134 related articles for article (PubMed ID: 18059343)
1. Differential motility of p190bcr-abl- and p210bcr-abl-expressing cells: respective roles of Vav and Bcr-Abl GEFs.
Daubon T; Chasseriau J; El Ali A; Rivet J; Kitzis A; Constantin B; Bourmeyster N
Oncogene; 2008 Apr; 27(19):2673-85. PubMed ID: 18059343
[TBL] [Abstract][Full Text] [Related]
2. Invadopodia and rolling-type motility are specific features of highly invasive p190(bcr-abl) leukemic cells.
Daubon T; Rochelle T; Bourmeyster N; Génot E
Eur J Cell Biol; 2012; 91(11-12):978-87. PubMed ID: 22717125
[TBL] [Abstract][Full Text] [Related]
3. Differential interaction and activation of Rho family GTPases by p210bcr-abl and p190bcr-abl.
Harnois T; Constantin B; Rioux A; Grenioux E; Kitzis A; Bourmeyster N
Oncogene; 2003 Sep; 22(41):6445-54. PubMed ID: 14508524
[TBL] [Abstract][Full Text] [Related]
4. p210(Bcr-Abl) desensitizes Cdc42 GTPase signaling for SDF-1alpha-directed migration in chronic myeloid leukemia cells.
Chang YC; Tien SC; Tien HF; Zhang H; Bokoch GM; Chang ZF
Oncogene; 2009 Nov; 28(46):4105-15. PubMed ID: 19718053
[TBL] [Abstract][Full Text] [Related]
5. p210bcr-abl induces amoeboid motility by recruiting ADF/destrin through RhoA/ROCK1.
Rochelle T; Daubon T; Van Troys M; Harnois T; Waterschoot D; Ampe C; Roy L; Bourmeyster N; Constantin B
FASEB J; 2013 Jan; 27(1):123-34. PubMed ID: 23047898
[TBL] [Abstract][Full Text] [Related]
6. Phosphorylation and activation of the Rac1 and Cdc42 GEF Asef in A431 cells stimulated by EGF.
Itoh RE; Kiyokawa E; Aoki K; Nishioka T; Akiyama T; Matsuda M
J Cell Sci; 2008 Aug; 121(Pt 16):2635-42. PubMed ID: 18653540
[TBL] [Abstract][Full Text] [Related]
7. BCR-ABL and constitutively active erythropoietin receptor (cEpoR) activate distinct mechanisms for growth factor-independence and inhibition of apoptosis in Ba/F3 cell line.
Ahmed M; Dusanter-Fourt I; Bernard M; Mayeux P; Hawley RG; Bennardo T; Novault S; Bonnet ML; Gisselbrecht S; Varet B; Turhan AG
Oncogene; 1998 Jan; 16(4):489-96. PubMed ID: 9484838
[TBL] [Abstract][Full Text] [Related]
8. p210BCR/ABL, p190BCR/ABL, and TEL/ABL activate similar signal transduction pathways in hematopoietic cell lines.
Okuda K; Golub TR; Gilliland DG; Griffin JD
Oncogene; 1996 Sep; 13(6):1147-52. PubMed ID: 8808688
[TBL] [Abstract][Full Text] [Related]
9. p38 MAPK-mediated activation of NF-kappaB by the RhoGEF domain of Bcr.
Korus M; Mahon GM; Cheng L; Whitehead IP
Oncogene; 2002 Jul; 21(30):4601-12. PubMed ID: 12096337
[TBL] [Abstract][Full Text] [Related]
10. Bcr-Abl has a greater intrinsic capacity than v-Abl to induce the neoplastic expansion of myeloid cells.
Gross AW; Ren R
Oncogene; 2000 Dec; 19(54):6286-96. PubMed ID: 11175343
[TBL] [Abstract][Full Text] [Related]
11. Bcr/Abl P190 interaction with Spa-1, a GTPase activating protein for the small GTPase Rap1.
Yi SJ; Lee HT; Groffen J; Heisterkamp N
Int J Mol Med; 2008 Oct; 22(4):453-8. PubMed ID: 18813851
[TBL] [Abstract][Full Text] [Related]
12. Regulation of RhoA GTP hydrolysis by the GTPase-activating proteins p190, p50RhoGAP, Bcr, and 3BP-1.
Zhang B; Zheng Y
Biochemistry; 1998 Apr; 37(15):5249-57. PubMed ID: 9548756
[TBL] [Abstract][Full Text] [Related]
13. BCR and its mutants, the reciprocal t(9;22)-associated ABL/BCR fusion proteins, differentially regulate the cytoskeleton and cell motility.
Zheng X; Güller S; Beissert T; Puccetti E; Ruthardt M
BMC Cancer; 2006 Nov; 6():262. PubMed ID: 17090304
[TBL] [Abstract][Full Text] [Related]
14. Interleukin-3 and p210 BCR/ABL activate both unique and overlapping pathways of signal transduction in a factor-dependent myeloid cell line.
Matulonis U; Salgia R; Okuda K; Druker B; Griffin JD
Exp Hematol; 1993 Oct; 21(11):1460-6. PubMed ID: 8405226
[TBL] [Abstract][Full Text] [Related]
15. Role of SPA-1 in phenotypes of chronic myelogenous leukemia induced by BCR-ABL-expressing hematopoietic progenitors in a mouse model.
Kometani K; Aoki M; Kawamata S; Shinozuka Y; Era T; Taniwaki M; Hattori M; Minato N
Cancer Res; 2006 Oct; 66(20):9967-76. PubMed ID: 17047059
[TBL] [Abstract][Full Text] [Related]
16. Ribozyme-mediated inhibition of a Philadelphia chromosome-positive acute lymphoblastic leukemia cell line expressing the p190 bcr-abl oncogene.
Snyder DS; Wu Y; McMahon R; Yu L; Rossi JJ; Forman SJ
Biol Blood Marrow Transplant; 1997 Oct; 3(4):179-86. PubMed ID: 9360779
[TBL] [Abstract][Full Text] [Related]
17. Activation of Rac1 and the exchange factor Vav3 are involved in NPM-ALK signaling in anaplastic large cell lymphomas.
Colomba A; Courilleau D; Ramel D; Billadeau DD; Espinos E; Delsol G; Payrastre B; Gaits-Iacovoni F
Oncogene; 2008 Apr; 27(19):2728-36. PubMed ID: 17998938
[TBL] [Abstract][Full Text] [Related]
18. Vav cooperates with Ras to transform rodent fibroblasts but is not a Ras GDP/GTP exchange factor.
Bustelo XR; Suen KL; Leftheris K; Meyers CA; Barbacid M
Oncogene; 1994 Aug; 9(8):2405-13. PubMed ID: 8036025
[TBL] [Abstract][Full Text] [Related]
19. Vav transformation requires activation of multiple GTPases and regulation of gene expression.
Palmby TR; Abe K; Karnoub AE; Der CJ
Mol Cancer Res; 2004 Dec; 2(12):702-11. PubMed ID: 15634759
[TBL] [Abstract][Full Text] [Related]
20. The hematopoietically expressed vav proto-oncogene shares homology with the dbl GDP-GTP exchange factor, the bcr gene and a yeast gene (CDC24) involved in cytoskeletal organization.
Adams JM; Houston H; Allen J; Lints T; Harvey R
Oncogene; 1992 Apr; 7(4):611-8. PubMed ID: 1565462
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]