332 related articles for article (PubMed ID: 19202324)
1. Phosphorylated H2AX foci in tumor cells have no correlation with their radiation sensitivities.
Yoshikawa T; Kashino G; Ono K; Watanabe M
J Radiat Res; 2009 Mar; 50(2):151-60. PubMed ID: 19202324
[TBL] [Abstract][Full Text] [Related]
2. Kinetics and dose-response of residual 53BP1/gamma-H2AX foci: co-localization, relationship with DSB repair and clonogenic survival.
Marková E; Schultz N; Belyaev IY
Int J Radiat Biol; 2007 May; 83(5):319-29. PubMed ID: 17457757
[TBL] [Abstract][Full Text] [Related]
3. Histone H2AX phosphorylation in normal human cells irradiated with focused ultrasoft X rays: evidence for chromatin movement during repair.
Hamada N; Schettino G; Kashino G; Vaid M; Suzuki K; Kodama S; Vojnovic B; Folkard M; Watanabe M; Michael BD; Prise KM
Radiat Res; 2006 Jul; 166(1 Pt 1):31-8. PubMed ID: 16808616
[TBL] [Abstract][Full Text] [Related]
4. Predicting Radiosensitivity with Gamma-H2AX Foci Assay after Single High-Dose-Rate and Pulsed Dose-Rate Ionizing Irradiation.
van Oorschot B; Hovingh S; Dekker A; Stalpers LJ; Franken NA
Radiat Res; 2016 Feb; 185(2):190-8. PubMed ID: 26789702
[TBL] [Abstract][Full Text] [Related]
5. Long time persistence of residual 53BP1/γ-H2AX foci in human lymphocytes in relationship to apoptosis, chromatin condensation and biological dosimetry.
Marková E; Torudd J; Belyaev I
Int J Radiat Biol; 2011 Jul; 87(7):736-45. PubMed ID: 21718211
[TBL] [Abstract][Full Text] [Related]
6. Phosphorylated histone H2AX foci persist on rejoined mitotic chromosomes in normal human diploid cells exposed to ionizing radiation.
Suzuki M; Suzuki K; Kodama S; Watanabe M
Radiat Res; 2006 Mar; 165(3):269-76. PubMed ID: 16494514
[TBL] [Abstract][Full Text] [Related]
7. Low-dose hyper-radiosensitivity is not caused by a failure to recognize DNA double-strand breaks.
Wykes SM; Piasentin E; Joiner MC; Wilson GD; Marples B
Radiat Res; 2006 May; 165(5):516-24. PubMed ID: 16669705
[TBL] [Abstract][Full Text] [Related]
8. gammaH2AX foci form preferentially in euchromatin after ionising-radiation.
Cowell IG; Sunter NJ; Singh PB; Austin CA; Durkacz BW; Tilby MJ
PLoS One; 2007 Oct; 2(10):e1057. PubMed ID: 17957241
[TBL] [Abstract][Full Text] [Related]
9. In vivo formation of gamma-H2AX and 53BP1 DNA repair foci in blood cells after radioiodine therapy of differentiated thyroid cancer.
Lassmann M; Hänscheid H; Gassen D; Biko J; Meineke V; Reiners C; Scherthan H
J Nucl Med; 2010 Aug; 51(8):1318-25. PubMed ID: 20660387
[TBL] [Abstract][Full Text] [Related]
10. Focus formation of DNA repair proteins in normal and repair-deficient cells irradiated with high-LET ions.
Karlsson KH; Stenerlöw B
Radiat Res; 2004 May; 161(5):517-27. PubMed ID: 15161372
[TBL] [Abstract][Full Text] [Related]
11. Induction and rejoining of DNA double strand breaks assessed by H2AX phosphorylation in melanoma cells irradiated with proton and lithium beams.
Ibañez IL; Bracalente C; Molinari BL; Palmieri MA; Policastro L; Kreiner AJ; Burlón AA; Valda A; Navalesi D; Davidson J; Davidson M; Vázquez M; Ozafrán M; Durán H
Int J Radiat Oncol Biol Phys; 2009 Jul; 74(4):1226-35. PubMed ID: 19545788
[TBL] [Abstract][Full Text] [Related]
12. The profiles of gamma-H2AX along with ATM/DNA-PKcs activation in the lymphocytes and granulocytes of rat and human blood exposed to gamma rays.
Wang J; Yin L; Zhang J; Zhang Y; Zhang X; Ding D; Gao Y; Li Q; Chen H
Radiat Environ Biophys; 2016 Aug; 55(3):359-70. PubMed ID: 27260225
[TBL] [Abstract][Full Text] [Related]
13. Gamma-H2AX expression pattern in non-irradiated neonatal mouse germ cells and after low-dose gamma-radiation: relationships between chromatid breaks and DNA double-strand breaks.
Forand A; Dutrillaux B; Bernardino-Sgherri J
Biol Reprod; 2004 Aug; 71(2):643-9. PubMed ID: 15115728
[TBL] [Abstract][Full Text] [Related]
14. Qualitative and quantitative analysis of phosphorylated ATM foci induced by low-dose ionizing radiation.
Suzuki K; Okada H; Yamauchi M; Oka Y; Kodama S; Watanabe M
Radiat Res; 2006 May; 165(5):499-504. PubMed ID: 16669703
[TBL] [Abstract][Full Text] [Related]
15. DNA-PKcs plays a dominant role in the regulation of H2AX phosphorylation in response to DNA damage and cell cycle progression.
An J; Huang YC; Xu QZ; Zhou LJ; Shang ZF; Huang B; Wang Y; Liu XD; Wu DC; Zhou PK
BMC Mol Biol; 2010 Mar; 11():18. PubMed ID: 20205745
[TBL] [Abstract][Full Text] [Related]
16. A critical role for histone H2AX in recruitment of repair factors to nuclear foci after DNA damage.
Paull TT; Rogakou EP; Yamazaki V; Kirchgessner CU; Gellert M; Bonner WM
Curr Biol; 2000 Jul 27-Aug 10; 10(15):886-95. PubMed ID: 10959836
[TBL] [Abstract][Full Text] [Related]
17. Imaging features that discriminate between foci induced by high- and low-LET radiation in human fibroblasts.
Costes SV; Boissière A; Ravani S; Romano R; Parvin B; Barcellos-Hoff MH
Radiat Res; 2006 May; 165(5):505-15. PubMed ID: 16669704
[TBL] [Abstract][Full Text] [Related]
18. Dephosphorylation of histone gamma-H2AX during repair of DNA double-strand breaks in mammalian cells and its inhibition by calyculin A.
Nazarov IB; Smirnova AN; Krutilina RI; Svetlova MP; Solovjeva LV; Nikiforov AA; Oei SL; Zalenskaya IA; Yau PM; Bradbury EM; Tomilin NV
Radiat Res; 2003 Sep; 160(3):309-17. PubMed ID: 12926989
[TBL] [Abstract][Full Text] [Related]
19. Relationship between DNA double-strand break rejoining and cell survival after exposure to ionizing radiation in human fibroblast strains with differing ATM/p53 status: implications for evaluation of clinical radiosensitivity.
Mirzayans R; Severin D; Murray D
Int J Radiat Oncol Biol Phys; 2006 Dec; 66(5):1498-505. PubMed ID: 17126209
[TBL] [Abstract][Full Text] [Related]
20. Influence of Different Antioxidants on X-Ray Induced DNA Double-Strand Breaks (DSBs) Using γ-H2AX Immunofluorescence Microscopy in a Preliminary Study.
Brand M; Sommer M; Ellmann S; Wuest W; May MS; Eller A; Vogt S; Lell MM; Kuefner MA; Uder M
PLoS One; 2015; 10(5):e0127142. PubMed ID: 25996998
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
