285 related articles for article (PubMed ID: 19389700)
1. Caspase-cleaved tau expression induces mitochondrial dysfunction in immortalized cortical neurons: implications for the pathogenesis of Alzheimer disease.
Quintanilla RA; Matthews-Roberson TA; Dolan PJ; Johnson GV
J Biol Chem; 2009 Jul; 284(28):18754-66. PubMed ID: 19389700
[TBL] [Abstract][Full Text] [Related]
2. Caspase-Cleaved Tau Impairs Mitochondrial Dynamics in Alzheimer's Disease.
Pérez MJ; Vergara-Pulgar K; Jara C; Cabezas-Opazo F; Quintanilla RA
Mol Neurobiol; 2018 Feb; 55(2):1004-1018. PubMed ID: 28084594
[TBL] [Abstract][Full Text] [Related]
3. Immortalized cortical neurons expressing caspase-cleaved tau are sensitized to endoplasmic reticulum stress induced cell death.
Matthews-Roberson TA; Quintanilla RA; Ding H; Johnson GV
Brain Res; 2008 Oct; 1234():206-12. PubMed ID: 18718455
[TBL] [Abstract][Full Text] [Related]
4. Site-specific phosphorylation and caspase cleavage differentially impact tau-microtubule interactions and tau aggregation.
Ding H; Matthews TA; Johnson GV
J Biol Chem; 2006 Jul; 281(28):19107-14. PubMed ID: 16687396
[TBL] [Abstract][Full Text] [Related]
5. Cyclosporine A (CsA) prevents synaptic impairment caused by truncated tau by caspase-3.
Tapia-Monsalves C; Olesen MA; Villavicencio-Tejo F; Quintanilla RA
Mol Cell Neurosci; 2023 Jun; 125():103861. PubMed ID: 37182572
[TBL] [Abstract][Full Text] [Related]
6. The Role of Mitochondrial Impairment in Alzheimer´s Disease Neurodegeneration: The Tau Connection.
Quntanilla RA; Tapia-Monsalves C
Curr Neuropharmacol; 2020; 18(11):1076-1091. PubMed ID: 32448104
[TBL] [Abstract][Full Text] [Related]
7. Mitochondria-tau association promotes cognitive decline and hippocampal bioenergetic deficits during the aging.
Olesen MA; Pradenas E; Villavicencio-Tejo F; Porter GA; Johnson GVW; Quintanilla RA
Free Radic Biol Med; 2024 May; 217():141-156. PubMed ID: 38552927
[TBL] [Abstract][Full Text] [Related]
8. Tau localises within mitochondrial sub-compartments and its caspase cleavage affects ER-mitochondria interactions and cellular Ca
Cieri D; Vicario M; Vallese F; D'Orsi B; Berto P; Grinzato A; Catoni C; De Stefani D; Rizzuto R; Brini M; Calì T
Biochim Biophys Acta Mol Basis Dis; 2018 Oct; 1864(10):3247-3256. PubMed ID: 30006151
[TBL] [Abstract][Full Text] [Related]
9. Site-specific phosphorylation of tau impacts mitochondrial function and response to stressors.
Isei MO; Girardi PA; Rodwell-Bullock J; Nehrke K; Johnson GVW
J Neurochem; 2024 Jun; 168(6):1019-1029. PubMed ID: 37787052
[TBL] [Abstract][Full Text] [Related]
10. Truncated tau and Aβ cooperatively impair mitochondria in primary neurons.
Quintanilla RA; Dolan PJ; Jin YN; Johnson GV
Neurobiol Aging; 2012 Mar; 33(3):619.e25-35. PubMed ID: 21450370
[TBL] [Abstract][Full Text] [Related]
11. Activation of glycogen synthase kinase 3beta promotes the intermolecular association of tau. The use of fluorescence resonance energy transfer microscopy.
Chun W; Johnson GV
J Biol Chem; 2007 Aug; 282(32):23410-7. PubMed ID: 17565981
[TBL] [Abstract][Full Text] [Related]
12. Pathological Impact of Tau Proteolytical Process on Neuronal and Mitochondrial Function: a Crucial Role in Alzheimer's Disease.
Olesen MA; Quintanilla RA
Mol Neurobiol; 2023 Oct; 60(10):5691-5707. PubMed ID: 37332018
[TBL] [Abstract][Full Text] [Related]
13. Tau accumulation causes mitochondrial distribution deficits in neurons in a mouse model of tauopathy and in human Alzheimer's disease brain.
Kopeikina KJ; Carlson GA; Pitstick R; Ludvigson AE; Peters A; Luebke JI; Koffie RM; Frosch MP; Hyman BT; Spires-Jones TL
Am J Pathol; 2011 Oct; 179(4):2071-82. PubMed ID: 21854751
[TBL] [Abstract][Full Text] [Related]
14. Abnormal interaction between the mitochondrial fission protein Drp1 and hyperphosphorylated tau in Alzheimer's disease neurons: implications for mitochondrial dysfunction and neuronal damage.
Manczak M; Reddy PH
Hum Mol Genet; 2012 Jun; 21(11):2538-47. PubMed ID: 22367970
[TBL] [Abstract][Full Text] [Related]
15. Caspase-3 cleaved tau impairs mitochondrial function through the opening of the mitochondrial permeability transition pore.
Pérez MJ; Ibarra-García-Padilla R; Tang M; Porter GA; Johnson GVW; Quintanilla RA
Biochim Biophys Acta Mol Basis Dis; 2024 Jan; 1870(1):166898. PubMed ID: 37774936
[TBL] [Abstract][Full Text] [Related]
16. Amyloid Beta and Phosphorylated Tau-Induced Defective Autophagy and Mitophagy in Alzheimer's Disease.
Reddy PH; Oliver DM
Cells; 2019 May; 8(5):. PubMed ID: 31121890
[TBL] [Abstract][Full Text] [Related]
17. Hippocampal tau oligomerization early in tau pathology coincides with a transient alteration of mitochondrial homeostasis and DNA repair in a mouse model of tauopathy.
Zheng J; Akbari M; Schirmer C; Reynaert ML; Loyens A; Lefebvre B; Buée L; Croteau DL; Galas MC; Bohr VA
Acta Neuropathol Commun; 2020 Mar; 8(1):25. PubMed ID: 32131898
[TBL] [Abstract][Full Text] [Related]
18. Caspase-9 activation and caspase cleavage of tau in the Alzheimer's disease brain.
Rohn TT; Rissman RA; Davis MC; Kim YE; Cotman CW; Head E
Neurobiol Dis; 2002 Nov; 11(2):341-54. PubMed ID: 12505426
[TBL] [Abstract][Full Text] [Related]
19. Tau facilitates Aβ-induced loss of mitochondrial membrane potential independent of cytosolic calcium fluxes in mouse cortical neurons.
Pallo SP; Johnson GV
Neurosci Lett; 2015 Jun; 597():32-7. PubMed ID: 25888814
[TBL] [Abstract][Full Text] [Related]
20. Truncated Tau Induces Mitochondrial Transport Failure Through the Impairment of TRAK2 Protein and Bioenergetics Decline in Neuronal Cells.
Quintanilla RA; Tapia-Monsalves C; Vergara EH; Pérez MJ; Aranguiz A
Front Cell Neurosci; 2020; 14():175. PubMed ID: 32848607
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]