These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.
10. Effects of ectopically expressed neuronal Wiskott-Aldrich syndrome protein domains on Rickettsia rickettsii actin-based motility. Harlander RS; Way M; Ren Q; Howe D; Grieshaber SS; Heinzen RA Infect Immun; 2003 Mar; 71(3):1551-6. PubMed ID: 12595475 [TBL] [Abstract][Full Text] [Related]
11. Neural Wiskott-Aldrich syndrome protein (N-WASP) is the specific ligand for Shigella VirG among the WASP family and determines the host cell type allowing actin-based spreading. Suzuki T; Mimuro H; Suetsugu S; Miki H; Takenawa T; Sasakawa C Cell Microbiol; 2002 Apr; 4(4):223-33. PubMed ID: 11952639 [TBL] [Abstract][Full Text] [Related]
12. Identification of Shigella flexneri IcsA residues affecting interaction with N-WASP, and evidence for IcsA-IcsA co-operative interaction. Teh MY; Morona R PLoS One; 2013; 8(2):e55152. PubMed ID: 23405119 [TBL] [Abstract][Full Text] [Related]
13. A comparative study of the actin-based motilities of the pathogenic bacteria Listeria monocytogenes, Shigella flexneri and Rickettsia conorii. Gouin E; Gantelet H; Egile C; Lasa I; Ohayon H; Villiers V; Gounon P; Sansonetti PJ; Cossart P J Cell Sci; 1999 Jun; 112 ( Pt 11)():1697-708. PubMed ID: 10318762 [TBL] [Abstract][Full Text] [Related]
14. N-WASP deficiency reveals distinct pathways for cell surface projections and microbial actin-based motility. Snapper SB; Takeshima F; Antón I; Liu CH; Thomas SM; Nguyen D; Dudley D; Fraser H; Purich D; Lopez-Ilasaca M; Klein C; Davidson L; Bronson R; Mulligan RC; Southwick F; Geha R; Goldberg MB; Rosen FS; Hartwig JH; Alt FW Nat Cell Biol; 2001 Oct; 3(10):897-904. PubMed ID: 11584271 [TBL] [Abstract][Full Text] [Related]
15. Absence of O antigen suppresses Shigella flexneri IcsA autochaperone region mutations. Teh MY; Tran ENH; Morona R Microbiology (Reading); 2012 Nov; 158(Pt 11):2835-2850. PubMed ID: 22936034 [TBL] [Abstract][Full Text] [Related]
16. N-WASP is an important protein for the actin-based motility of Shigella flexneri in the infected epithelial cells. Suzuki T; Sasakawa C Jpn J Med Sci Biol; 1998; 51 Suppl():S63-8. PubMed ID: 10211437 [No Abstract] [Full Text] [Related]
17. Cdc42 facilitates invasion but not the actin-based motility of Shigella. Shibata T; Takeshima F; Chen F; Alt FW; Snapper SB Curr Biol; 2002 Feb; 12(4):341-5. PubMed ID: 11864577 [TBL] [Abstract][Full Text] [Related]
18. A complex of N-WASP and WIP integrates signalling cascades that lead to actin polymerization. Moreau V; Frischknecht F; Reckmann I; Vincentelli R; Rabut G; Stewart D; Way M Nat Cell Biol; 2000 Jul; 2(7):441-8. PubMed ID: 10878810 [TBL] [Abstract][Full Text] [Related]
19. Actin pedestal formation by enteropathogenic Escherichia coli and intracellular motility of Shigella flexneri are abolished in N-WASP-defective cells. Lommel S; Benesch S; Rottner K; Franz T; Wehland J; Kühn R EMBO Rep; 2001 Sep; 2(9):850-7. PubMed ID: 11559594 [TBL] [Abstract][Full Text] [Related]
20. Enteropathogenic Escherichia coli, Shigella flexneri, and Listeria monocytogenes recruit a junctional protein, zonula occludens-1, to actin tails and pedestals. Hanajima-Ozawa M; Matsuzawa T; Fukui A; Kamitani S; Ohnishi H; Abe A; Horiguchi Y; Miyake M Infect Immun; 2007 Feb; 75(2):565-73. PubMed ID: 17118974 [TBL] [Abstract][Full Text] [Related] [Next] [New Search]