461 related articles for article (PubMed ID: 18664457)
1. Replication independent ATR signalling leads to G2/M arrest requiring Nbs1, 53BP1 and MDC1.
Stiff T; Cerosaletti K; Concannon P; O'Driscoll M; Jeggo PA
Hum Mol Genet; 2008 Oct; 17(20):3247-53. PubMed ID: 18664457
[TBL] [Abstract][Full Text] [Related]
2. Nbs1 is required for ATR-dependent phosphorylation events.
Stiff T; Reis C; Alderton GK; Woodbine L; O'Driscoll M; Jeggo PA
EMBO J; 2005 Jan; 24(1):199-208. PubMed ID: 15616588
[TBL] [Abstract][Full Text] [Related]
3. Role of ATM and the damage response mediator proteins 53BP1 and MDC1 in the maintenance of G(2)/M checkpoint arrest.
Shibata A; Barton O; Noon AT; Dahm K; Deckbar D; Goodarzi AA; Löbrich M; Jeggo PA
Mol Cell Biol; 2010 Jul; 30(13):3371-83. PubMed ID: 20421415
[TBL] [Abstract][Full Text] [Related]
4. ATR-dependent phosphorylation and activation of ATM in response to UV treatment or replication fork stalling.
Stiff T; Walker SA; Cerosaletti K; Goodarzi AA; Petermann E; Concannon P; O'Driscoll M; Jeggo PA
EMBO J; 2006 Dec; 25(24):5775-82. PubMed ID: 17124492
[TBL] [Abstract][Full Text] [Related]
5. MDC1 is a mediator of the mammalian DNA damage checkpoint.
Stewart GS; Wang B; Bignell CR; Taylor AM; Elledge SJ
Nature; 2003 Feb; 421(6926):961-6. PubMed ID: 12607005
[TBL] [Abstract][Full Text] [Related]
6. STAT-1 facilitates the ATM activated checkpoint pathway following DNA damage.
Townsend PA; Cragg MS; Davidson SM; McCormick J; Barry S; Lawrence KM; Knight RA; Hubank M; Chen PL; Latchman DS; Stephanou A
J Cell Sci; 2005 Apr; 118(Pt 8):1629-39. PubMed ID: 15784679
[TBL] [Abstract][Full Text] [Related]
7. Cep164 is a mediator protein required for the maintenance of genomic stability through modulation of MDC1, RPA, and CHK1.
Sivasubramaniam S; Sun X; Pan YR; Wang S; Lee EY
Genes Dev; 2008 Mar; 22(5):587-600. PubMed ID: 18283122
[TBL] [Abstract][Full Text] [Related]
8. Nucleotide excision repair-induced H2A ubiquitination is dependent on MDC1 and RNF8 and reveals a universal DNA damage response.
Marteijn JA; Bekker-Jensen S; Mailand N; Lans H; Schwertman P; Gourdin AM; Dantuma NP; Lukas J; Vermeulen W
J Cell Biol; 2009 Sep; 186(6):835-47. PubMed ID: 19797077
[TBL] [Abstract][Full Text] [Related]
9. Growth of persistent foci of DNA damage checkpoint factors is essential for amplification of G1 checkpoint signaling.
Yamauchi M; Oka Y; Yamamoto M; Niimura K; Uchida M; Kodama S; Watanabe M; Sekine I; Yamashita S; Suzuki K
DNA Repair (Amst); 2008 Mar; 7(3):405-17. PubMed ID: 18248856
[TBL] [Abstract][Full Text] [Related]
10. Distinct roles of ATR and DNA-PKcs in triggering DNA damage responses in ATM-deficient cells.
Tomimatsu N; Mukherjee B; Burma S
EMBO Rep; 2009 Jun; 10(6):629-35. PubMed ID: 19444312
[TBL] [Abstract][Full Text] [Related]
11. RNF8-dependent and RNF8-independent regulation of 53BP1 in response to DNA damage.
Sakasai R; Tibbetts R
J Biol Chem; 2008 May; 283(20):13549-55. PubMed ID: 18337245
[TBL] [Abstract][Full Text] [Related]
12. Wild-type p53-induced phosphatase 1 dephosphorylates histone variant gamma-H2AX and suppresses DNA double strand break repair.
Moon SH; Lin L; Zhang X; Nguyen TA; Darlington Y; Waldman AS; Lu X; Donehower LA
J Biol Chem; 2010 Apr; 285(17):12935-47. PubMed ID: 20118229
[TBL] [Abstract][Full Text] [Related]
13. The Mre11-Rad50-Nbs1 complex acts both upstream and downstream of ataxia telangiectasia mutated and Rad3-related protein (ATR) to regulate the S-phase checkpoint following UV treatment.
Olson E; Nievera CJ; Lee AY; Chen L; Wu X
J Biol Chem; 2007 Aug; 282(31):22939-52. PubMed ID: 17526493
[TBL] [Abstract][Full Text] [Related]
14. Suppression of Tousled-like kinase activity after DNA damage or replication block requires ATM, NBS1 and Chk1.
Krause DR; Jonnalagadda JC; Gatei MH; Sillje HH; Zhou BB; Nigg EA; Khanna K
Oncogene; 2003 Sep; 22(38):5927-37. PubMed ID: 12955071
[TBL] [Abstract][Full Text] [Related]
15. Opposing effects of the UV lesion repair protein XPA and UV bypass polymerase eta on ATR checkpoint signaling.
Bomgarden RD; Lupardus PJ; Soni DV; Yee MC; Ford JM; Cimprich KA
EMBO J; 2006 Jun; 25(11):2605-14. PubMed ID: 16675950
[TBL] [Abstract][Full Text] [Related]
16. Conserved modes of recruitment of ATM, ATR and DNA-PKcs to sites of DNA damage.
Falck J; Coates J; Jackson SP
Nature; 2005 Mar; 434(7033):605-11. PubMed ID: 15758953
[TBL] [Abstract][Full Text] [Related]
17. 53BP1-dependent robust localized KAP-1 phosphorylation is essential for heterochromatic DNA double-strand break repair.
Noon AT; Shibata A; Rief N; Löbrich M; Stewart GS; Jeggo PA; Goodarzi AA
Nat Cell Biol; 2010 Feb; 12(2):177-84. PubMed ID: 20081839
[TBL] [Abstract][Full Text] [Related]
18. Nucleotide excision repair-dependent DNA double-strand break formation and ATM signaling activation in mammalian quiescent cells.
Wakasugi M; Sasaki T; Matsumoto M; Nagaoka M; Inoue K; Inobe M; Horibata K; Tanaka K; Matsunaga T
J Biol Chem; 2014 Oct; 289(41):28730-7. PubMed ID: 25164823
[TBL] [Abstract][Full Text] [Related]
19. Distinct functions of Nijmegen breakage syndrome in ataxia telangiectasia mutated-dependent responses to DNA damage.
Lee JH; Xu B; Lee CH; Ahn JY; Song MS; Lee H; Canman CE; Lee JS; Kastan MB; Lim DS
Mol Cancer Res; 2003 Jul; 1(9):674-81. PubMed ID: 12861053
[TBL] [Abstract][Full Text] [Related]
20. 53BP1 and NFBD1/MDC1-Nbs1 function in parallel interacting pathways activating ataxia-telangiectasia mutated (ATM) in response to DNA damage.
Mochan TA; Venere M; DiTullio RA; Halazonetis TD
Cancer Res; 2003 Dec; 63(24):8586-91. PubMed ID: 14695167
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]