These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.
283 related articles for article (PubMed ID: 18203851)
1. Modulation of late sodium current by Ca2+, calmodulin, and CaMKII in normal and failing dog cardiomyocytes: similarities and differences. Maltsev VA; Reznikov V; Undrovinas NA; Sabbah HN; Undrovinas A Am J Physiol Heart Circ Physiol; 2008 Apr; 294(4):H1597-608. PubMed ID: 18203851 [TBL] [Abstract][Full Text] [Related]
2. Chronic heart failure slows late sodium current in human and canine ventricular myocytes: implications for repolarization variability. Maltsev VA; Silverman N; Sabbah HN; Undrovinas AI Eur J Heart Fail; 2007 Mar; 9(3):219-27. PubMed ID: 17067855 [TBL] [Abstract][Full Text] [Related]
3. Ranolazine improves abnormal repolarization and contraction in left ventricular myocytes of dogs with heart failure by inhibiting late sodium current. Undrovinas AI; Belardinelli L; Undrovinas NA; Sabbah HN J Cardiovasc Electrophysiol; 2006 May; 17 Suppl 1(Suppl 1):S169-S177. PubMed ID: 16686675 [TBL] [Abstract][Full Text] [Related]
4. Enhanced Depolarization Drive in Failing Rabbit Ventricular Myocytes: Calcium-Dependent and β-Adrenergic Effects on Late Sodium, L-Type Calcium, and Sodium-Calcium Exchange Currents. Hegyi B; Morotti S; Liu C; Ginsburg KS; Bossuyt J; Belardinelli L; Izu LT; Chen-Izu Y; Bányász T; Grandi E; Bers DM Circ Arrhythm Electrophysiol; 2019 Mar; 12(3):e007061. PubMed ID: 30879336 [TBL] [Abstract][Full Text] [Related]
5. Nav1.5-dependent persistent Na+ influx activates CaMKII in rat ventricular myocytes and N1325S mice. Yao L; Fan P; Jiang Z; Viatchenko-Karpinski S; Wu Y; Kornyeyev D; Hirakawa R; Budas GR; Rajamani S; Shryock JC; Belardinelli L Am J Physiol Cell Physiol; 2011 Sep; 301(3):C577-86. PubMed ID: 21677263 [TBL] [Abstract][Full Text] [Related]
6. Calmodulin kinase II and protein kinase C mediate the effect of increased intracellular calcium to augment late sodium current in rabbit ventricular myocytes. Ma J; Luo A; Wu L; Wan W; Zhang P; Ren Z; Zhang S; Qian C; Shryock JC; Belardinelli L Am J Physiol Cell Physiol; 2012 Apr; 302(8):C1141-51. PubMed ID: 22189558 [TBL] [Abstract][Full Text] [Related]
7. Modulation of late sodium current by Ca Fu C; Hao J; Zeng M; Song Y; Jiang W; Zhang P; Luo A; Cao Z; Belardinelli L; Ma J Exp Physiol; 2017 Jul; 102(7):818-834. PubMed ID: 28436159 [TBL] [Abstract][Full Text] [Related]
8. Calcium/calmodulin-dependent protein kinase II contributes to cardiac arrhythmogenesis in heart failure. Sag CM; Wadsack DP; Khabbazzadeh S; Abesser M; Grefe C; Neumann K; Opiela MK; Backs J; Olson EN; Brown JH; Neef S; Maier SK; Maier LS Circ Heart Fail; 2009 Nov; 2(6):664-75. PubMed ID: 19919992 [TBL] [Abstract][Full Text] [Related]
9. Constitutive CaMKII activity regulates Na+ channel in rat ventricular myocytes. Yoon JY; Ho WK; Kim ST; Cho H J Mol Cell Cardiol; 2009 Oct; 47(4):475-84. PubMed ID: 19591836 [TBL] [Abstract][Full Text] [Related]
10. Reactive oxygen species-activated Ca/calmodulin kinase IIδ is required for late I(Na) augmentation leading to cellular Na and Ca overload. Wagner S; Ruff HM; Weber SL; Bellmann S; Sowa T; Schulte T; Anderson ME; Grandi E; Bers DM; Backs J; Belardinelli L; Maier LS Circ Res; 2011 Mar; 108(5):555-65. PubMed ID: 21252154 [TBL] [Abstract][Full Text] [Related]
11. Ca2+/calmodulin-dependent protein kinase II-dependent remodeling of Ca2+ current in pressure overload heart failure. Wang Y; Tandan S; Cheng J; Yang C; Nguyen L; Sugianto J; Johnstone JL; Sun Y; Hill JA J Biol Chem; 2008 Sep; 283(37):25524-25532. PubMed ID: 18622016 [TBL] [Abstract][Full Text] [Related]
12. Enhanced late INa induces proarrhythmogenic SR Ca leak in a CaMKII-dependent manner. Sag CM; Mallwitz A; Wagner S; Hartmann N; Schotola H; Fischer TH; Ungeheuer N; Herting J; Shah AM; Maier LS; Sossalla S; Unsöld B J Mol Cell Cardiol; 2014 Nov; 76():94-105. PubMed ID: 25173923 [TBL] [Abstract][Full Text] [Related]
13. Calcium/calmodulin-dependent protein kinase II (CaMKII) inhibition ameliorates arrhythmias elicited by junctin ablation under stress conditions. Tzimas C; Terrovitis J; Lehnart SE; Kranias EG; Sanoudou D Heart Rhythm; 2015 Jul; 12(7):1599-610. PubMed ID: 25814413 [TBL] [Abstract][Full Text] [Related]
14. Simulation of Ca-calmodulin-dependent protein kinase II on rabbit ventricular myocyte ion currents and action potentials. Grandi E; Puglisi JL; Wagner S; Maier LS; Severi S; Bers DM Biophys J; 2007 Dec; 93(11):3835-47. PubMed ID: 17704163 [TBL] [Abstract][Full Text] [Related]
15. Post-transcriptional silencing of SCN1B and SCN2B genes modulates late sodium current in cardiac myocytes from normal dogs and dogs with chronic heart failure. Mishra S; Undrovinas NA; Maltsev VA; Reznikov V; Sabbah HN; Undrovinas A Am J Physiol Heart Circ Physiol; 2011 Oct; 301(4):H1596-605. PubMed ID: 21705762 [TBL] [Abstract][Full Text] [Related]
16. Role of late sodium current as a potential arrhythmogenic mechanism in the progression of pressure-induced heart disease. Toischer K; Hartmann N; Wagner S; Fischer TH; Herting J; Danner BC; Sag CM; Hund TJ; Mohler PJ; Belardinelli L; Hasenfuss G; Maier LS; Sossalla S J Mol Cell Cardiol; 2013 Aug; 61():111-22. PubMed ID: 23570977 [TBL] [Abstract][Full Text] [Related]