BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

697 related articles for article (PubMed ID: 19379791)

  • 1. Mislocalization of TDP-43 in the G93A mutant SOD1 transgenic mouse model of ALS.
    Shan X; Vocadlo D; Krieger C
    Neurosci Lett; 2009 Jul; 458(2):70-4. PubMed ID: 19379791
    [TBL] [Abstract][Full Text] [Related]  

  • 2. TDP-43 expression in mouse models of amyotrophic lateral sclerosis and spinal muscular atrophy.
    Turner BJ; Bäumer D; Parkinson NJ; Scaber J; Ansorge O; Talbot K
    BMC Neurosci; 2008 Oct; 9():104. PubMed ID: 18957104
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Lack of TDP-43 abnormalities in mutant SOD1 transgenic mice shows disparity with ALS.
    Robertson J; Sanelli T; Xiao S; Yang W; Horne P; Hammond R; Pioro EP; Strong MJ
    Neurosci Lett; 2007 Jun; 420(2):128-32. PubMed ID: 17543992
    [TBL] [Abstract][Full Text] [Related]  

  • 4. The Overexpression of TDP-43 Protein in the Neuron and Oligodendrocyte Cells Causes the Progressive Motor Neuron Degeneration in the SOD1 G93A Transgenic Mouse Model of Amyotrophic Lateral Sclerosis.
    Lu Y; Tang C; Zhu L; Li J; Liang H; Zhang J; Xu R
    Int J Biol Sci; 2016; 12(9):1140-9. PubMed ID: 27570488
    [TBL] [Abstract][Full Text] [Related]  

  • 5. Pathological Modification of TDP-43 in Amyotrophic Lateral Sclerosis with SOD1 Mutations.
    Jeon GS; Shim YM; Lee DY; Kim JS; Kang M; Ahn SH; Shin JY; Geum D; Hong YH; Sung JJ
    Mol Neurobiol; 2019 Mar; 56(3):2007-2021. PubMed ID: 29982983
    [TBL] [Abstract][Full Text] [Related]  

  • 6. Nuclear TAR DNA binding protein 43 expression in spinal cord neurons correlates with the clinical course in amyotrophic lateral sclerosis.
    Sumi H; Kato S; Mochimaru Y; Fujimura H; Etoh M; Sakoda S
    J Neuropathol Exp Neurol; 2009 Jan; 68(1):37-47. PubMed ID: 19104447
    [TBL] [Abstract][Full Text] [Related]  

  • 7. TDP-43 modification in the hSOD1(G93A) amyotrophic lateral sclerosis mouse model.
    Cai M; Lee KW; Choi SM; Yang EJ
    Neurol Res; 2015 Mar; 37(3):253-62. PubMed ID: 25213598
    [TBL] [Abstract][Full Text] [Related]  

  • 8. Pathological TDP-43 distinguishes sporadic amyotrophic lateral sclerosis from amyotrophic lateral sclerosis with SOD1 mutations.
    Mackenzie IR; Bigio EH; Ince PG; Geser F; Neumann M; Cairns NJ; Kwong LK; Forman MS; Ravits J; Stewart H; Eisen A; McClusky L; Kretzschmar HA; Monoranu CM; Highley JR; Kirby J; Siddique T; Shaw PJ; Lee VM; Trojanowski JQ
    Ann Neurol; 2007 May; 61(5):427-34. PubMed ID: 17469116
    [TBL] [Abstract][Full Text] [Related]  

  • 9. Human Cu/Zn superoxide dismutase (SOD1) overexpression in mice causes mitochondrial vacuolization, axonal degeneration, and premature motoneuron death and accelerates motoneuron disease in mice expressing a familial amyotrophic lateral sclerosis mutant SOD1.
    Jaarsma D; Haasdijk ED; Grashorn JA; Hawkins R; van Duijn W; Verspaget HW; London J; Holstege JC
    Neurobiol Dis; 2000 Dec; 7(6 Pt B):623-43. PubMed ID: 11114261
    [TBL] [Abstract][Full Text] [Related]  

  • 10. TDP-43 physically interacts with amyotrophic lateral sclerosis-linked mutant CuZn superoxide dismutase.
    Higashi S; Tsuchiya Y; Araki T; Wada K; Kabuta T
    Neurochem Int; 2010 Dec; 57(8):906-13. PubMed ID: 20933032
    [TBL] [Abstract][Full Text] [Related]  

  • 11. Tar DNA binding protein of 43 kDa (TDP-43), 14-3-3 proteins and copper/zinc superoxide dismutase (SOD1) interact to modulate NFL mRNA stability. Implications for altered RNA processing in amyotrophic lateral sclerosis (ALS).
    Volkening K; Leystra-Lantz C; Yang W; Jaffee H; Strong MJ
    Brain Res; 2009 Dec; 1305():168-82. PubMed ID: 19815002
    [TBL] [Abstract][Full Text] [Related]  

  • 12. Histological evidence of protein aggregation in mutant SOD1 transgenic mice and in amyotrophic lateral sclerosis neural tissues.
    Watanabe M; Dykes-Hoberg M; Culotta VC; Price DL; Wong PC; Rothstein JD
    Neurobiol Dis; 2001 Dec; 8(6):933-41. PubMed ID: 11741389
    [TBL] [Abstract][Full Text] [Related]  

  • 13. TDP-43 is consistently co-localized with ubiquitinated inclusions in sporadic and Guam amyotrophic lateral sclerosis but not in familial amyotrophic lateral sclerosis with and without SOD1 mutations.
    Maekawa S; Leigh PN; King A; Jones E; Steele JC; Bodi I; Shaw CE; Hortobagyi T; Al-Sarraj S
    Neuropathology; 2009 Dec; 29(6):672-83. PubMed ID: 19496940
    [TBL] [Abstract][Full Text] [Related]  

  • 14. TDP-43 immunoreactivity in neuronal inclusions in familial amyotrophic lateral sclerosis with or without SOD1 gene mutation.
    Tan CF; Eguchi H; Tagawa A; Onodera O; Iwasaki T; Tsujino A; Nishizawa M; Kakita A; Takahashi H
    Acta Neuropathol; 2007 May; 113(5):535-42. PubMed ID: 17333220
    [TBL] [Abstract][Full Text] [Related]  

  • 15. Neuron-specific overexpression of the co-chaperone Bcl-2-associated athanogene-1 in superoxide dismutase 1(G93A)-transgenic mice.
    Rohde G; Kermer P; Reed JC; Bähr M; Weishaupt JH
    Neuroscience; 2008 Dec; 157(4):844-9. PubMed ID: 18955116
    [TBL] [Abstract][Full Text] [Related]  

  • 16. Spinal inhibitory interneuron pathology follows motor neuron degeneration independent of glial mutant superoxide dismutase 1 expression in SOD1-ALS mice.
    Hossaini M; Cardona Cano S; van Dis V; Haasdijk ED; Hoogenraad CC; Holstege JC; Jaarsma D
    J Neuropathol Exp Neurol; 2011 Aug; 70(8):662-77. PubMed ID: 21760539
    [TBL] [Abstract][Full Text] [Related]  

  • 17. Aggregates of mutant protein appear progressively in dendrites, in periaxonal processes of oligodendrocytes, and in neuronal and astrocytic perikarya of mice expressing the SOD1(G93A) mutation of familial amyotrophic lateral sclerosis.
    Stieber A; Gonatas JO; Gonatas NK
    J Neurol Sci; 2000 Aug; 177(2):114-23. PubMed ID: 10980307
    [TBL] [Abstract][Full Text] [Related]  

  • 18. Characterization of human sporadic ALS biomarkers in the familial ALS transgenic mSOD1(G93A) mouse model.
    Lilo E; Wald-Altman S; Solmesky LJ; Ben Yaakov K; Gershoni-Emek N; Bulvik S; Kassis I; Karussis D; Perlson E; Weil M
    Hum Mol Genet; 2013 Dec; 22(23):4720-5. PubMed ID: 23836781
    [TBL] [Abstract][Full Text] [Related]  

  • 19. Rab1-dependent ER-Golgi transport dysfunction is a common pathogenic mechanism in SOD1, TDP-43 and FUS-associated ALS.
    Soo KY; Halloran M; Sundaramoorthy V; Parakh S; Toth RP; Southam KA; McLean CA; Lock P; King A; Farg MA; Atkin JD
    Acta Neuropathol; 2015 Nov; 130(5):679-97. PubMed ID: 26298469
    [TBL] [Abstract][Full Text] [Related]  

  • 20. Co-deposition of SOD1, TDP-43 and p62 proteinopathies in ALS: evidence for multifaceted pathways underlying neurodegeneration.
    Trist BG; Fifita JA; Hogan A; Grima N; Smith B; Troakes C; Vance C; Shaw C; Al-Sarraj S; Blair IP; Double KL
    Acta Neuropathol Commun; 2022 Aug; 10(1):122. PubMed ID: 36008843
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 35.