308 related articles for article (PubMed ID: 18487447)
1. ACTH-induced hypertension is dependent on the ouabain-binding site of the alpha2-Na+-K+-ATPase subunit.
Lorenz JN; Loreaux EL; Dostanic-Larson I; Lasko V; Schnetzer JR; Paul RJ; Lingrel JB
Am J Physiol Heart Circ Physiol; 2008 Jul; 295(1):H273-80. PubMed ID: 18487447
[TBL] [Abstract][Full Text] [Related]
2. Hypertension from chronic central sodium chloride in mice is mediated by the ouabain-binding site on the Na,K-ATPase α₂-isoform.
Van Huysse JW; Dostanic I; Lingrel JB; Hou X; Wu H
Am J Physiol Heart Circ Physiol; 2011 Nov; 301(5):H2147-53. PubMed ID: 21856907
[TBL] [Abstract][Full Text] [Related]
3. The ouabain-binding site of the α2 isoform of Na,K-ATPase plays a role in blood pressure regulation during pregnancy.
Oshiro N; Dostanic-Larson I; Neumann JC; Lingrel JB
Am J Hypertens; 2010 Dec; 23(12):1279-85. PubMed ID: 20940714
[TBL] [Abstract][Full Text] [Related]
4. Renovascular hypertension using a modified two-kidney, one-clip approach in mice is not dependent on the α1 or α2 Na-K-ATPase ouabain-binding site.
Lorenz JN; Lasko VM; Nieman ML; Damhoff T; Prasad V; Beierwaltes WH; Lingrel JB
Am J Physiol Renal Physiol; 2011 Sep; 301(3):F615-21. PubMed ID: 21632957
[TBL] [Abstract][Full Text] [Related]
5. Mice expressing ouabain-sensitive α1-Na,K-ATPase have increased susceptibility to pressure overload-induced cardiac hypertrophy.
Wansapura AN; Lasko VM; Lingrel JB; Lorenz JN
Am J Physiol Heart Circ Physiol; 2011 Jan; 300(1):H347-55. PubMed ID: 20952666
[TBL] [Abstract][Full Text] [Related]
6. DOCA-salt hypertension does not require the ouabain-sensitive binding site of the α2 Na,K-ATPase.
Lorenz JN; Oshiro N; Loreaux EL; Lingrel JB
Am J Hypertens; 2012 Apr; 25(4):421-9. PubMed ID: 22258333
[TBL] [Abstract][Full Text] [Related]
7. The highly conserved cardiac glycoside binding site of Na,K-ATPase plays a role in blood pressure regulation.
Dostanic-Larson I; Van Huysse JW; Lorenz JN; Lingrel JB
Proc Natl Acad Sci U S A; 2005 Nov; 102(44):15845-50. PubMed ID: 16243970
[TBL] [Abstract][Full Text] [Related]
8. The alpha2-isoform of Na-K-ATPase mediates ouabain-induced hypertension in mice and increased vascular contractility in vitro.
Dostanic I; Paul RJ; Lorenz JN; Theriault S; Van Huysse JW; Lingrel JB
Am J Physiol Heart Circ Physiol; 2005 Feb; 288(2):H477-85. PubMed ID: 15458945
[TBL] [Abstract][Full Text] [Related]
9. Physiological role of the alpha1- and alpha2-isoforms of the Na+-K+-ATPase and biological significance of their cardiac glycoside binding site.
Dostanic-Larson I; Lorenz JN; Van Huysse JW; Neumann JC; Moseley AE; Lingrel JB
Am J Physiol Regul Integr Comp Physiol; 2006 Mar; 290(3):R524-8. PubMed ID: 16467499
[TBL] [Abstract][Full Text] [Related]
10. The change and significance of the Na+-K+-ATPase alpha-subunit in ouabain-hypertensive rats.
Tian G; Dang C; Lu Z
Hypertens Res; 2001 Nov; 24(6):729-34. PubMed ID: 11768735
[TBL] [Abstract][Full Text] [Related]
11. The α2 isoform Na,K-ATPase modulates contraction of rat mesenteric small artery via cSrc-dependent Ca
Bouzinova EV; Hangaard L; Staehr C; Mazur A; Ferreira A; Chibalin AV; Sandow SL; Xie Z; Aalkjaer C; Matchkov VV
Acta Physiol (Oxf); 2018 Sep; 224(1):e13059. PubMed ID: 29480968
[TBL] [Abstract][Full Text] [Related]
12. Marinobufagenin enhances cardiac contractility in mice with ouabain-sensitive alpha1 Na+-K+-ATPase.
Wansapura AN; Lasko V; Xie Z; Fedorova OV; Bagrov AY; Lingrel JB; Lorenz JN
Am J Physiol Heart Circ Physiol; 2009 Jun; 296(6):H1833-9. PubMed ID: 19376809
[TBL] [Abstract][Full Text] [Related]
13. How does pressure overload cause cardiac hypertrophy and dysfunction? High-ouabain affinity cardiac Na
Blaustein MP
Am J Physiol Heart Circ Physiol; 2017 Nov; 313(5):H919-H930. PubMed ID: 28733446
[TBL] [Abstract][Full Text] [Related]
14. Targeted mutations in the Na,K-ATPase α 2 isoform confer ouabain resistance and result in abnormal behavior in mice.
Schaefer TL; Lingrel JB; Moseley AE; Vorhees CV; Williams MT
Synapse; 2011 Jun; 65(6):520-31. PubMed ID: 20936682
[TBL] [Abstract][Full Text] [Related]
15. Enhanced pressor response to increased CSF sodium concentration and to central ANG I in heterozygous alpha2 Na+ -K+ -ATPase knockout mice.
Hou X; Theriault SF; Dostanic-Larson I; Moseley AE; Lingrel JB; Wu H; Dean S; Van Huysse JW
Am J Physiol Regul Integr Comp Physiol; 2009 May; 296(5):R1427-38. PubMed ID: 19244589
[TBL] [Abstract][Full Text] [Related]
16. Cardiac Oxidative Signaling and Physiological Hypertrophy in the Na/K-ATPase α1
Marck PV; Pessoa MT; Xu Y; Kutz LC; Collins DM; Yan Y; King C; Wang X; Duan Q; Cai L; Xie JX; Lingrel JB; Xie Z; Tian J; Pierre SV
Int J Mol Sci; 2021 Mar; 22(7):. PubMed ID: 33801629
[TBL] [Abstract][Full Text] [Related]
17. Increased constrictor tone induced by ouabain treatment in rats.
Pulgar VM; Jeffers AB; Rashad HM; Diz DI; Aileru AA
J Cardiovasc Pharmacol; 2013 Aug; 62(2):174-83. PubMed ID: 23615157
[TBL] [Abstract][Full Text] [Related]
18. Na(+)/K)+)-ATPase α2-isoform preferentially modulates Ca2(+) transients and sarcoplasmic reticulum Ca2(+) release in cardiac myocytes.
Despa S; Lingrel JB; Bers DM
Cardiovasc Res; 2012 Sep; 95(4):480-6. PubMed ID: 22739122
[TBL] [Abstract][Full Text] [Related]
19. Pivotal role of α2 Na
Blaustein MP; Chen L; Hamlyn JM; Leenen FH; Lingrel JB; Wier WG; Zhang J
J Physiol; 2016 Nov; 594(21):6079-6103. PubMed ID: 27350568
[TBL] [Abstract][Full Text] [Related]
20. Nanomolar ouabain increases NCX1 expression and enhances Ca2+ signaling in human arterial myocytes: a mechanism that links salt to increased vascular resistance?
Linde CI; Antos LK; Golovina VA; Blaustein MP
Am J Physiol Heart Circ Physiol; 2012 Oct; 303(7):H784-94. PubMed ID: 22842068
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]