173 related articles for article (PubMed ID: 19351612)
1. Different prelamin A forms accumulate in human fibroblasts: a study in experimental models and progeria.
Dominici S; Fiori V; Magnani M; Schena E; Capanni C; Camozzi D; D'Apice MR; Le Dour C; Auclair M; Caron M; Novelli G; Vigouroux C; Maraldi NM; Lattanzi G
Eur J Histochem; 2009; 53(1):43-52. PubMed ID: 19351612
[TBL] [Abstract][Full Text] [Related]
2. Different prelamin A forms accumulate in human fibroblasts: a study in experimental models and progeria.
Dominici S; Fiori V; Magnani M; Schena E; Capanni C; Camozzi D; D'Apice MR; Le Dour C; Auclair M; Caron M; Novelli G; Vigouroux C; Maraldi NM; Lattanzi G
Eur J Histochem; 2009 Mar; 53(1):e6. PubMed ID: 30256865
[TBL] [Abstract][Full Text] [Related]
3. Requirements for efficient proteolytic cleavage of prelamin A by ZMPSTE24.
Barrowman J; Hamblet C; Kane MS; Michaelis S
PLoS One; 2012; 7(2):e32120. PubMed ID: 22355414
[TBL] [Abstract][Full Text] [Related]
4. Autophagic degradation of farnesylated prelamin A as a therapeutic approach to lamin-linked progeria.
Cenni V; Capanni C; Columbaro M; Ortolani M; D'Apice MR; Novelli G; Fini M; Marmiroli S; Scarano E; Maraldi NM; Squarzoni S; Prencipe S; Lattanzi G
Eur J Histochem; 2011 Oct; 55(4):e36. PubMed ID: 22297442
[TBL] [Abstract][Full Text] [Related]
5. Prelamin A farnesylation and progeroid syndromes.
Young SG; Meta M; Yang SH; Fong LG
J Biol Chem; 2006 Dec; 281(52):39741-5. PubMed ID: 17090536
[TBL] [Abstract][Full Text] [Related]
6. The farnesyl transferase inhibitor (FTI) lonafarnib improves nuclear morphology in ZMPSTE24-deficient fibroblasts from patients with the progeroid disorder MAD-B.
Odinammadu KO; Shilagardi K; Tuminelli K; Judge DP; Gordon LB; Michaelis S
Nucleus; 2023 Dec; 14(1):2288476. PubMed ID: 38050983
[TBL] [Abstract][Full Text] [Related]
7. Lamin A precursor induces barrier-to-autointegration factor nuclear localization.
Capanni C; Cenni V; Haraguchi T; Squarzoni S; Schüchner S; Ogris E; Novelli G; Maraldi N; Lattanzi G
Cell Cycle; 2010 Jul; 9(13):2600-10. PubMed ID: 20581439
[TBL] [Abstract][Full Text] [Related]
8. Blocking protein farnesyltransferase improves nuclear shape in fibroblasts from humans with progeroid syndromes.
Toth JI; Yang SH; Qiao X; Beigneux AP; Gelb MH; Moulson CL; Miner JH; Young SG; Fong LG
Proc Natl Acad Sci U S A; 2005 Sep; 102(36):12873-8. PubMed ID: 16129834
[TBL] [Abstract][Full Text] [Related]
9. An accumulation of non-farnesylated prelamin A causes cardiomyopathy but not progeria.
Davies BS; Barnes RH; Tu Y; Ren S; Andres DA; Spielmann HP; Lammerding J; Wang Y; Young SG; Fong LG
Hum Mol Genet; 2010 Jul; 19(13):2682-94. PubMed ID: 20421363
[TBL] [Abstract][Full Text] [Related]
10. Abolishing the prelamin A ZMPSTE24 cleavage site leads to progeroid phenotypes with near-normal longevity in mice.
Wang Y; Shilagardi K; Hsu T; Odinammadu KO; Maruyama T; Wu W; Lin CS; Damoci CB; Spear ED; Shin JY; Hsu W; Michaelis S; Worman HJ
Proc Natl Acad Sci U S A; 2022 Mar; 119(9):. PubMed ID: 35197292
[TBL] [Abstract][Full Text] [Related]
11. Inhibiting farnesylation reverses the nuclear morphology defect in a HeLa cell model for Hutchinson-Gilford progeria syndrome.
Mallampalli MP; Huyer G; Bendale P; Gelb MH; Michaelis S
Proc Natl Acad Sci U S A; 2005 Oct; 102(40):14416-21. PubMed ID: 16186497
[TBL] [Abstract][Full Text] [Related]
12. Prelamin A, Zmpste24, misshapen cell nuclei, and progeria--new evidence suggesting that protein farnesylation could be important for disease pathogenesis.
Young SG; Fong LG; Michaelis S
J Lipid Res; 2005 Dec; 46(12):2531-58. PubMed ID: 16207929
[TBL] [Abstract][Full Text] [Related]
13. Accumulation of distinct prelamin A variants in human diploid fibroblasts differentially affects cell homeostasis.
Candelario J; Borrego S; Reddy S; Comai L
Exp Cell Res; 2011 Feb; 317(3):319-29. PubMed ID: 20974128
[TBL] [Abstract][Full Text] [Related]
14. Emerin-prelamin A interplay in human fibroblasts.
Capanni C; Del Coco R; Mattioli E; Camozzi D; Columbaro M; Schena E; Merlini L; Squarzoni S; Maraldi NM; Lattanzi G
Biol Cell; 2009 Sep; 101(9):541-54. PubMed ID: 19323649
[TBL] [Abstract][Full Text] [Related]
15. A mutation abolishing the ZMPSTE24 cleavage site in prelamin A causes a progeroid disorder.
Wang Y; Lichter-Konecki U; Anyane-Yeboa K; Shaw JE; Lu JT; Östlund C; Shin JY; Clark LN; Gundersen GG; Nagy PL; Worman HJ
J Cell Sci; 2016 May; 129(10):1975-80. PubMed ID: 27034136
[TBL] [Abstract][Full Text] [Related]
16. Heterozygosity for Lmna deficiency eliminates the progeria-like phenotypes in Zmpste24-deficient mice.
Fong LG; Ng JK; Meta M; Coté N; Yang SH; Stewart CL; Sullivan T; Burghardt A; Majumdar S; Reue K; Bergo MO; Young SG
Proc Natl Acad Sci U S A; 2004 Dec; 101(52):18111-6. PubMed ID: 15608054
[TBL] [Abstract][Full Text] [Related]
17. HIV protease inhibitors block the zinc metalloproteinase ZMPSTE24 and lead to an accumulation of prelamin A in cells.
Coffinier C; Hudon SE; Farber EA; Chang SY; Hrycyna CA; Young SG; Fong LG
Proc Natl Acad Sci U S A; 2007 Aug; 104(33):13432-7. PubMed ID: 17652517
[TBL] [Abstract][Full Text] [Related]
18. Prelamin A processing, accumulation and distribution in normal cells and laminopathy disorders.
Casasola A; Scalzo D; Nandakumar V; Halow J; Recillas-Targa F; Groudine M; Rincón-Arano H
Nucleus; 2016; 7(1):84-102. PubMed ID: 26900797
[TBL] [Abstract][Full Text] [Related]
19. RNAi of FACE1 protease results in growth inhibition of human cells expressing lamin A: implications for Hutchinson-Gilford progeria syndrome.
Gruber J; Lampe T; Osborn M; Weber K
J Cell Sci; 2005 Feb; 118(Pt 4):689-96. PubMed ID: 15671064
[TBL] [Abstract][Full Text] [Related]
20. N6-isopentenyladenosine improves nuclear shape in fibroblasts from humans with progeroid syndromes by inhibiting the farnesylation of prelamin A.
Bifulco M; D'Alessandro A; Paladino S; Malfitano AM; Notarnicola M; Caruso MG; Laezza C
FEBS J; 2013 Dec; 280(23):6223-32. PubMed ID: 24112551
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]