence for a protective role of galectin-3 as an AGE receptor. Iacobini C; Menini S; Oddi G; Ricci C; Amadio L; Pricci F; Olivieri A; Sorcini M; Di Mario U; Pesce C; Pugliese G FASEB J; 2004 Nov; 18(14):1773-5. PubMed ID: 15361471 [TBL] [Abstract][Full Text] [Related]
4. Deletion of the ageing gene p66(Shc) reduces early stroke size following ischaemia/reperfusion brain injury. Spescha RD; Shi Y; Wegener S; Keller S; Weber B; Wyss MM; Lauinger N; Tabatabai G; Paneni F; Cosentino F; Hock C; Weller M; Nitsch RM; Lüscher TF; Camici GG Eur Heart J; 2013 Jan; 34(2):96-103. PubMed ID: 23008506 [TBL] [Abstract][Full Text] [Related]
5. AGE-receptor-1 counteracts cellular oxidant stress induced by AGEs via negative regulation of p66shc-dependent FKHRL1 phosphorylation. Cai W; He JC; Zhu L; Chen X; Striker GE; Vlassara H Am J Physiol Cell Physiol; 2008 Jan; 294(1):C145-52. PubMed ID: 18032526 [TBL] [Abstract][Full Text] [Related]
6. Genetic deletion of p66(Shc) adaptor protein prevents hyperglycemia-induced endothelial dysfunction and oxidative stress. Camici GG; Schiavoni M; Francia P; Bachschmid M; Martin-Padura I; Hersberger M; Tanner FC; Pelicci P; Volpe M; Anversa P; Lüscher TF; Cosentino F Proc Natl Acad Sci U S A; 2007 Mar; 104(12):5217-22. PubMed ID: 17360381 [TBL] [Abstract][Full Text] [Related]
7. Preventing hypoxia/reoxygenation damage to hepatocytes by p66(shc) ablation: up-regulation of anti-oxidant and anti-apoptotic proteins. Haga S; Terui K; Fukai M; Oikawa Y; Irani K; Furukawa H; Todo S; Ozaki M J Hepatol; 2008 Mar; 48(3):422-32. PubMed ID: 18191273 [TBL] [Abstract][Full Text] [Related]
8. The cardioprotective effects elicited by p66(Shc) ablation demonstrate the crucial role of mitochondrial ROS formation in ischemia/reperfusion injury. Carpi A; Menabò R; Kaludercic N; Pelicci P; Di Lisa F; Giorgio M Biochim Biophys Acta; 2009 Jul; 1787(7):774-80. PubMed ID: 19362067 [TBL] [Abstract][Full Text] [Related]
9. Development of age-dependent glomerular lesions in galectin-3/AGE-receptor-3 knockout mice. Iacobini C; Oddi G; Menini S; Amadio L; Ricci C; Di Pippo C; Sorcini M; Pricci F; Pugliese F; Pugliese G Am J Physiol Renal Physiol; 2005 Sep; 289(3):F611-21. PubMed ID: 15870382 [TBL] [Abstract][Full Text] [Related]
10. Genetic deletion of the adaptor protein p66Shc increases susceptibility to short-term ischaemic myocardial injury via intracellular salvage pathways. Akhmedov A; Montecucco F; Braunersreuther V; Camici GG; Jakob P; Reiner MF; Glanzmann M; Burger F; Paneni F; Galan K; Pelli G; Vuilleumier N; Belin A; Vallée JP; Mach F; Lüscher TF Eur Heart J; 2015 Feb; 36(8):516-26a. PubMed ID: 25336219 [TBL] [Abstract][Full Text] [Related]
11. Enhanced age-dependent cerebrovascular dysfunction is mediated by adaptor protein p66Shc. Shi Y; Savarese G; Perrone-Filardi P; Lüscher TF; Camici GG Int J Cardiol; 2014 Aug; 175(3):446-50. PubMed ID: 25012499 [TBL] [Abstract][Full Text] [Related]
12. Final common molecular pathways of aging and cardiovascular disease: role of the p66Shc protein. Cosentino F; Francia P; Camici GG; Pelicci PG; Lüscher TF; Volpe M Arterioscler Thromb Vasc Biol; 2008 Apr; 28(4):622-8. PubMed ID: 18162611 [TBL] [Abstract][Full Text] [Related]
13. Diabetes induces p66shc gene expression in human peripheral blood mononuclear cells: relationship to oxidative stress. Pagnin E; Fadini G; de Toni R; Tiengo A; Calò L; Avogaro A J Clin Endocrinol Metab; 2005 Feb; 90(2):1130-6. PubMed ID: 15562031 [TBL] [Abstract][Full Text] [Related]
14. Deletion of p66shc gene protects against age-related endothelial dysfunction. Francia P; delli Gatti C; Bachschmid M; Martin-Padura I; Savoia C; Migliaccio E; Pelicci PG; Schiavoni M; Lüscher TF; Volpe M; Cosentino F Circulation; 2004 Nov; 110(18):2889-95. PubMed ID: 15505103 [TBL] [Abstract][Full Text] [Related]
15. p66(ShcA) and oxidative stress modulate myogenic differentiation and skeletal muscle regeneration after hind limb ischemia. Zaccagnini G; Martelli F; Magenta A; Cencioni C; Fasanaro P; Nicoletti C; Biglioli P; Pelicci PG; Capogrossi MC J Biol Chem; 2007 Oct; 282(43):31453-9. PubMed ID: 17726026 [TBL] [Abstract][Full Text] [Related]
16. Reduction of p66Shc suppresses oxidative damage in retinal pigmented epithelial cells and retina. Wu Z; Rogers B; Kachi S; Hackett SF; Sick A; Campochiaro PA J Cell Physiol; 2006 Dec; 209(3):996-1005. PubMed ID: 16972253 [TBL] [Abstract][Full Text] [Related]