137 related articles for article (PubMed ID: 19553389)
1. Quantitative evaluation of the role of Epstein-Barr virus immediate-early protein BZLF1 in B-cell transformation.
Katsumura KR; Maruo S; Wu Y; Kanda T; Takada K
J Gen Virol; 2009 Oct; 90(Pt 10):2331-2341. PubMed ID: 19553389
[TBL] [Abstract][Full Text] [Related]
2. The B-cell specific transcription factor, Oct-2, promotes Epstein-Barr virus latency by inhibiting the viral immediate-early protein, BZLF1.
Robinson AR; Kwek SS; Kenney SC
PLoS Pathog; 2012 Feb; 8(2):e1002516. PubMed ID: 22346751
[TBL] [Abstract][Full Text] [Related]
3. B cells infected with Type 2 Epstein-Barr virus (EBV) have increased NFATc1/NFATc2 activity and enhanced lytic gene expression in comparison to Type 1 EBV infection.
Romero-Masters JC; Huebner SM; Ohashi M; Bristol JA; Benner BE; Barlow EA; Turk GL; Nelson SE; Baiu DC; Van Sciver N; Ranheim EA; Gumperz J; Sherer NM; Farrell PJ; Johannsen EC; Kenney SC
PLoS Pathog; 2020 Feb; 16(2):e1008365. PubMed ID: 32059024
[TBL] [Abstract][Full Text] [Related]
4. Epstein-Barr virus BZLF1 gene, a switch from latency to lytic infection, is expressed as an immediate-early gene after primary infection of B lymphocytes.
Wen W; Iwakiri D; Yamamoto K; Maruo S; Kanda T; Takada K
J Virol; 2007 Jan; 81(2):1037-42. PubMed ID: 17079287
[TBL] [Abstract][Full Text] [Related]
5. Methylation profiling of Epstein-Barr virus immediate-early gene promoters, BZLF1 and BRLF1 in tumors of epithelial, NK- and B-cell origins.
Li L; Su X; Choi GC; Cao Y; Ambinder RF; Tao Q
BMC Cancer; 2012 Mar; 12():125. PubMed ID: 22458933
[TBL] [Abstract][Full Text] [Related]
6. Shutoff of BZLF1 gene expression is necessary for immortalization of primary B cells by Epstein-Barr virus.
Yu X; McCarthy PJ; Wang Z; Gorlen DA; Mertz JE
J Virol; 2012 Aug; 86(15):8086-96. PubMed ID: 22623769
[TBL] [Abstract][Full Text] [Related]
7. Epstein-Barr Virus BZLF1-Mediated Downregulation of Proinflammatory Factors Is Essential for Optimal Lytic Viral Replication.
Li Y; Long X; Huang L; Yang M; Yuan Y; Wang Y; Delecluse HJ; Kuang E
J Virol; 2016 Jan; 90(2):887-903. PubMed ID: 26537683
[TBL] [Abstract][Full Text] [Related]
8. Epstein-Barr virus (EBV) EB1/Zta protein provided in trans and competent for the activation of productive cycle genes does not activate the BZLF1 gene in the EBV genome.
Le Roux F; Sergeant A; Corbo L
J Gen Virol; 1996 Mar; 77 ( Pt 3)():501-9. PubMed ID: 8601788
[TBL] [Abstract][Full Text] [Related]
9. The ZIIR element of the Epstein-Barr virus BZLF1 promoter plays a central role in establishment and maintenance of viral latency.
Yu X; McCarthy PJ; Lim HJ; Iempridee T; Kraus RJ; Gorlen DA; Mertz JE
J Virol; 2011 May; 85(10):5081-90. PubMed ID: 21389123
[TBL] [Abstract][Full Text] [Related]
10. Interaction of phospholipid scramblase 1 with the Epstein-Barr virus protein BZLF1 represses BZLF1-mediated lytic gene transcription.
Kusano S; Ikeda M
J Biol Chem; 2019 Oct; 294(41):15104-15116. PubMed ID: 31434743
[TBL] [Abstract][Full Text] [Related]
11. BZLF1 transcript variants in Epstein-Barr virus-positive epithelial cell lines.
Needham J; Adamson AL
Virus Genes; 2019 Dec; 55(6):779-785. PubMed ID: 31552622
[TBL] [Abstract][Full Text] [Related]
12. The lytic phase of epstein-barr virus requires a viral genome with 5-methylcytosine residues in CpG sites.
Kalla M; Göbel C; Hammerschmidt W
J Virol; 2012 Jan; 86(1):447-58. PubMed ID: 22031942
[TBL] [Abstract][Full Text] [Related]
13. Regulation and dysregulation of Epstein-Barr virus latency: implications for the development of autoimmune diseases.
Niller HH; Wolf H; Minarovits J
Autoimmunity; 2008 May; 41(4):298-328. PubMed ID: 18432410
[TBL] [Abstract][Full Text] [Related]
14. Identification of a novel element involved in regulation of the lytic switch BZLF1 gene promoter of Epstein-Barr virus.
Kraus RJ; Mirocha SJ; Stephany HM; Puchalski JR; Mertz JE
J Virol; 2001 Jan; 75(2):867-77. PubMed ID: 11134300
[TBL] [Abstract][Full Text] [Related]
15. Glucocorticoids activate Epstein Barr virus lytic replication through the upregulation of immediate early BZLF1 gene expression.
Yang EV; Webster Marketon JI; Chen M; Lo KW; Kim SJ; Glaser R
Brain Behav Immun; 2010 Oct; 24(7):1089-96. PubMed ID: 20466055
[TBL] [Abstract][Full Text] [Related]
16. EBV lytic infection enhances transformation of B-lymphocytes infected with EBV in the presence of T-lymphocytes.
Katsumura KR; Maruo S; Takada K
J Med Virol; 2012 Mar; 84(3):504-10. PubMed ID: 22246839
[TBL] [Abstract][Full Text] [Related]
17. hTERT inhibits the Epstein-Barr virus lytic cycle and promotes the proliferation of primary B lymphocytes: implications for EBV-driven lymphomagenesis.
Terrin L; Dolcetti R; Corradini I; Indraccolo S; Dal Col J; Bertorelle R; Bonaldi L; Esposito G; De Rossi A
Int J Cancer; 2007 Aug; 121(3):576-87. PubMed ID: 17417773
[TBL] [Abstract][Full Text] [Related]
18. Cellular differentiation regulator BLIMP1 induces Epstein-Barr virus lytic reactivation in epithelial and B cells by activating transcription from both the R and Z promoters.
Reusch JA; Nawandar DM; Wright KL; Kenney SC; Mertz JE
J Virol; 2015 Feb; 89(3):1731-43. PubMed ID: 25410866
[TBL] [Abstract][Full Text] [Related]
19. Cross talk between EBV and telomerase: the role of TERT and NOTCH2 in the switch of latent/lytic cycle of the virus.
Giunco S; Celeghin A; Gianesin K; Dolcetti R; Indraccolo S; De Rossi A
Cell Death Dis; 2015 May; 6(5):e1774. PubMed ID: 26018735
[TBL] [Abstract][Full Text] [Related]
20. Differences in the extent of activation of Epstein-Barr virus replicative gene expression among four nonproducer cell lines stably transformed by oriP/BZLF1 plasmids.
Gradoville L; Grogan E; Taylor N; Miller G
Virology; 1990 Oct; 178(2):345-54. PubMed ID: 2171186
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
