BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

217 related articles for article (PubMed ID: 19352084)

  • 1. [Molecular bases of leukemia, MPD and MDS].
    Kitamura T
    Rinsho Ketsueki; 2009 Mar; 50(3):174-81. PubMed ID: 19352084
    [No Abstract]   [Full Text] [Related]  

  • 2. [Molecular mechanisms in myeloproliferative diseases and myelodysplastic syndromes].
    Harada H; Harada Y; Kimura A
    Nihon Rinsho; 2009 Oct; 67(10):1901-5. PubMed ID: 19860187
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Simultaneous occurrence of the JAK2V617F mutation and BCR-ABL gene rearrangement in patients with chronic myeloproliferative disorders.
    Kim YK; Shin MG; Kim HR; Yang DH; Cho SH; Lee JJ; Chung IJ; Ryang DW; Kim HJ
    Leuk Res; 2008 Jun; 32(6):993-5. PubMed ID: 18055011
    [No Abstract]   [Full Text] [Related]  

  • 4. JAK2-V617F and BCR-ABL--double jeopardy?
    Krämer A
    Leuk Res; 2008 Oct; 32(10):1489-90. PubMed ID: 18439674
    [No Abstract]   [Full Text] [Related]  

  • 5. Myeloproliferative disorders with coexisting BCR-ABL translocation and JAK2(V617F) mutation.
    Go RS
    Leukemia; 2007 Sep; 21(9):2051; author reply 2052. PubMed ID: 17597806
    [No Abstract]   [Full Text] [Related]  

  • 6. Refractory anemia with ringed sideroblasts associated with marked thrombocytosis harbors JAK2 mutation and shows overlapping myeloproliferative and myelodysplastic features.
    Wang SA; Hasserjian RP; Loew JM; Sechman EV; Jones D; Hao S; Liu Q; Zhao W; Mehdi M; Galili N; Woda B; Raza A
    Leukemia; 2006 Sep; 20(9):1641-4. PubMed ID: 16871284
    [No Abstract]   [Full Text] [Related]  

  • 7. Chronic myeloproliferative diseases with concurrent BCR-ABL junction and JAK2V617F mutation.
    Hussein K; Bock O; Theophile K; Seegers A; Arps H; Basten O; Grips KH; Franz-Werner J; Büsche G; Kreipe H
    Leukemia; 2008 May; 22(5):1059-62. PubMed ID: 17972958
    [No Abstract]   [Full Text] [Related]  

  • 8. Concomitant detection of BCR-ABL translocation and JAK2 V617F mutation in five patients with myeloproliferative neoplasm at diagnosis.
    Cappetta M; Pérez V; Zubillaga MN; Elizondo V; Manrique G; Prosper I; Boschi S; Bonomi R; Pomoli S; Díaz L; Martínez L; Uriarte MR
    Int J Lab Hematol; 2013 Feb; 35(1):e4-5. PubMed ID: 23062104
    [No Abstract]   [Full Text] [Related]  

  • 9. Molecular bases of myelodysplastic syndromes: lessons from animal models.
    Komeno Y; Kitaura J; Kitamura T
    J Cell Physiol; 2009 Jun; 219(3):529-34. PubMed ID: 19259975
    [TBL] [Abstract][Full Text] [Related]  

  • 10. The JAK2 V617F mutation occurs frequently in myelodysplastic/myeloproliferative diseases, but is absent in true myelodysplastic syndromes with fibrosis.
    Kremer M; Horn T; Dechow T; Tzankov A; Quintanilla-Martínez L; Fend F
    Leukemia; 2006 Jul; 20(7):1315-6. PubMed ID: 16617316
    [No Abstract]   [Full Text] [Related]  

  • 11. [Molecular diagnosis of chronic myeloproliferative diseases and myelodysplastic syndromes].
    Bock O
    Verh Dtsch Ges Pathol; 2007; 91():140-53. PubMed ID: 18314608
    [TBL] [Abstract][Full Text] [Related]  

  • 12. Reply to Kremer M et al., the JAK2 V617F mutation occurs frequently in myelodysplastic/myeloproliferative diseases, but is absent in true myelodysplastic syndromes with fibrosis.
    Ohyashiki K; Ohyashiki JH
    Leukemia; 2006 Jul; 20(7):1297-8. PubMed ID: 16617315
    [No Abstract]   [Full Text] [Related]  

  • 13. The JAK2 V617F tyrosine kinase mutation in myelodysplastic syndromes (MDS) developing myelofibrosis indicates the myeloproliferative nature in a subset of MDS patients.
    Ohyashiki K; Aota Y; Akahane D; Gotoh A; Miyazawa K; Kimura Y; Ohyashiki JH
    Leukemia; 2005 Dec; 19(12):2359-60. PubMed ID: 16239910
    [No Abstract]   [Full Text] [Related]  

  • 14. Clinicopathological features of unbalanced translocation Der(1;7)(q10;p10) in myeloid neoplasms.
    So CC; Ma ES; Wan TS; Yip SF; Chan LC
    Leuk Res; 2008 Jun; 32(6):1000-1. PubMed ID: 17980911
    [No Abstract]   [Full Text] [Related]  

  • 15. [Molecular pathogenesis of MDS].
    Mitani K
    Rinsho Ketsueki; 2009 Oct; 50(10):1325-31. PubMed ID: 19915339
    [No Abstract]   [Full Text] [Related]  

  • 16. Mutations of N-RAS, FLT3 and p53 genes are not involved in the development of acute leukemia transformed from myeloproliferative diseases with JAK2 mutation.
    Suzuki M; Abe A; Kiyoi H; Murata M; Ito Y; Shimada K; Morishita Y; Kinoshita T; Naoe T
    Leukemia; 2006 Jun; 20(6):1168-9. PubMed ID: 16557239
    [No Abstract]   [Full Text] [Related]  

  • 17. Evolution of secondary hematologic disorders: preMDS-->MDS-->sAML.
    Preisler HD
    Cancer Treat Res; 2001; 108():185-230. PubMed ID: 11702600
    [No Abstract]   [Full Text] [Related]  

  • 18. [Application of novel tyrosine-kinase inhibitors for the treatment of leukemia and Ph-negative myeloproliferative neoplasms].
    Kirito K
    Rinsho Ketsueki; 2011 Jul; 52(7):460-8. PubMed ID: 21821977
    [No Abstract]   [Full Text] [Related]  

  • 19. Chronic myeloproliferative neoplasm with concurrent BCR-ABL translocation and JAK2 mutation.
    Siddiqui NS; Ssiddiqui HS; Qurat-Ul-Ain ; Siddiqui SA
    J Coll Physicians Surg Pak; 2010 Jun; 20(6):423. PubMed ID: 20642980
    [No Abstract]   [Full Text] [Related]  

  • 20. Gain of function, loss of control - a molecular basis for chronic myeloproliferative disorders.
    Cazzola M; Skoda R
    Haematologica; 2005 Jul; 90(7):871-4. PubMed ID: 15996923
    [No Abstract]   [Full Text] [Related]  

    [Next]    [New Search]
    of 11.