249 related articles for article (PubMed ID: 19366591)
1. The interaction of beta-amyloid protein with cellular membranes stimulates its own production.
Peters I; Igbavboa U; Schütt T; Haidari S; Hartig U; Rosello X; Böttner S; Copanaki E; Deller T; Kögel D; Wood WG; Müller WE; Eckert GP
Biochim Biophys Acta; 2009 May; 1788(5):964-72. PubMed ID: 19366591
[TBL] [Abstract][Full Text] [Related]
2. Lipid membranes and beta-amyloid: a harmful connection.
Eckert GP; Wood WG; Müller WE
Curr Protein Pept Sci; 2010 Aug; 11(5):319-25. PubMed ID: 20423299
[TBL] [Abstract][Full Text] [Related]
3. Cellular membrane fluidity in amyloid precursor protein processing.
Yang X; Sun GY; Eckert GP; Lee JC
Mol Neurobiol; 2014 Aug; 50(1):119-29. PubMed ID: 24553856
[TBL] [Abstract][Full Text] [Related]
4. Alzheimer's disease.
De-Paula VJ; Radanovic M; Diniz BS; Forlenza OV
Subcell Biochem; 2012; 65():329-52. PubMed ID: 23225010
[TBL] [Abstract][Full Text] [Related]
5. Cellular prion protein regulates beta-secretase cleavage of the Alzheimer's amyloid precursor protein.
Parkin ET; Watt NT; Hussain I; Eckman EA; Eckman CB; Manson JC; Baybutt HN; Turner AJ; Hooper NM
Proc Natl Acad Sci U S A; 2007 Jun; 104(26):11062-7. PubMed ID: 17573534
[TBL] [Abstract][Full Text] [Related]
6. Amyloid-β protein (Aβ) Glu11 is the major β-secretase site of β-site amyloid-β precursor protein-cleaving enzyme 1(BACE1), and shifting the cleavage site to Aβ Asp1 contributes to Alzheimer pathogenesis.
Deng Y; Wang Z; Wang R; Zhang X; Zhang S; Wu Y; Staufenbiel M; Cai F; Song W
Eur J Neurosci; 2013 Jun; 37(12):1962-9. PubMed ID: 23773065
[TBL] [Abstract][Full Text] [Related]
7. Zinc and Copper Differentially Modulate Amyloid Precursor Protein Processing by γ-Secretase and Amyloid-β Peptide Production.
Gerber H; Wu F; Dimitrov M; Garcia Osuna GM; Fraering PC
J Biol Chem; 2017 Mar; 292(9):3751-3767. PubMed ID: 28096459
[TBL] [Abstract][Full Text] [Related]
8. Alternative Selection of β-Site APP-Cleaving Enzyme 1 (BACE1) Cleavage Sites in Amyloid β-Protein Precursor (APP) Harboring Protective and Pathogenic Mutations within the Aβ Sequence.
Kimura A; Hata S; Suzuki T
J Biol Chem; 2016 Nov; 291(46):24041-24053. PubMed ID: 27687728
[TBL] [Abstract][Full Text] [Related]
9. Amyloid beta-protein interactions with membranes and cholesterol: causes or casualties of Alzheimer's disease.
Gibson Wood W; Eckert GP; Igbavboa U; Müller WE
Biochim Biophys Acta; 2003 Mar; 1610(2):281-90. PubMed ID: 12648781
[TBL] [Abstract][Full Text] [Related]
10. Oxidative lipid modification of nicastrin enhances amyloidogenic γ-secretase activity in Alzheimer's disease.
Gwon AR; Park JS; Arumugam TV; Kwon YK; Chan SL; Kim SH; Baik SH; Yang S; Yun YK; Choi Y; Kim S; Tang SC; Hyun DH; Cheng A; Dann CE; Bernier M; Lee J; Markesbery WR; Mattson MP; Jo DG
Aging Cell; 2012 Aug; 11(4):559-68. PubMed ID: 22404891
[TBL] [Abstract][Full Text] [Related]
11. Amylin and pramlintide modulate γ-secretase level and APP processing in lipid rafts.
Mousa YM; Abdallah IM; Hwang M; Martin DR; Kaddoumi A
Sci Rep; 2020 Feb; 10(1):3751. PubMed ID: 32111883
[TBL] [Abstract][Full Text] [Related]
12. Effects of membrane lipids on the activity and processivity of purified γ-secretase.
Holmes O; Paturi S; Ye W; Wolfe MS; Selkoe DJ
Biochemistry; 2012 May; 51(17):3565-75. PubMed ID: 22489600
[TBL] [Abstract][Full Text] [Related]
13. Generation of Alzheimer disease-associated amyloid β42/43 peptide by γ-secretase can be inhibited directly by modulation of membrane thickness.
Winkler E; Kamp F; Scheuring J; Ebke A; Fukumori A; Steiner H
J Biol Chem; 2012 Jun; 287(25):21326-34. PubMed ID: 22532566
[TBL] [Abstract][Full Text] [Related]
14. The Alzheimer's Disease γ-Secretase Generates Higher 42:40 Ratios for β-Amyloid Than for p3 Peptides.
Siegel G; Gerber H; Koch P; Bruestle O; Fraering PC; Rajendran L
Cell Rep; 2017 Jun; 19(10):1967-1976. PubMed ID: 28591569
[TBL] [Abstract][Full Text] [Related]
15. Metabolism of the "Swedish" amyloid precursor protein variant in neuro2a (N2a) cells. Evidence that cleavage at the "beta-secretase" site occurs in the golgi apparatus.
Thinakaran G; Teplow DB; Siman R; Greenberg B; Sisodia SS
J Biol Chem; 1996 Apr; 271(16):9390-7. PubMed ID: 8621605
[TBL] [Abstract][Full Text] [Related]
16. Overexpression of SNX7 reduces Aβ production by enhancing lysosomal degradation of APP.
Xu S; Zhang L; Brodin L
Biochem Biophys Res Commun; 2018 Jan; 495(1):12-19. PubMed ID: 29080748
[TBL] [Abstract][Full Text] [Related]
17. Role of amyloid beta in lipid homeostasis.
Grösgen S; Grimm MO; Friess P; Hartmann T
Biochim Biophys Acta; 2010 Aug; 1801(8):966-74. PubMed ID: 20452461
[TBL] [Abstract][Full Text] [Related]
18. Muscarinic acetylcholine receptor inhibition in transgenic Alzheimer-like Tg2576 mice by scopolamine favours the amyloidogenic route of processing of amyloid precursor protein.
Liskowsky W; Schliebs R
Int J Dev Neurosci; 2006; 24(2-3):149-56. PubMed ID: 16423497
[TBL] [Abstract][Full Text] [Related]
19. Vascular endothelial growth factor (VEGF) affects processing of amyloid precursor protein and beta-amyloidogenesis in brain slice cultures derived from transgenic Tg2576 mouse brain.
Bürger S; Noack M; Kirazov LP; Kirazov EP; Naydenov CL; Kouznetsova E; Yafai Y; Schliebs R
Int J Dev Neurosci; 2009 Oct; 27(6):517-23. PubMed ID: 19589380
[TBL] [Abstract][Full Text] [Related]
20. Gleevec shifts APP processing from a β-cleavage to a nonamyloidogenic cleavage.
Netzer WJ; Bettayeb K; Sinha SC; Flajolet M; Greengard P; Bustos V
Proc Natl Acad Sci U S A; 2017 Feb; 114(6):1389-1394. PubMed ID: 28115709
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]