These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.


BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

217 related articles for article (PubMed ID: 18337307)

  • 1. Biological effects of CCS in the absence of SOD1 enzyme activation: implications for disease in a mouse model for ALS.
    Proescher JB; Son M; Elliott JL; Culotta VC
    Hum Mol Genet; 2008 Jun; 17(12):1728-37. PubMed ID: 18337307
    [TBL] [Abstract][Full Text] [Related]  

  • 2. Immature copper-zinc superoxide dismutase and familial amyotrophic lateral sclerosis.
    Seetharaman SV; Prudencio M; Karch C; Holloway SP; Borchelt DR; Hart PJ
    Exp Biol Med (Maywood); 2009 Oct; 234(10):1140-54. PubMed ID: 19596823
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Histological evidence of protein aggregation in mutant SOD1 transgenic mice and in amyotrophic lateral sclerosis neural tissues.
    Watanabe M; Dykes-Hoberg M; Culotta VC; Price DL; Wong PC; Rothstein JD
    Neurobiol Dis; 2001 Dec; 8(6):933-41. PubMed ID: 11741389
    [TBL] [Abstract][Full Text] [Related]  

  • 4. Different regulation of wild-type and mutant Cu,Zn superoxide dismutase localization in mammalian mitochondria.
    Kawamata H; Manfredi G
    Hum Mol Genet; 2008 Nov; 17(21):3303-17. PubMed ID: 18703498
    [TBL] [Abstract][Full Text] [Related]  

  • 5. Copper chaperone for superoxide dismutase co-aggregates with superoxide dismutase 1 (SOD1) in neuronal Lewy body-like hyaline inclusions: an immunohistochemical study on familial amyotrophic lateral sclerosis with SOD1 gene mutation.
    Kato S; Sumi-Akamaru H; Fujimura H; Sakoda S; Kato M; Hirano A; Takikawa M; Ohama E
    Acta Neuropathol; 2001 Sep; 102(3):233-8. PubMed ID: 11585247
    [TBL] [Abstract][Full Text] [Related]  

  • 6. Over-expression of Hsp27 does not influence disease in the mutant SOD1(G93A) mouse model of amyotrophic lateral sclerosis.
    Krishnan J; Vannuvel K; Andries M; Waelkens E; Robberecht W; Van Den Bosch L
    J Neurochem; 2008 Sep; 106(5):2170-83. PubMed ID: 18624915
    [TBL] [Abstract][Full Text] [Related]  

  • 7. Molecular chaperone mediated late-stage neuroprotection in the SOD1(G93A) mouse model of amyotrophic lateral sclerosis.
    Novoselov SS; Mustill WJ; Gray AL; Dick JR; Kanuga N; Kalmar B; Greensmith L; Cheetham ME
    PLoS One; 2013; 8(8):e73944. PubMed ID: 24023695
    [TBL] [Abstract][Full Text] [Related]  

  • 8. Differential effects of mutant SOD1 on protein structure of skeletal muscle and spinal cord of familial amyotrophic lateral sclerosis: role of chaperone network.
    Wei R; Bhattacharya A; Hamilton RT; Jernigan AL; Chaudhuri AR
    Biochem Biophys Res Commun; 2013 Aug; 438(1):218-23. PubMed ID: 23886956
    [TBL] [Abstract][Full Text] [Related]  

  • 9. Posttranslational modifications in Cu,Zn-superoxide dismutase and mutations associated with amyotrophic lateral sclerosis.
    Furukawa Y; O'Halloran TV
    Antioxid Redox Signal; 2006; 8(5-6):847-67. PubMed ID: 16771675
    [TBL] [Abstract][Full Text] [Related]  

  • 10. Oxygen-induced maturation of SOD1: a key role for disulfide formation by the copper chaperone CCS.
    Furukawa Y; Torres AS; O'Halloran TV
    EMBO J; 2004 Jul; 23(14):2872-81. PubMed ID: 15215895
    [TBL] [Abstract][Full Text] [Related]  

  • 11. Effect of CCS on the accumulation of FALS SOD1 mutant-containing aggregates and on mitochondrial translocation of SOD1 mutants: implication of a free radical hypothesis.
    Kim HK; Chung YW; Chock PB; Yim MB
    Arch Biochem Biophys; 2011 May; 509(2):177-85. PubMed ID: 21354101
    [TBL] [Abstract][Full Text] [Related]  

  • 12. Copper delivery to the CNS by CuATSM effectively treats motor neuron disease in SOD(G93A) mice co-expressing the Copper-Chaperone-for-SOD.
    Williams JR; Trias E; Beilby PR; Lopez NI; Labut EM; Bradford CS; Roberts BR; McAllum EJ; Crouch PJ; Rhoads TW; Pereira C; Son M; Elliott JL; Franco MC; Estévez AG; Barbeito L; Beckman JS
    Neurobiol Dis; 2016 May; 89():1-9. PubMed ID: 26826269
    [TBL] [Abstract][Full Text] [Related]  

  • 13. Mechanisms for activating Cu- and Zn-containing superoxide dismutase in the absence of the CCS Cu chaperone.
    Carroll MC; Girouard JB; Ulloa JL; Subramaniam JR; Wong PC; Valentine JS; Culotta VC
    Proc Natl Acad Sci U S A; 2004 Apr; 101(16):5964-9. PubMed ID: 15069187
    [TBL] [Abstract][Full Text] [Related]  

  • 14. Import, maturation, and function of SOD1 and its copper chaperone CCS in the mitochondrial intermembrane space.
    Kawamata H; Manfredi G
    Antioxid Redox Signal; 2010 Nov; 13(9):1375-84. PubMed ID: 20367259
    [TBL] [Abstract][Full Text] [Related]  

  • 15. Copper chaperone for superoxide dismutase is essential to activate mammalian Cu/Zn superoxide dismutase.
    Wong PC; Waggoner D; Subramaniam JR; Tessarollo L; Bartnikas TB; Culotta VC; Price DL; Rothstein J; Gitlin JD
    Proc Natl Acad Sci U S A; 2000 Mar; 97(6):2886-91. PubMed ID: 10694572
    [TBL] [Abstract][Full Text] [Related]  

  • 16. Mutant SOD1 causes motor neuron disease independent of copper chaperone-mediated copper loading.
    Subramaniam JR; Lyons WE; Liu J; Bartnikas TB; Rothstein J; Price DL; Cleveland DW; Gitlin JD; Wong PC
    Nat Neurosci; 2002 Apr; 5(4):301-7. PubMed ID: 11889469
    [TBL] [Abstract][Full Text] [Related]  

  • 17. S-acylation of SOD1, CCS, and a stable SOD1-CCS heterodimer in human spinal cords from ALS and non-ALS subjects.
    Antinone SE; Ghadge GD; Ostrow LW; Roos RP; Green WN
    Sci Rep; 2017 Jan; 7():41141. PubMed ID: 28120938
    [TBL] [Abstract][Full Text] [Related]  

  • 18. Overexpression of Abeta is associated with acceleration of onset of motor impairment and superoxide dismutase 1 aggregation in an amyotrophic lateral sclerosis mouse model.
    Li QX; Mok SS; Laughton KM; McLean CA; Volitakis I; Cherny RA; Cheung NS; White AR; Masters CL
    Aging Cell; 2006 Apr; 5(2):153-65. PubMed ID: 16626394
    [TBL] [Abstract][Full Text] [Related]  

  • 19. Complete loss of post-translational modifications triggers fibrillar aggregation of SOD1 in the familial form of amyotrophic lateral sclerosis.
    Furukawa Y; Kaneko K; Yamanaka K; O'Halloran TV; Nukina N
    J Biol Chem; 2008 Aug; 283(35):24167-76. PubMed ID: 18552350
    [TBL] [Abstract][Full Text] [Related]  

  • 20. Overexpression of metallothionein-I, a copper-regulating protein, attenuates intracellular copper dyshomeostasis and extends lifespan in a mouse model of amyotrophic lateral sclerosis caused by mutant superoxide dismutase-1.
    Tokuda E; Okawa E; Watanabe S; Ono S
    Hum Mol Genet; 2014 Mar; 23(5):1271-85. PubMed ID: 24163136
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 11.