375 related articles for article (PubMed ID: 17634426)
1. Endogenous gamma-H2AX-ATM-Chk2 checkpoint activation in Bloom's syndrome helicase deficient cells is related to DNA replication arrested forks.
Rao VA; Conti C; Guirouilh-Barbat J; Nakamura A; Miao ZH; Davies SL; Saccá B; Hickson ID; Bensimon A; Pommier Y
Mol Cancer Res; 2007 Jul; 5(7):713-24. PubMed ID: 17634426
[TBL] [Abstract][Full Text] [Related]
2. Phosphorylation of BLM, dissociation from topoisomerase IIIalpha, and colocalization with gamma-H2AX after topoisomerase I-induced replication damage.
Rao VA; Fan AM; Meng L; Doe CF; North PS; Hickson ID; Pommier Y
Mol Cell Biol; 2005 Oct; 25(20):8925-37. PubMed ID: 16199871
[TBL] [Abstract][Full Text] [Related]
3. Possible anti-recombinogenic role of Bloom's syndrome helicase in double-strand break processing.
Onclercq-Delic R; Calsou P; Delteil C; Salles B; Papadopoulo D; Amor-Guéret M
Nucleic Acids Res; 2003 Nov; 31(21):6272-82. PubMed ID: 14576316
[TBL] [Abstract][Full Text] [Related]
4. ATR and ATM-dependent movement of BLM helicase during replication stress ensures optimal ATM activation and 53BP1 focus formation.
Davalos AR; Kaminker P; Hansen RK; Campisi J
Cell Cycle; 2004 Dec; 3(12):1579-86. PubMed ID: 15539948
[TBL] [Abstract][Full Text] [Related]
5. ATM-dependent phosphorylation and accumulation of endogenous BLM protein in response to ionizing radiation.
Ababou M; Dutertre S; Lécluse Y; Onclercq R; Chatton B; Amor-Guéret M
Oncogene; 2000 Dec; 19(52):5955-63. PubMed ID: 11146546
[TBL] [Abstract][Full Text] [Related]
6. Phosphorylation of the Bloom's syndrome helicase and its role in recovery from S-phase arrest.
Davies SL; North PS; Dart A; Lakin ND; Hickson ID
Mol Cell Biol; 2004 Feb; 24(3):1279-91. PubMed ID: 14729972
[TBL] [Abstract][Full Text] [Related]
7. Bloom's syndrome protein response to ultraviolet-C radiation and hydroxyurea-mediated DNA synthesis inhibition.
Ababou M; Dumaire V; Lécluse Y; Amor-Guéret M
Oncogene; 2002 Mar; 21(13):2079-88. PubMed ID: 11960380
[TBL] [Abstract][Full Text] [Related]
8. Assessment of ATM phosphorylation on Ser-1981 induced by DNA topoisomerase I and II inhibitors in relation to Ser-139-histone H2AX phosphorylation, cell cycle phase, and apoptosis.
Kurose A; Tanaka T; Huang X; Halicka HD; Traganos F; Dai W; Darzynkiewicz Z
Cytometry A; 2005 Nov; 68(1):1-9. PubMed ID: 16184611
[TBL] [Abstract][Full Text] [Related]
9. Phosphorylation of p53 on Ser15 during cell cycle caused by Topo I and Topo II inhibitors in relation to ATM and Chk2 activation.
Zhao H; Traganos F; Darzynkiewicz Z
Cell Cycle; 2008 Oct; 7(19):3048-55. PubMed ID: 18802408
[TBL] [Abstract][Full Text] [Related]
10. Functional link between BLM defective in Bloom's syndrome and the ataxia-telangiectasia-mutated protein, ATM.
Beamish H; Kedar P; Kaneko H; Chen P; Fukao T; Peng C; Beresten S; Gueven N; Purdie D; Lees-Miller S; Ellis N; Kondo N; Lavin MF
J Biol Chem; 2002 Aug; 277(34):30515-23. PubMed ID: 12034743
[TBL] [Abstract][Full Text] [Related]
11. ATR and H2AX cooperate in maintaining genome stability under replication stress.
Chanoux RA; Yin B; Urtishak KA; Asare A; Bassing CH; Brown EJ
J Biol Chem; 2009 Feb; 284(9):5994-6003. PubMed ID: 19049966
[TBL] [Abstract][Full Text] [Related]
12. BLM helicase-dependent transport of p53 to sites of stalled DNA replication forks modulates homologous recombination.
Sengupta S; Linke SP; Pedeux R; Yang Q; Farnsworth J; Garfield SH; Valerie K; Shay JW; Ellis NA; Wasylyk B; Harris CC
EMBO J; 2003 Mar; 22(5):1210-22. PubMed ID: 12606585
[TBL] [Abstract][Full Text] [Related]
13. Death receptor-induced activation of the Chk2- and histone H2AX-associated DNA damage response pathways.
Solier S; Sordet O; Kohn KW; Pommier Y
Mol Cell Biol; 2009 Jan; 29(1):68-82. PubMed ID: 18955500
[TBL] [Abstract][Full Text] [Related]
14. ATM activation accompanies histone H2AX phosphorylation in A549 cells upon exposure to tobacco smoke.
Tanaka T; Huang X; Jorgensen E; Gietl D; Traganos F; Darzynkiewicz Z; Albino AP
BMC Cell Biol; 2007 Jun; 8():26. PubMed ID: 17594478
[TBL] [Abstract][Full Text] [Related]
15. Effects of hydroxyurea and aphidicolin on phosphorylation of ataxia telangiectasia mutated on Ser 1981 and histone H2AX on Ser 139 in relation to cell cycle phase and induction of apoptosis.
Kurose A; Tanaka T; Huang X; Traganos F; Dai W; Darzynkiewicz Z
Cytometry A; 2006 Apr; 69(4):212-21. PubMed ID: 16528719
[TBL] [Abstract][Full Text] [Related]
16. Activation of the S phase DNA damage checkpoint by mitomycin C.
Mladenov E; Tsaneva I; Anachkova B
J Cell Physiol; 2007 May; 211(2):468-76. PubMed ID: 17167777
[TBL] [Abstract][Full Text] [Related]
17. Oxidative stress induces cell cycle-dependent Mre11 recruitment, ATM and Chk2 activation and histone H2AX phosphorylation.
Zhao H; Traganos F; Albino AP; Darzynkiewicz Z
Cell Cycle; 2008 May; 7(10):1490-5. PubMed ID: 18418078
[TBL] [Abstract][Full Text] [Related]
18. Bloom's syndrome protein is required for correct relocalization of RAD50/MRE11/NBS1 complex after replication fork arrest.
Franchitto A; Pichierri P
J Cell Biol; 2002 Apr; 157(1):19-30. PubMed ID: 11916980
[TBL] [Abstract][Full Text] [Related]
19. H2AX phosphorylation marks gemcitabine-induced stalled replication forks and their collapse upon S-phase checkpoint abrogation.
Ewald B; Sampath D; Plunkett W
Mol Cancer Ther; 2007 Apr; 6(4):1239-48. PubMed ID: 17406032
[TBL] [Abstract][Full Text] [Related]
20. Expression and nuclear localization of BLM, a chromosome stability protein mutated in Bloom's syndrome, suggest a role in recombination during meiotic prophase.
Moens PB; Freire R; Tarsounas M; Spyropoulos B; Jackson SP
J Cell Sci; 2000 Feb; 113 ( Pt 4)():663-72. PubMed ID: 10652259
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
