252 related articles for article (PubMed ID: 19088846)
1. Analysis of STAT1 activation by six FGFR3 mutants associated with skeletal dysplasia undermines dominant role of STAT1 in FGFR3 signaling in cartilage.
Krejci P; Salazar L; Kashiwada TA; Chlebova K; Salasova A; Thompson LM; Bryja V; Kozubik A; Wilcox WR
PLoS One; 2008; 3(12):e3961. PubMed ID: 19088846
[TBL] [Abstract][Full Text] [Related]
2. Meclozine facilitates proliferation and differentiation of chondrocytes by attenuating abnormally activated FGFR3 signaling in achondroplasia.
Matsushita M; Kitoh H; Ohkawara B; Mishima K; Kaneko H; Ito M; Masuda A; Ishiguro N; Ohno K
PLoS One; 2013; 8(12):e81569. PubMed ID: 24324705
[TBL] [Abstract][Full Text] [Related]
3. Deregulated FGFR3 mutants in multiple myeloma cell lines with t(4;14): comparative analysis of Y373C, K650E and the novel G384D mutations.
Ronchetti D; Greco A; Compasso S; Colombo G; Dell'Era P; Otsuki T; Lombardi L; Neri A
Oncogene; 2001 Jun; 20(27):3553-62. PubMed ID: 11429702
[TBL] [Abstract][Full Text] [Related]
4. Sixteen years and counting: the current understanding of fibroblast growth factor receptor 3 (FGFR3) signaling in skeletal dysplasias.
Foldynova-Trantirkova S; Wilcox WR; Krejci P
Hum Mutat; 2012 Jan; 33(1):29-41. PubMed ID: 22045636
[TBL] [Abstract][Full Text] [Related]
5. Sustained phosphorylation of mutated FGFR3 is a crucial feature of genetic dwarfism and induces apoptosis in the ATDC5 chondrogenic cell line via PLCgamma-activated STAT1.
Harada D; Yamanaka Y; Ueda K; Nishimura R; Morishima T; Seino Y; Tanaka H
Bone; 2007 Aug; 41(2):273-81. PubMed ID: 17561467
[TBL] [Abstract][Full Text] [Related]
6. STAT1 and STAT3 do not participate in FGF-mediated growth arrest in chondrocytes.
Krejci P; Salazar L; Goodridge HS; Kashiwada TA; Schibler MJ; Jelinkova P; Thompson LM; Wilcox WR
J Cell Sci; 2008 Feb; 121(Pt 3):272-81. PubMed ID: 18198189
[TBL] [Abstract][Full Text] [Related]
7. NF449 is a novel inhibitor of fibroblast growth factor receptor 3 (FGFR3) signaling active in chondrocytes and multiple myeloma cells.
Krejci P; Murakami S; Prochazkova J; Trantirek L; Chlebova K; Ouyang Z; Aklian A; Smutny J; Bryja V; Kozubik A; Wilcox WR
J Biol Chem; 2010 Jul; 285(27):20644-53. PubMed ID: 20439987
[TBL] [Abstract][Full Text] [Related]
8. Identification of tyrosine residues in constitutively activated fibroblast growth factor receptor 3 involved in mitogenesis, Stat activation, and phosphatidylinositol 3-kinase activation.
Hart KC; Robertson SC; Donoghue DJ
Mol Biol Cell; 2001 Apr; 12(4):931-42. PubMed ID: 11294897
[TBL] [Abstract][Full Text] [Related]
9. The paradox of FGFR3 signaling in skeletal dysplasia: why chondrocytes growth arrest while other cells over proliferate.
Krejci P
Mutat Res Rev Mutat Res; 2014; 759():40-8. PubMed ID: 24295726
[TBL] [Abstract][Full Text] [Related]
10. Expression of fibroblast growth factor receptor-3 (FGFR3), signal transducer and activator of transcription-1, and cyclin-dependent kinase inhibitor p21 during endochondral ossification: differential role of FGFR3 in skeletal development and fracture repair.
Nakajima A; Shimizu S; Moriya H; Yamazaki M
Endocrinology; 2003 Oct; 144(10):4659-68. PubMed ID: 12960068
[TBL] [Abstract][Full Text] [Related]
11. Overexpression of FGFR3, Stat1, Stat5 and p21Cip1 correlates with phenotypic severity and defective chondrocyte differentiation in FGFR3-related chondrodysplasias.
Legeai-Mallet L; Benoist-Lasselin C; Munnich A; Bonaventure J
Bone; 2004 Jan; 34(1):26-36. PubMed ID: 14751560
[TBL] [Abstract][Full Text] [Related]
12. The thanatophoric dysplasia type II mutation hampers complete maturation of fibroblast growth factor receptor 3 (FGFR3), which activates signal transducer and activator of transcription 1 (STAT1) from the endoplasmic reticulum.
Lievens PM; Liboi E
J Biol Chem; 2003 May; 278(19):17344-9. PubMed ID: 12624096
[TBL] [Abstract][Full Text] [Related]
13. A Lys644Glu substitution in fibroblast growth factor receptor 3 (FGFR3) causes dwarfism in mice by activation of STATs and ink4 cell cycle inhibitors.
Li C; Chen L; Iwata T; Kitagawa M; Fu XY; Deng CX
Hum Mol Genet; 1999 Jan; 8(1):35-44. PubMed ID: 9887329
[TBL] [Abstract][Full Text] [Related]
14.
Sun X; Zhang R; Chen H; Du X; Chen S; Huang J; Liu M; Xu M; Luo F; Jin M; Su N; Qi H; Yang J; Tan Q; Zhang D; Ni Z; Liang S; Zhang B; Chen D; Zhang X; Luo L; Chen L; Xie Y
Theranostics; 2020; 10(16):7111-7130. PubMed ID: 32641982
[TBL] [Abstract][Full Text] [Related]
15. Mutant FGFR3 associated with SADDAN disease causes cytoskeleton disorganization through PLCγ1/Src-mediated paxillin hyperphosphorylation.
Montone R; Romanelli MG; Baruzzi A; Ferrarini F; Liboi E; Lievens PM
Int J Biochem Cell Biol; 2018 Feb; 95():17-26. PubMed ID: 29242050
[TBL] [Abstract][Full Text] [Related]
16. Bortezomib therapeutic effect is associated with expression and mutation of FGFR3 in human lymphoma cells.
Zheng W; Guan M; Zhu L; Cai Z; Chung V; Huang H; Yen Y
Anticancer Res; 2010 Jun; 30(6):1921-30. PubMed ID: 20651335
[TBL] [Abstract][Full Text] [Related]
17. The localization of FGFR3 mutations causing thanatophoric dysplasia type I differentially affects phosphorylation, processing and ubiquitylation of the receptor.
Bonaventure J; Horne WC; Baron R
FEBS J; 2007 Jun; 274(12):3078-93. PubMed ID: 17509076
[TBL] [Abstract][Full Text] [Related]
18. Measurement of FGFR3 signaling at the cell membrane via total internal reflection fluorescence microscopy to compare the activation of FGFR3 mutants.
Hartl I; Brumovska V; Striedner Y; Yasari A; Schütz GJ; Sevcsik E; Tiemann-Boege I
J Biol Chem; 2023 Feb; 299(2):102832. PubMed ID: 36581204
[TBL] [Abstract][Full Text] [Related]
19. K644E/M FGFR3 mutants activate Erk1/2 from the endoplasmic reticulum through FRS2 alpha and PLC gamma-independent pathways.
Lievens PM; Roncador A; Liboi E
J Mol Biol; 2006 Mar; 357(3):783-92. PubMed ID: 16476447
[TBL] [Abstract][Full Text] [Related]
20. Mouse models orthologous to FGFR3-related skeletal dysplasias.
Brodie SG; Deng CX
Pediatr Pathol Mol Med; 2003; 22(1):87-103. PubMed ID: 12687892
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]