These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.
177 related articles for article (PubMed ID: 17768234)
1. The Helicobacter pylori CagF protein is a type IV secretion chaperone-like molecule that binds close to the C-terminal secretion signal of the CagA effector protein. Pattis I; Weiss E; Laugks R; Haas R; Fischer W Microbiology (Reading); 2007 Sep; 153(Pt 9):2896-2909. PubMed ID: 17768234 [TBL] [Abstract][Full Text] [Related]
2. Interaction with CagF is required for translocation of CagA into the host via the Helicobacter pylori type IV secretion system. Couturier MR; Tasca E; Montecucco C; Stein M Infect Immun; 2006 Jan; 74(1):273-81. PubMed ID: 16368981 [TBL] [Abstract][Full Text] [Related]
3. Characterization of the translocation-competent complex between the Helicobacter pylori oncogenic protein CagA and the accessory protein CagF. Bonsor DA; Weiss E; Iosub-Amir A; Reingewertz TH; Chen TW; Haas R; Friedler A; Fischer W; Sundberg EJ J Biol Chem; 2013 Nov; 288(46):32897-909. PubMed ID: 24072713 [TBL] [Abstract][Full Text] [Related]
4. The coupling protein Cagbeta and its interaction partner CagZ are required for type IV secretion of the Helicobacter pylori CagA protein. Jurik A; Hausser E; Kutter S; Pattis I; Prassl S; Weiss E; Fischer W Infect Immun; 2010 Dec; 78(12):5244-51. PubMed ID: 20876293 [TBL] [Abstract][Full Text] [Related]
5. CagI is an essential component of the Helicobacter pylori Cag type IV secretion system and forms a complex with CagL. Pham KT; Weiss E; Jiménez Soto LF; Breithaupt U; Haas R; Fischer W PLoS One; 2012; 7(4):e35341. PubMed ID: 22493745 [TBL] [Abstract][Full Text] [Related]
6. A C-terminal translocation signal is necessary, but not sufficient for type IV secretion of the Helicobacter pylori CagA protein. Hohlfeld S; Pattis I; Püls J; Plano GV; Haas R; Fischer W Mol Microbiol; 2006 Mar; 59(5):1624-37. PubMed ID: 16469000 [TBL] [Abstract][Full Text] [Related]
7. Helicobacter pylori chaperone-like protein CagT plays an essential role in the translocation of CagA into host cells. Ding H; Zeng H; Huang L; Dong Y; Duan Y; Mao X; Guo G; Zou Q J Microbiol Biotechnol; 2012 Oct; 22(10):1343-9. PubMed ID: 23075784 [TBL] [Abstract][Full Text] [Related]
8. Kinetics of CagA type IV secretion by Helicobacter pylori and the requirement for substrate unfolding. Lettl C; Haas R; Fischer W Mol Microbiol; 2021 Sep; 116(3):794-807. PubMed ID: 34121254 [TBL] [Abstract][Full Text] [Related]
9. Quantitative analysis of CagA type IV secretion by Helicobacter pylori reveals substrate recognition and translocation requirements. Schindele F; Weiss E; Haas R; Fischer W Mol Microbiol; 2016 Apr; 100(1):188-203. PubMed ID: 26713727 [TBL] [Abstract][Full Text] [Related]
10. Outer inflammatory protein a (OipA) of Helicobacter pylori is regulated by host cell contact and mediates CagA translocation and interleukin-8 response only in the presence of a functional cag pathogenicity island type IV secretion system. Horridge DN; Begley AA; Kim J; Aravindan N; Fan K; Forsyth MH Pathog Dis; 2017 Nov; 75(8):. PubMed ID: 29040466 [TBL] [Abstract][Full Text] [Related]
11. Helicobacter pylori CagA oncoprotein interacts with SHIP2 to increase its delivery into gastric epithelial cells. Fujii Y; Murata-Kamiya N; Hatakeyama M Cancer Sci; 2020 May; 111(5):1596-1606. PubMed ID: 32198795 [TBL] [Abstract][Full Text] [Related]
12. Molecular dissection of protein-protein interactions between integrin α5β1 and the Helicobacter pylori Cag type IV secretion system. Koelblen T; Bergé C; Cherrier MV; Brillet K; Jimenez-Soto L; Ballut L; Takagi J; Montserret R; Rousselle P; Fischer W; Haas R; Fronzes R; Terradot L FEBS J; 2017 Dec; 284(23):4143-4157. PubMed ID: 29055076 [TBL] [Abstract][Full Text] [Related]
16. Translocation of Helicobacter pylori CagA into gastric epithelial cells by type IV secretion. Odenbreit S; Püls J; Sedlmaier B; Gerland E; Fischer W; Haas R Science; 2000 Feb; 287(5457):1497-500. PubMed ID: 10688800 [TBL] [Abstract][Full Text] [Related]
17. Systematic mutagenesis of the Helicobacter pylori cag pathogenicity island: essential genes for CagA translocation in host cells and induction of interleukin-8. Fischer W; Püls J; Buhrdorf R; Gebert B; Odenbreit S; Haas R Mol Microbiol; 2001 Dec; 42(5):1337-48. PubMed ID: 11886563 [TBL] [Abstract][Full Text] [Related]
18. Helicobacter pylori type IV secretion apparatus exploits beta1 integrin in a novel RGD-independent manner. Jiménez-Soto LF; Kutter S; Sewald X; Ertl C; Weiss E; Kapp U; Rohde M; Pirch T; Jung K; Retta SF; Terradot L; Fischer W; Haas R PLoS Pathog; 2009 Dec; 5(12):e1000684. PubMed ID: 19997503 [TBL] [Abstract][Full Text] [Related]
19. Expression of CEACAM1 or CEACAM5 in AZ-521 cells restores the type IV secretion deficiency for translocation of CagA by Helicobacter pylori. Tegtmeyer N; Harrer A; Schmitt V; Singer BB; Backert S Cell Microbiol; 2019 Jan; 21(1):e12965. PubMed ID: 30321907 [TBL] [Abstract][Full Text] [Related]
20. Identification of a tyrosine-phosphorylated 35 kDa carboxy-terminal fragment (p35CagA) of the Helicobacter pylori CagA protein in phagocytic cells: processing or breakage? Moese S; Selbach M; Zimny-Arndt U; Jungblut PR; Meyer TF; Backert S Proteomics; 2001 Apr; 1(4):618-29. PubMed ID: 11681214 [TBL] [Abstract][Full Text] [Related] [Next] [New Search]