359 related articles for article (PubMed ID: 18978354)
1. FANCJ is a structure-specific DNA helicase associated with the maintenance of genomic G/C tracts.
London TB; Barber LJ; Mosedale G; Kelly GP; Balasubramanian S; Hickson ID; Boulton SJ; Hiom K
J Biol Chem; 2008 Dec; 283(52):36132-9. PubMed ID: 18978354
[TBL] [Abstract][Full Text] [Related]
2. FANCJ helicase defective in Fanconia anemia and breast cancer unwinds G-quadruplex DNA to defend genomic stability.
Wu Y; Shin-ya K; Brosh RM
Mol Cell Biol; 2008 Jun; 28(12):4116-28. PubMed ID: 18426915
[TBL] [Abstract][Full Text] [Related]
3. Interaction between the helicases genetically linked to Fanconi anemia group J and Bloom's syndrome.
Suhasini AN; Rawtani NA; Wu Y; Sommers JA; Sharma S; Mosedale G; North PS; Cantor SB; Hickson ID; Brosh RM
EMBO J; 2011 Feb; 30(4):692-705. PubMed ID: 21240188
[TBL] [Abstract][Full Text] [Related]
4. Specialization among iron-sulfur cluster helicases to resolve G-quadruplex DNA structures that threaten genomic stability.
Bharti SK; Sommers JA; George F; Kuper J; Hamon F; Shin-ya K; Teulade-Fichou MP; Kisker C; Brosh RM
J Biol Chem; 2013 Sep; 288(39):28217-29. PubMed ID: 23935105
[TBL] [Abstract][Full Text] [Related]
5. Fanconi anemia and Bloom's syndrome crosstalk through FANCJ-BLM helicase interaction.
Suhasini AN; Brosh RM
Trends Genet; 2012 Jan; 28(1):7-13. PubMed ID: 22024395
[TBL] [Abstract][Full Text] [Related]
6. G-quadruplex recognition and remodeling by the FANCJ helicase.
Wu CG; Spies M
Nucleic Acids Res; 2016 Oct; 44(18):8742-8753. PubMed ID: 27342280
[TBL] [Abstract][Full Text] [Related]
7. Purification and biochemical characterization of the G4 resolvase and DNA helicase FANCJ.
Kulikowicz T; Sommers JA; Fuchs KF; Wu Y; Brosh RM
Methods Enzymol; 2024; 695():1-27. PubMed ID: 38521581
[TBL] [Abstract][Full Text] [Related]
8. Novel function of the Fanconi anemia group J or RECQ1 helicase to disrupt protein-DNA complexes in a replication protein A-stimulated manner.
Sommers JA; Banerjee T; Hinds T; Wan B; Wold MS; Lei M; Brosh RM
J Biol Chem; 2014 Jul; 289(29):19928-41. PubMed ID: 24895130
[TBL] [Abstract][Full Text] [Related]
9. Genomic stability: FANCJ-dependent G4 DNA repair.
Maizels N
Curr Biol; 2008 Jul; 18(14):R613-4. PubMed ID: 18644339
[TBL] [Abstract][Full Text] [Related]
10. G-quadruplexes and helicases.
Mendoza O; Bourdoncle A; Boulé JB; Brosh RM; Mergny JL
Nucleic Acids Res; 2016 Mar; 44(5):1989-2006. PubMed ID: 26883636
[TBL] [Abstract][Full Text] [Related]
11. FANCJ helicase uniquely senses oxidative base damage in either strand of duplex DNA and is stimulated by replication protein A to unwind the damaged DNA substrate in a strand-specific manner.
Suhasini AN; Sommers JA; Mason AC; Voloshin ON; Camerini-Otero RD; Wold MS; Brosh RM
J Biol Chem; 2009 Jul; 284(27):18458-70. PubMed ID: 19419957
[TBL] [Abstract][Full Text] [Related]
12. DOG-1 is the Caenorhabditis elegans BRIP1/FANCJ homologue and functions in interstrand cross-link repair.
Youds JL; Barber LJ; Ward JD; Collis SJ; O'Neil NJ; Boulton SJ; Rose AM
Mol Cell Biol; 2008 Mar; 28(5):1470-9. PubMed ID: 18086896
[TBL] [Abstract][Full Text] [Related]
13. FANCJ (BACH1) helicase forms DNA damage inducible foci with replication protein A and interacts physically and functionally with the single-stranded DNA-binding protein.
Gupta R; Sharma S; Sommers JA; Kenny MK; Cantor SB; Brosh RM
Blood; 2007 Oct; 110(7):2390-8. PubMed ID: 17596542
[TBL] [Abstract][Full Text] [Related]
14. A minimal threshold of FANCJ helicase activity is required for its response to replication stress or double-strand break repair.
Bharti SK; Sommers JA; Awate S; Bellani MA; Khan I; Bradley L; King GA; Seol Y; Vidhyasagar V; Wu Y; Abe T; Kobayashi K; Shin-Ya K; Kitao H; Wold MS; Branzei D; Neuman KC; Brosh RM
Nucleic Acids Res; 2018 Jul; 46(12):6238-6256. PubMed ID: 29788478
[TBL] [Abstract][Full Text] [Related]
15. Insight into the roles of helicase motif Ia by characterizing Fanconi anemia group J protein (FANCJ) patient mutations.
Guo M; Vidhyasagar V; Ding H; Wu Y
J Biol Chem; 2014 Apr; 289(15):10551-10565. PubMed ID: 24573678
[TBL] [Abstract][Full Text] [Related]
16. Fanconi anemia group J mutation abolishes its DNA repair function by uncoupling DNA translocation from helicase activity or disruption of protein-DNA complexes.
Wu Y; Sommers JA; Suhasini AN; Leonard T; Deakyne JS; Mazin AV; Shin-Ya K; Kitao H; Brosh RM
Blood; 2010 Nov; 116(19):3780-91. PubMed ID: 20639400
[TBL] [Abstract][Full Text] [Related]
17. Mutagenic capacity of endogenous G4 DNA underlies genome instability in FANCJ-defective C. elegans.
Kruisselbrink E; Guryev V; Brouwer K; Pontier DB; Cuppen E; Tijsterman M
Curr Biol; 2008 Jun; 18(12):900-5. PubMed ID: 18538569
[TBL] [Abstract][Full Text] [Related]
18. FANCJ promotes DNA synthesis through G-quadruplex structures.
Castillo Bosch P; Segura-Bayona S; Koole W; van Heteren JT; Dewar JM; Tijsterman M; Knipscheer P
EMBO J; 2014 Nov; 33(21):2521-33. PubMed ID: 25193968
[TBL] [Abstract][Full Text] [Related]
19. FANCJ coordinates two pathways that maintain epigenetic stability at G-quadruplex DNA.
Sarkies P; Murat P; Phillips LG; Patel KJ; Balasubramanian S; Sale JE
Nucleic Acids Res; 2012 Feb; 40(4):1485-98. PubMed ID: 22021381
[TBL] [Abstract][Full Text] [Related]
20. Multistep mechanism of G-quadruplex resolution during DNA replication.
Sato K; Martin-Pintado N; Post H; Altelaar M; Knipscheer P
Sci Adv; 2021 Sep; 7(39):eabf8653. PubMed ID: 34559566
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
