BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

750 related articles for article (PubMed ID: 18678774)

  • 1. Improvement in left ventricular remodeling by the endothelial nitric oxide synthase enhancer AVE9488 after experimental myocardial infarction.
    Fraccarollo D; Widder JD; Galuppo P; Thum T; Tsikas D; Hoffmann M; Ruetten H; Ertl G; Bauersachs J
    Circulation; 2008 Aug; 118(8):818-27. PubMed ID: 18678774
    [TBL] [Abstract][Full Text] [Related]  

  • 2. Human endothelial nitric oxide synthase gene delivery protects against cardiac remodeling and reduces oxidative stress after myocardial infarction.
    Smith RS; Agata J; Xia CF; Chao L; Chao J
    Life Sci; 2005 Apr; 76(21):2457-71. PubMed ID: 15763077
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Valsartan therapy in heart failure after myocardial infarction: the role of endothelial dependent vasorelaxation.
    Thai H; Guarraia D; Johnson N; Goldman S; Gaballa MA
    J Cardiovasc Pharmacol; 2007 Dec; 50(6):703-7. PubMed ID: 18091589
    [TBL] [Abstract][Full Text] [Related]  

  • 4. Inhibition of TGF-beta1 signaling by eNOS gene transfer improves ventricular remodeling after myocardial infarction through angiogenesis and reduction of apoptosis.
    Chen LL; Yin H; Huang J
    Cardiovasc Pathol; 2007; 16(4):221-30. PubMed ID: 17637430
    [TBL] [Abstract][Full Text] [Related]  

  • 5. Central infusion of aldosterone synthase inhibitor attenuates left ventricular dysfunction and remodelling in rats after myocardial infarction.
    Huang BS; White RA; Ahmad M; Tan J; Jeng AY; Leenen FH
    Cardiovasc Res; 2009 Feb; 81(3):574-81. PubMed ID: 18689429
    [TBL] [Abstract][Full Text] [Related]  

  • 6. Simvastatin improves left ventricular function after myocardial infarction in hypercholesterolemic rabbits by anti-inflammatory effects.
    Bauersachs J; Hiss K; Fraccarollo D; Laufs U; Ruetten H
    Cardiovasc Res; 2006 Dec; 72(3):438-46. PubMed ID: 17026973
    [TBL] [Abstract][Full Text] [Related]  

  • 7. Antiatherosclerotic effects of small-molecular-weight compounds enhancing endothelial nitric-oxide synthase (eNOS) expression and preventing eNOS uncoupling.
    Wohlfart P; Xu H; Endlich A; Habermeier A; Closs EI; Hübschle T; Mang C; Strobel H; Suzuki T; Kleinert H; Förstermann U; Ruetten H; Li H
    J Pharmacol Exp Ther; 2008 May; 325(2):370-9. PubMed ID: 18252813
    [TBL] [Abstract][Full Text] [Related]  

  • 8. p38 mitogen-activated protein kinase inhibition improves cardiac function and attenuates left ventricular remodeling following myocardial infarction in the rat.
    See F; Thomas W; Way K; Tzanidis A; Kompa A; Lewis D; Itescu S; Krum H
    J Am Coll Cardiol; 2004 Oct; 44(8):1679-89. PubMed ID: 15489104
    [TBL] [Abstract][Full Text] [Related]  

  • 9. Beneficial effects of exercise training after myocardial infarction require full eNOS expression.
    de Waard MC; van Haperen R; Soullié T; Tempel D; de Crom R; Duncker DJ
    J Mol Cell Cardiol; 2010 Jun; 48(6):1041-9. PubMed ID: 20153335
    [TBL] [Abstract][Full Text] [Related]  

  • 10. Anti-monocyte chemoattractant protein-1 gene therapy attenuates left ventricular remodeling and failure after experimental myocardial infarction.
    Hayashidani S; Tsutsui H; Shiomi T; Ikeuchi M; Matsusaka H; Suematsu N; Wen J; Egashira K; Takeshita A
    Circulation; 2003 Oct; 108(17):2134-40. PubMed ID: 14517168
    [TBL] [Abstract][Full Text] [Related]  

  • 11. Allopurinol attenuates left ventricular remodeling and dysfunction after experimental myocardial infarction: a new action for an old drug?
    Engberding N; Spiekermann S; Schaefer A; Heineke A; Wiencke A; Müller M; Fuchs M; Hilfiker-Kleiner D; Hornig B; Drexler H; Landmesser U
    Circulation; 2004 Oct; 110(15):2175-9. PubMed ID: 15466649
    [TBL] [Abstract][Full Text] [Related]  

  • 12. K(ATP) activation prevents progression of cardiac hypertrophy to failure induced by pressure overload via protecting endothelial function.
    Gao S; Long CL; Wang RH; Wang H
    Cardiovasc Res; 2009 Aug; 83(3):444-56. PubMed ID: 19304734
    [TBL] [Abstract][Full Text] [Related]  

  • 13. Borderzone contractile dysfunction is transiently attenuated and left ventricular structural remodeling is markedly reduced following reperfused myocardial infarction in inducible nitric oxide synthase knockout mice.
    Gilson WD; Epstein FH; Yang Z; Xu Y; Prasad KM; Toufektsian MC; Laubach VE; French BA
    J Am Coll Cardiol; 2007 Oct; 50(18):1799-807. PubMed ID: 17964046
    [TBL] [Abstract][Full Text] [Related]  

  • 14. Critical role of the NAD(P)H oxidase subunit p47phox for left ventricular remodeling/dysfunction and survival after myocardial infarction.
    Doerries C; Grote K; Hilfiker-Kleiner D; Luchtefeld M; Schaefer A; Holland SM; Sorrentino S; Manes C; Schieffer B; Drexler H; Landmesser U
    Circ Res; 2007 Mar; 100(6):894-903. PubMed ID: 17332431
    [TBL] [Abstract][Full Text] [Related]  

  • 15. Statin-induced improvement of endothelial progenitor cell mobilization, myocardial neovascularization, left ventricular function, and survival after experimental myocardial infarction requires endothelial nitric oxide synthase.
    Landmesser U; Engberding N; Bahlmann FH; Schaefer A; Wiencke A; Heineke A; Spiekermann S; Hilfiker-Kleiner D; Templin C; Kotlarz D; Mueller M; Fuchs M; Hornig B; Haller H; Drexler H
    Circulation; 2004 Oct; 110(14):1933-9. PubMed ID: 15466656
    [TBL] [Abstract][Full Text] [Related]  

  • 16. Parathyroid hormone treatment after myocardial infarction promotes cardiac repair by enhanced neovascularization and cell survival.
    Zaruba MM; Huber BC; Brunner S; Deindl E; David R; Fischer R; Assmann G; Herbach N; Grundmann S; Wanke R; Mueller-Hoecker J; Franz WM
    Cardiovasc Res; 2008 Mar; 77(4):722-31. PubMed ID: 18055578
    [TBL] [Abstract][Full Text] [Related]  

  • 17. Postinfarction treatment with an adenoviral vector expressing hepatocyte growth factor relieves chronic left ventricular remodeling and dysfunction in mice.
    Li Y; Takemura G; Kosai K; Yuge K; Nagano S; Esaki M; Goto K; Takahashi T; Hayakawa K; Koda M; Kawase Y; Maruyama R; Okada H; Minatoguchi S; Mizuguchi H; Fujiwara T; Fujiwara H
    Circulation; 2003 May; 107(19):2499-506. PubMed ID: 12695295
    [TBL] [Abstract][Full Text] [Related]  

  • 18. Bone marrow molecular alterations after myocardial infarction: Impact on endothelial progenitor cells.
    Thum T; Fraccarollo D; Galuppo P; Tsikas D; Frantz S; Ertl G; Bauersachs J
    Cardiovasc Res; 2006 Apr; 70(1):50-60. PubMed ID: 16480696
    [TBL] [Abstract][Full Text] [Related]  

  • 19. Inhibition of granulation tissue cell apoptosis during the subacute stage of myocardial infarction improves cardiac remodeling and dysfunction at the chronic stage.
    Hayakawa K; Takemura G; Kanoh M; Li Y; Koda M; Kawase Y; Maruyama R; Okada H; Minatoguchi S; Fujiwara T; Fujiwara H
    Circulation; 2003 Jul; 108(1):104-9. PubMed ID: 12821555
    [TBL] [Abstract][Full Text] [Related]  

  • 20. C-type natriuretic peptide, a novel antifibrotic and antihypertrophic agent, prevents cardiac remodeling after myocardial infarction.
    Soeki T; Kishimoto I; Okumura H; Tokudome T; Horio T; Mori K; Kangawa K
    J Am Coll Cardiol; 2005 Feb; 45(4):608-16. PubMed ID: 15708711
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 38.