318 related articles for article (PubMed ID: 19519404)
1. FANCJ helicase operates in the Fanconi Anemia DNA repair pathway and the response to replicational stress.
Wu Y; Brosh RM
Curr Mol Med; 2009 May; 9(4):470-82. PubMed ID: 19519404
[TBL] [Abstract][Full Text] [Related]
2. FANCJ uses its motor ATPase to destabilize protein-DNA complexes, unwind triplexes, and inhibit RAD51 strand exchange.
Sommers JA; Rawtani N; Gupta R; Bugreev DV; Mazin AV; Cantor SB; Brosh RM
J Biol Chem; 2009 Mar; 284(12):7505-17. PubMed ID: 19150983
[TBL] [Abstract][Full Text] [Related]
3. The FANCJ/MutLalpha interaction is required for correction of the cross-link response in FA-J cells.
Peng M; Litman R; Xie J; Sharma S; Brosh RM; Cantor SB
EMBO J; 2007 Jul; 26(13):3238-49. PubMed ID: 17581638
[TBL] [Abstract][Full Text] [Related]
4. Fanconi anemia group J helicase and MRE11 nuclease interact to facilitate the DNA damage response.
Suhasini AN; Sommers JA; Muniandy PA; Coulombe Y; Cantor SB; Masson JY; Seidman MM; Brosh RM
Mol Cell Biol; 2013 Jun; 33(11):2212-27. PubMed ID: 23530059
[TBL] [Abstract][Full Text] [Related]
5. Fanconi anemia group J mutation abolishes its DNA repair function by uncoupling DNA translocation from helicase activity or disruption of protein-DNA complexes.
Wu Y; Sommers JA; Suhasini AN; Leonard T; Deakyne JS; Mazin AV; Shin-Ya K; Kitao H; Brosh RM
Blood; 2010 Nov; 116(19):3780-91. PubMed ID: 20639400
[TBL] [Abstract][Full Text] [Related]
6. FANCJ (BACH1) helicase forms DNA damage inducible foci with replication protein A and interacts physically and functionally with the single-stranded DNA-binding protein.
Gupta R; Sharma S; Sommers JA; Kenny MK; Cantor SB; Brosh RM
Blood; 2007 Oct; 110(7):2390-8. PubMed ID: 17596542
[TBL] [Abstract][Full Text] [Related]
7. The Fanconi anemia proteins FANCD2 and FANCJ interact and regulate each other's chromatin localization.
Chen X; Wilson JB; McChesney P; Williams SA; Kwon Y; Longerich S; Marriott AS; Sung P; Jones NJ; Kupfer GM
J Biol Chem; 2014 Sep; 289(37):25774-82. PubMed ID: 25070891
[TBL] [Abstract][Full Text] [Related]
8. Assessing the link between BACH1 and BRCA1 in the FA pathway.
Cantor SB; Andreassen PR
Cell Cycle; 2006 Jan; 5(2):164-7. PubMed ID: 16357529
[TBL] [Abstract][Full Text] [Related]
9. FANCD2, FANCJ and BRCA2 cooperate to promote replication fork recovery independently of the Fanconi Anemia core complex.
Raghunandan M; Chaudhury I; Kelich SL; Hanenberg H; Sobeck A
Cell Cycle; 2015; 14(3):342-53. PubMed ID: 25659033
[TBL] [Abstract][Full Text] [Related]
10. FANCJ helicase defective in Fanconia anemia and breast cancer unwinds G-quadruplex DNA to defend genomic stability.
Wu Y; Shin-ya K; Brosh RM
Mol Cell Biol; 2008 Jun; 28(12):4116-28. PubMed ID: 18426915
[TBL] [Abstract][Full Text] [Related]
11. Fanconi anemia and Bloom's syndrome crosstalk through FANCJ-BLM helicase interaction.
Suhasini AN; Brosh RM
Trends Genet; 2012 Jan; 28(1):7-13. PubMed ID: 22024395
[TBL] [Abstract][Full Text] [Related]
12. FANCJ protein is important for the stability of FANCD2/FANCI proteins and protects them from proteasome and caspase-3 dependent degradation.
Clark DW; Tripathi K; Dorsman JC; Palle K
Oncotarget; 2015 Oct; 6(30):28816-32. PubMed ID: 26336824
[TBL] [Abstract][Full Text] [Related]
13. DOG-1 is the Caenorhabditis elegans BRIP1/FANCJ homologue and functions in interstrand cross-link repair.
Youds JL; Barber LJ; Ward JD; Collis SJ; O'Neil NJ; Boulton SJ; Rose AM
Mol Cell Biol; 2008 Mar; 28(5):1470-9. PubMed ID: 18086896
[TBL] [Abstract][Full Text] [Related]
14. FancJ (Brip1) loss-of-function allele results in spermatogonial cell depletion during embryogenesis and altered processing of crossover sites during meiotic prophase I in mice.
Sun X; Brieño-Enríquez MA; Cornelius A; Modzelewski AJ; Maley TT; Campbell-Peterson KM; Holloway JK; Cohen PE
Chromosoma; 2016 Jun; 125(2):237-52. PubMed ID: 26490168
[TBL] [Abstract][Full Text] [Related]
15. FANCM-FAAP24 and FANCJ: FA proteins that metabolize DNA.
Ali AM; Singh TR; Meetei AR
Mutat Res; 2009 Jul; 668(1-2):20-6. PubMed ID: 19379763
[TBL] [Abstract][Full Text] [Related]
16. A minimal threshold of FANCJ helicase activity is required for its response to replication stress or double-strand break repair.
Bharti SK; Sommers JA; Awate S; Bellani MA; Khan I; Bradley L; King GA; Seol Y; Vidhyasagar V; Wu Y; Abe T; Kobayashi K; Shin-Ya K; Kitao H; Wold MS; Branzei D; Neuman KC; Brosh RM
Nucleic Acids Res; 2018 Jul; 46(12):6238-6256. PubMed ID: 29788478
[TBL] [Abstract][Full Text] [Related]
17. Crosstalk between BRCA-Fanconi anemia and mismatch repair pathways prevents MSH2-dependent aberrant DNA damage responses.
Peng M; Xie J; Ucher A; Stavnezer J; Cantor SB
EMBO J; 2014 Aug; 33(15):1698-712. PubMed ID: 24966277
[TBL] [Abstract][Full Text] [Related]
18. FANCJ helicase controls the balance between short- and long-tract gene conversions between sister chromatids.
Nath S; Somyajit K; Mishra A; Scully R; Nagaraju G
Nucleic Acids Res; 2017 Sep; 45(15):8886-8900. PubMed ID: 28911102
[TBL] [Abstract][Full Text] [Related]
19. FANCJ suppresses microsatellite instability and lymphomagenesis independent of the Fanconi anemia pathway.
Matsuzaki K; Borel V; Adelman CA; Schindler D; Boulton SJ
Genes Dev; 2015 Dec; 29(24):2532-46. PubMed ID: 26637282
[TBL] [Abstract][Full Text] [Related]
20. [The role of the Fanconi anemia pathway in DNA repair and maintenance of genome stability].
Koczorowska AM; Białkowska A; Kluzek K; Zdzienicka MZ
Postepy Hig Med Dosw (Online); 2014 May; 68():459-72. PubMed ID: 24864098
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]