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9. New ABCC8 mutations in relapsing neonatal diabetes and clinical features. Vaxillaire M; Dechaume A; Busiah K; Cavé H; Pereira S; Scharfmann R; de Nanclares GP; Castano L; Froguel P; Polak M; Diabetes; 2007 Jun; 56(6):1737-41. PubMed ID: 17389331 [TBL] [Abstract][Full Text] [Related]
10. Mutations in the genes encoding the pancreatic beta-cell KATP channel subunits Kir6.2 (KCNJ11) and SUR1 (ABCC8) in diabetes mellitus and hyperinsulinism. Gloyn AL; Siddiqui J; Ellard S Hum Mutat; 2006 Mar; 27(3):220-31. PubMed ID: 16416420 [TBL] [Abstract][Full Text] [Related]
11. Mutations in ATP-sensitive K+ channel genes cause transient neonatal diabetes and permanent diabetes in childhood or adulthood. Flanagan SE; Patch AM; Mackay DJ; Edghill EL; Gloyn AL; Robinson D; Shield JP; Temple K; Ellard S; Hattersley AT Diabetes; 2007 Jul; 56(7):1930-7. PubMed ID: 17446535 [TBL] [Abstract][Full Text] [Related]
12. Increased ATPase activity produced by mutations at arginine-1380 in nucleotide-binding domain 2 of ABCC8 causes neonatal diabetes. de Wet H; Rees MG; Shimomura K; Aittoniemi J; Patch AM; Flanagan SE; Ellard S; Hattersley AT; Sansom MS; Ashcroft FM Proc Natl Acad Sci U S A; 2007 Nov; 104(48):18988-92. PubMed ID: 18025464 [TBL] [Abstract][Full Text] [Related]
13. Mutations in the ABCC8 (SUR1 subunit of the K(ATP) channel) gene are associated with a variable clinical phenotype. Klupa T; Kowalska I; Wyka K; Skupien J; Patch AM; Flanagan SE; Noczynska A; Arciszewska M; Ellard S; Hattersley AT; Sieradzki J; Mlynarski W; Malecki MT Clin Endocrinol (Oxf); 2009 Sep; 71(3):358-62. PubMed ID: 19021632 [TBL] [Abstract][Full Text] [Related]
14. Diabetes in very young children and mutations in the insulin-secreting cell potassium channel genes: therapeutic consequences. Flechtner I; Vaxillaire M; Cavé H; Scharfmann R; Froguel P; Polak M Endocr Dev; 2007; 12():86-98. PubMed ID: 17923772 [TBL] [Abstract][Full Text] [Related]
15. Compound heterozygous mutations in the SUR1 (ABCC 8) subunit of pancreatic K(ATP) channels cause neonatal diabetes by perturbing the coupling between Kir6.2 and SUR1 subunits. Lin YW; Akrouh A; Hsu Y; Hughes N; Nichols CG; De León DD Channels (Austin); 2012; 6(2):133-8. PubMed ID: 22562119 [TBL] [Abstract][Full Text] [Related]
16. Molecular basis of Kir6.2 mutations associated with neonatal diabetes or neonatal diabetes plus neurological features. Proks P; Antcliff JF; Lippiat J; Gloyn AL; Hattersley AT; Ashcroft FM Proc Natl Acad Sci U S A; 2004 Dec; 101(50):17539-44. PubMed ID: 15583126 [TBL] [Abstract][Full Text] [Related]
17. Heterozygous ABCC8 mutations are a cause of MODY. Bowman P; Flanagan SE; Edghill EL; Damhuis A; Shepherd MH; Paisey R; Hattersley AT; Ellard S Diabetologia; 2012 Jan; 55(1):123-7. PubMed ID: 21989597 [TBL] [Abstract][Full Text] [Related]
18. Mosaic paternal uniparental isodisomy and an ABCC8 gene mutation in a patient with permanent neonatal diabetes and hemihypertrophy. Shield JP; Flanagan SE; Mackay DJ; Harries LW; Proks P; Girard C; Ashcroft FM; Temple IK; Ellard S Diabetes; 2008 Jan; 57(1):255-8. PubMed ID: 17942821 [TBL] [Abstract][Full Text] [Related]
19. [Neonatal diabetes: a disease linked to multiple mechanisms]. Flechtner I; Vaxillaire M; Cavé H; Froguel P; Polak M Arch Pediatr; 2007 Nov; 14(11):1356-65. PubMed ID: 17931842 [TBL] [Abstract][Full Text] [Related]
20. Co-inheritance of two ABCC8 mutations causing an unresponsive congenital hyperinsulinism: clinical and functional characterization of two novel ABCC8 mutations. Faletra F; Snider K; Shyng SL; Bruno I; Athanasakis E; Gasparini P; Dionisi-Vici C; Ventura A; Zhou Q; Stanley CA; Burlina A Gene; 2013 Mar; 516(1):122-5. PubMed ID: 23266803 [TBL] [Abstract][Full Text] [Related] [Next] [New Search]