BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

215 related articles for article (PubMed ID: 18558628)

  • 1. High-mobility group box 1 restores cardiac function after myocardial infarction in transgenic mice.
    Kitahara T; Takeishi Y; Harada M; Niizeki T; Suzuki S; Sasaki T; Ishino M; Bilim O; Nakajima O; Kubota I
    Cardiovasc Res; 2008 Oct; 80(1):40-6. PubMed ID: 18558628
    [TBL] [Abstract][Full Text] [Related]  

  • 2. High Mobility Group Box 1 Promotes Angiogenesis from Bone Marrow-derived Endothelial Progenitor Cells after Myocardial Infarction.
    Nakamura Y; Suzuki S; Shimizu T; Miyata M; Shishido T; Ikeda K; Saitoh S; Kubota I; Takeishi Y
    J Atheroscler Thromb; 2015; 22(6):570-81. PubMed ID: 25735431
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Cardiac overexpression of monocyte chemoattractant protein-1 in transgenic mice prevents cardiac dysfunction and remodeling after myocardial infarction.
    Morimoto H; Takahashi M; Izawa A; Ise H; Hongo M; Kolattukudy PE; Ikeda U
    Circ Res; 2006 Oct; 99(8):891-9. PubMed ID: 16990567
    [TBL] [Abstract][Full Text] [Related]  

  • 4. Transgenic alpha1A-adrenergic activation limits post-infarct ventricular remodeling and dysfunction and improves survival.
    Du XJ; Gao XM; Kiriazis H; Moore XL; Ming Z; Su Y; Finch AM; Hannan RA; Dart AM; Graham RM
    Cardiovasc Res; 2006 Sep; 71(4):735-43. PubMed ID: 16859660
    [TBL] [Abstract][Full Text] [Related]  

  • 5. Cardiomyocyte-specific overexpression of human stem cell factor improves cardiac function and survival after myocardial infarction in mice.
    Xiang FL; Lu X; Hammoud L; Zhu P; Chidiac P; Robbins J; Feng Q
    Circulation; 2009 Sep; 120(12):1065-74, 9 p following 1074. PubMed ID: 19738140
    [TBL] [Abstract][Full Text] [Related]  

  • 6. Overexpression of glutathione peroxidase prevents left ventricular remodeling and failure after myocardial infarction in mice.
    Shiomi T; Tsutsui H; Matsusaka H; Murakami K; Hayashidani S; Ikeuchi M; Wen J; Kubota T; Utsumi H; Takeshita A
    Circulation; 2004 Feb; 109(4):544-9. PubMed ID: 14744974
    [TBL] [Abstract][Full Text] [Related]  

  • 7. Cardiac S100A1 protein levels determine contractile performance and propensity toward heart failure after myocardial infarction.
    Most P; Seifert H; Gao E; Funakoshi H; Völkers M; Heierhorst J; Remppis A; Pleger ST; DeGeorge BR; Eckhart AD; Feldman AM; Koch WJ
    Circulation; 2006 Sep; 114(12):1258-68. PubMed ID: 16952982
    [TBL] [Abstract][Full Text] [Related]  

  • 8. Role of high-mobility group box 1 protein in post-infarction healing process and left ventricular remodelling.
    Kohno T; Anzai T; Naito K; Miyasho T; Okamoto M; Yokota H; Yamada S; Maekawa Y; Takahashi T; Yoshikawa T; Ishizaka A; Ogawa S
    Cardiovasc Res; 2009 Feb; 81(3):565-73. PubMed ID: 18984601
    [TBL] [Abstract][Full Text] [Related]  

  • 9. Cellular FLICE-inhibitory protein protects against cardiac remodelling after myocardial infarction.
    Xiao J; Moon M; Yan L; Nian M; Zhang Y; Liu C; Lu J; Guan H; Chen M; Jiang D; Jiang H; Liu PP; Li H
    Basic Res Cardiol; 2012 Jan; 107(1):239. PubMed ID: 22202974
    [TBL] [Abstract][Full Text] [Related]  

  • 10. Overexpression of mitochondrial peroxiredoxin-3 prevents left ventricular remodeling and failure after myocardial infarction in mice.
    Matsushima S; Ide T; Yamato M; Matsusaka H; Hattori F; Ikeuchi M; Kubota T; Sunagawa K; Hasegawa Y; Kurihara T; Oikawa S; Kinugawa S; Tsutsui H
    Circulation; 2006 Apr; 113(14):1779-86. PubMed ID: 16585391
    [TBL] [Abstract][Full Text] [Related]  

  • 11. [Time course change in the high mobility group box-1 after myocardial infarction in rats].
    Wang XW; Zhang WD; Wang XL; Li J; Yuan BB
    Nan Fang Yi Ke Da Xue Xue Bao; 2008 Aug; 28(9):1688-90. PubMed ID: 18819896
    [TBL] [Abstract][Full Text] [Related]  

  • 12. HSPA12B attenuates cardiac dysfunction and remodelling after myocardial infarction through an eNOS-dependent mechanism.
    Li J; Zhang Y; Li C; Xie J; Liu Y; Zhu W; Zhang X; Jiang S; Liu L; Ding Z
    Cardiovasc Res; 2013 Sep; 99(4):674-84. PubMed ID: 23729663
    [TBL] [Abstract][Full Text] [Related]  

  • 13. Exogenous high-mobility group box 1 protein induces myocardial regeneration after infarction via enhanced cardiac C-kit+ cell proliferation and differentiation.
    Limana F; Germani A; Zacheo A; Kajstura J; Di Carlo A; Borsellino G; Leoni O; Palumbo R; Battistini L; Rastaldo R; Müller S; Pompilio G; Anversa P; Bianchi ME; Capogrossi MC
    Circ Res; 2005 Oct; 97(8):e73-83. PubMed ID: 16166556
    [TBL] [Abstract][Full Text] [Related]  

  • 14. CHIP enhances angiogenesis and restores cardiac function after infarction in transgenic mice.
    Xu CW; Zhang TP; Wang HX; Yang H; Li HH
    Cell Physiol Biochem; 2013; 31(2-3):199-208. PubMed ID: 23485987
    [TBL] [Abstract][Full Text] [Related]  

  • 15. Cardiac-specific overexpression of diacylglycerol kinase zeta prevents Gq protein-coupled receptor agonist-induced cardiac hypertrophy in transgenic mice.
    Arimoto T; Takeishi Y; Takahashi H; Shishido T; Niizeki T; Koyama Y; Shiga R; Nozaki N; Nakajima O; Nishimaru K; Abe J; Endoh M; Walsh RA; Goto K; Kubota I
    Circulation; 2006 Jan; 113(1):60-6. PubMed ID: 16380548
    [TBL] [Abstract][Full Text] [Related]  

  • 16. DPP4 Inhibition Ameliorates Cardiac Function by Blocking the Cleavage of HMGB1 in Diabetic Mice After Myocardial Infarction.
    Sato A; Suzuki S; Watanabe S; Shimizu T; Nakamura Y; Misaka T; Yokokawa T; Shishido T; Saitoh SI; Ishida T; Kubota I; Takeishi Y
    Int Heart J; 2017 Oct; 58(5):778-786. PubMed ID: 28966327
    [TBL] [Abstract][Full Text] [Related]  

  • 17. Overexpression of mitochondrial transcription factor a ameliorates mitochondrial deficiencies and cardiac failure after myocardial infarction.
    Ikeuchi M; Matsusaka H; Kang D; Matsushima S; Ide T; Kubota T; Fujiwara T; Hamasaki N; Takeshita A; Sunagawa K; Tsutsui H
    Circulation; 2005 Aug; 112(5):683-90. PubMed ID: 16043643
    [TBL] [Abstract][Full Text] [Related]  

  • 18. Hypoxia-inducible factor 1-alpha reduces infarction and attenuates progression of cardiac dysfunction after myocardial infarction in the mouse.
    Kido M; Du L; Sullivan CC; Li X; Deutsch R; Jamieson SW; Thistlethwaite PA
    J Am Coll Cardiol; 2005 Dec; 46(11):2116-24. PubMed ID: 16325051
    [TBL] [Abstract][Full Text] [Related]  

  • 19. TIMP-3 deficiency accelerates cardiac remodeling after myocardial infarction.
    Tian H; Cimini M; Fedak PW; Altamentova S; Fazel S; Huang ML; Weisel RD; Li RK
    J Mol Cell Cardiol; 2007 Dec; 43(6):733-43. PubMed ID: 17945252
    [TBL] [Abstract][Full Text] [Related]  

  • 20. Inhibition of endogenous Mst1 prevents apoptosis and cardiac dysfunction without affecting cardiac hypertrophy after myocardial infarction.
    Odashima M; Usui S; Takagi H; Hong C; Liu J; Yokota M; Sadoshima J
    Circ Res; 2007 May; 100(9):1344-52. PubMed ID: 17395874
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 11.